Literature DB >> 35666457

Association between innate immunity gene polymorphisms and neonatal sepsis development: a systematic review and meta-analysis.

Tamara Sljivancanin Jakovljevic1, Jelena Martic2,3, Jelena Jacimovic4, Nadja Nikolic5, Jelena Milasin5, Tanja Lazić Mitrović6.   

Abstract

BACKGROUND: The aim of this meta-analysis was to analyze all available data from studies investigating associations between polymorphisms in genes responsible for innate immunity and neonatal sepsis development.
METHODS: A comprehensive literature search, reported following the Preferred Reporting Items for Systematic Reviews and Meta-Analyses-S guidelines, was performed with no language restriction. Studies derived using the PICO (population, intervention, comparison and outcomes) strategy, with data on the genotype distribution for innate immunity gene polymorphisms in newborns with and without sepsis. Data were analyzed using Review Manager. The Cochran-Mantel-Haenszel test was used to calculate odds ratios with 95% confidence intervals. Heterogeneity was tested using the I2 index.
RESULTS: From a total of 9428 possibly relevant articles, 33 qualified for inclusion in this systematic review. According to the STrengthening the REporting of Genetic Association Studies, 23 studies were found to be of moderate quality, while 10 were of low quality. The results showed an association of the mannose-binding lectin (MBL) exon 1 genetic polymorphism with the risk of culture-proven sepsis. Toll-like receptor (TLR) 4 rs4986791 genotype distribution suggests its association with the increased risk of culture-proven sepsis. The certainty of evidence per GRADE (Grading of Recommendations, Assessment, Development, and Evaluation) varied from very low to low. Publication bias was not detected.
CONCLUSIONS: Out of the 11 investigated single-nucleotide polymorphisms, this meta-analysis found a possible association between the risk for culture-proven sepsis and MBL exon 1 and TLR4 rs4986791 polymorphisms. There is an evident need for larger well-designed, multicentric observational studies investigating inflammatory gene polymorphisms in neonatal sepsis.
© 2022. Children's Hospital, Zhejiang University School of Medicine.

Entities:  

Keywords:  Genetic; Immunity; Innate; Neonatal sepsis; Polymorphism

Mesh:

Substances:

Year:  2022        PMID: 35666457     DOI: 10.1007/s12519-022-00569-7

Source DB:  PubMed          Journal:  World J Pediatr            Impact factor:   9.186


  61 in total

1.  Mutations of genes involved in the innate immune system as predictors of sepsis in very low birth weight infants.

Authors:  Peter Ahrens; Evelyn Kattner; Birgit Köhler; Christoph Härtel; Jürgen Seidenberg; Hugo Segerer; Jens Möller; Wolfgang Göpel
Journal:  Pediatr Res       Date:  2004-01-22       Impact factor: 3.756

2.  Innate immune gene polymorphisms and their association with neonatal sepsis.

Authors:  Snehal L Martin; Saumil Desai; Ruchi Nanavati; Roshan B Colah; Kanjaksha Ghosh; Malay B Mukherjee
Journal:  Infect Genet Evol       Date:  2018-04-30       Impact factor: 3.342

Review 3.  Challenges in developing a consensus definition of neonatal sepsis.

Authors:  Matthew McGovern; Eric Giannoni; Helmut Kuester; Mark A Turner; Agnes van den Hoogen; Joseph M Bliss; Joyce M Koenig; Fleur M Keij; Jan Mazela; Rebecca Finnegan; Marina Degtyareva; Sinno H P Simons; Willem P de Boode; Tobias Strunk; Irwin K M Reiss; James L Wynn; Eleanor J Molloy
Journal:  Pediatr Res       Date:  2020-03-03       Impact factor: 3.756

Review 4.  The pathogenesis of sepsis.

Authors:  Deborah J Stearns-Kurosawa; Marcin F Osuchowski; Catherine Valentine; Shinichiro Kurosawa; Daniel G Remick
Journal:  Annu Rev Pathol       Date:  2011       Impact factor: 23.472

5.  Role of polymorphic variants as genetic modulators of infection in neonatal sepsis.

Authors:  Asmaa Abu-Maziad; Kendra Schaa; Edward F Bell; John M Dagle; Margaret Cooper; Mary L Marazita; Jeffrey C Murray
Journal:  Pediatr Res       Date:  2010-10       Impact factor: 3.756

6.  Neonatal cause-of-death estimates for the early and late neonatal periods for 194 countries: 2000-2013.

Authors:  Shefali Oza; Joy E Lawn; Daniel R Hogan; Colin Mathers; Simon N Cousens
Journal:  Bull World Health Organ       Date:  2014-11-17       Impact factor: 9.408

7.  The interleukin-27 -964A>G polymorphism enhances sepsis-induced inflammatory responses and confers susceptibility to the development of sepsis.

Authors:  Junbing He; Quanfu Zhang; Wenying Zhang; Feng Chen; Tian Zhao; Yao Lin; Jia Li; Yansong Liu; Yuchun Liu; Yiming Shao
Journal:  Crit Care       Date:  2018-09-30       Impact factor: 9.097

8.  The association between interleukin-6 gene -174G/C single nucleotide polymorphism and sepsis: an updated meta-analysis with trial sequential analysis.

Authors:  Yao Chen; Yanyan Hu; Zhenju Song
Journal:  BMC Med Genet       Date:  2019-02-19       Impact factor: 2.103

9.  Association of Gene Polymorphism of Bactericidal Permeability Increasing Protein Rs4358188, Cluster of Differentiation 14 Rs2569190, Interleukin 1β Rs1143643 and Matrix Metalloproteinase-16 Rs2664349 with Neonatal Sepsis.

Authors:  Mustarim Mustarim; Yanwirasti Yanwirasti; Jamsari Jamsari; Rukmono Rukmono; Ricvan Dana Nindrea
Journal:  Open Access Maced J Med Sci       Date:  2019-08-31

10.  Neonatal infections in Saudi Arabia: Association with cytokine gene polymorphisms.

Authors:  Gamal Allam; Adnan A Alsulaimani; Ali K Alzaharani; Amre Nasr
Journal:  Cent Eur J Immunol       Date:  2015-04-22       Impact factor: 2.085

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