Literature DB >> 35601164

Yes-associated protein promotes endothelial-to-mesenchymal transition of endothelial cells in choroidal neovascularization fibrosis.

Rong Zou1, Yi-Fan Feng1, Ya-Hui Xu1, Min-Qian Shen1, Xi Zhang1, Yuan-Zhi Yuan1,2.   

Abstract

AIM: To reveal whether and how Yes-associated protein (YAP) promotes the occurrence of subretinal fibrosis in age-related macular degeneration (AMD).
METHODS: Cobalt chloride (CoCl2) was used in primary human umbilical vein endothelial cells (HUVECs) to induce hypoxia in vitro. Eight-week-old male C57BL/6J mice weighing 19-25 g were used for a choroidal neovascularization (CNV) model induced by laser photocoagulation in vivo. Expression levels of YAP, phosphorylated YAP, mesenchymal markers [α smooth muscle actin (α-SMA), vimentin, and Snail], and endothelial cell markers (CD31 and zonula occludens 1) were measured by Western blotting, quantitative real-time PCR, and immunofluorescence microscopy. Small molecules YC-1 (Lificiguat, a specific inhibitor of hypoxia-inducible factor 1α), CA3 (CIL56, an inhibitor of YAP), and XMU-MP-1 (an inhibitor of Hippo kinase MST1/2, which activates YAP) were used to explore the underlying mechanism.
RESULTS: CoCl2 increased expression of mesenchymal markers, decreased expression of endothelial cell markers, and enhanced the ability of primary HUVECs to proliferate and migrate. YC-1 suppressed hypoxia-induced endothelial-to-mesenchymal transition (EndMT). Moreover, hypoxia promoted total expression, inhibited phosphorylation, and enhanced the transcriptional activity of YAP. XMU-MP-1 enhanced hypoxia-induced EndMT, whereas CA3 elicited the opposite effect. Expression of YAP, α-SMA, and vimentin were upregulated in the laser-induced CNV model. However, silencing of YAP by vitreous injection of small interfering RNA targeting YAP could reverse these changes.
CONCLUSION: The findings reveal a critical role of the hypoxia-inducible factor-1α (HIF-1α)/YAP signaling axis in EndMT and provide a new therapeutic target for treatment of subretinal fibrosis in AMD. International Journal of Ophthalmology Press.

Entities:  

Keywords:  Yes-associated protein; age-related macular degeneration; choroidal neovascularization; endothelial-to-mesenchymal transition; hypoxia-inducible factor-1α

Year:  2022        PMID: 35601164      PMCID: PMC9091893          DOI: 10.18240/ijo.2022.05.03

Source DB:  PubMed          Journal:  Int J Ophthalmol        ISSN: 2222-3959            Impact factor:   1.645


  42 in total

1.  Subchoroidal Release of VEGF and bFGF Produces Choroidal Neovascularization in Rabbit.

Authors:  Corinne G Wong; Mehran Taban; Kathryn Osann; Fred N Ross-Cisneros; T C Bruice; Grit Zahn; Timothy You
Journal:  Curr Eye Res       Date:  2016-10-17       Impact factor: 2.424

2.  A Novel YAP1 Inhibitor Targets CSC-Enriched Radiation-Resistant Cells and Exerts Strong Antitumor Activity in Esophageal Adenocarcinoma.

Authors:  Shumei Song; Min Xie; Ailing W Scott; Jiankang Jin; Lang Ma; Xiaochuan Dong; Heath D Skinner; Randy L Johnson; Sheng Ding; Jaffer A Ajani
Journal:  Mol Cancer Ther       Date:  2017-11-22       Impact factor: 6.261

3.  YAP-Induced Endothelial-Mesenchymal Transition in Oral Submucous Fibrosis.

Authors:  J Li; M Yao; X Zhu; Q Li; J He; L Chen; W Wang; C Zhu; T Shen; R Cao; C Fang
Journal:  J Dent Res       Date:  2019-07       Impact factor: 6.116

4.  Silencing Snail suppresses tumor cell proliferation and invasion by reversing epithelial-to-mesenchymal transition and arresting G2/M phase in non-small cell lung cancer.

Authors:  Xueying Yang; Mengmeng Han; Haibo Han; Bingjing Wang; Sheng Li; Zhiqian Zhang; Wei Zhao
Journal:  Int J Oncol       Date:  2017-02-21       Impact factor: 5.650

5.  Galectin-1 promotes choroidal neovascularization and subretinal fibrosis mediated via epithelial-mesenchymal transition.

Authors:  Di Wu; Atsuhiro Kanda; Ye Liu; Satoru Kase; Kousuke Noda; Susumu Ishida
Journal:  FASEB J       Date:  2018-10-02       Impact factor: 5.191

6.  Inhibition of RACK1 ameliorates choroidal neovascularization formation in vitro and in vivo.

Authors:  Xiaojuan Liu; Manhui Zhu; Xiaowei Yang; Ying Wang; Bai Qin; Chen Cui; Hui Chen; Aimin Sang
Journal:  Exp Mol Pathol       Date:  2016-04-22       Impact factor: 3.362

7.  Endothelial Mesenchymal Transition in Hypoxic Microvascular Endothelial Cells and Paracrine Induction of Cardiomyocyte Apoptosis Are Mediated via TGFβ₁/SMAD Signaling.

Authors:  Isabella Sniegon; Mona Prieß; Jacqueline Heger; Rainer Schulz; Gerhild Euler
Journal:  Int J Mol Sci       Date:  2017-10-31       Impact factor: 5.923

8.  RUNX3 modulates hypoxia-induced endothelial-to-mesenchymal transition of human cardiac microvascular endothelial cells.

Authors:  Yanhua Liu; Jin Zou; Bingong Li; Yuqin Wang; Delong Wang; Yanqin Hao; Xuan Ke; Xingxing Li
Journal:  Int J Mol Med       Date:  2017-05-22       Impact factor: 4.101

9.  Inhibition of YAP ameliorates choroidal neovascularization via inhibiting endothelial cell proliferation.

Authors:  Zhenzhen Yan; Haihong Shi; Rongrong Zhu; Lele Li; Bai Qin; Lihua Kang; Hui Chen; Huaijin Guan
Journal:  Mol Vis       Date:  2018-01-31       Impact factor: 2.367

10.  WWC3 inhibits epithelial-mesenchymal transition of lung cancer by activating Hippo-YAP signaling.

Authors:  Qiang Han; Joachim Kremerskothen; Xuyong Lin; Xiupeng Zhang; Xuezhu Rong; Di Zhang; Enhua Wang
Journal:  Onco Targets Ther       Date:  2018-05-08       Impact factor: 4.147

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