| Literature DB >> 3559758 |
Y Yamamoto, E Niki, Y Kamiya, M Miki, H Tamai, M Mino.
Abstract
Erythrocytes of vitamin E-deficient rats and normal rats were oxidized at 37 degrees C by molecular oxygen using a free radical initiator. The erythrocytes were oxidized by a free radical chain mechanism with kinetic chain length considerably larger than 1 and resulted in hemolysis. Vitamin E suppressed both oxidation and hemolysis, but the extent of hemolysis was determined primarily by the extent of oxidation independent of the presence or absence of vitamin E.Entities:
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Year: 1986 PMID： 3559758 DOI： 10.3177/jnsv.32.475
Source DB: PubMed Journal: J Nutr Sci Vitaminol (Tokyo) ISSN： 0301-4800 Impact factor: 2.000