Shanmei Chen1, Junnan Feng1,2, Chuanke Zhao3, Lixin Wang1, Lin Meng1, Caiyun Liu1, Shaoqing Cai4, Yanxing Jia4, Like Qu5, Chengchao Shou6. 1. Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Department of Biochemistry and Molecular Biology, Peking University Cancer Hospital & Institute, Beijing, China. 2. Key Laboratory of Molecular Pathology, The Affiliated Cancer Hospital of Zhengzhou University, Zhengzhou, China. 3. Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Department of Biochemistry and Molecular Biology, Peking University Cancer Hospital & Institute, Beijing, China. zhaochk@bjmu.edu.cn. 4. Key Laboratory of Natural and Biomimetic Drugs, School of Pharmaceutical Sciences, Peking University, Beijing, China. 5. Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Department of Biochemistry and Molecular Biology, Peking University Cancer Hospital & Institute, Beijing, China. qulike99@163.com. 6. Key Laboratory of Carcinogenesis and Translational Research (Ministry of Education/Beijing), Department of Biochemistry and Molecular Biology, Peking University Cancer Hospital & Institute, Beijing, China. cshou@vip.sina.com.
Correction to: Signal Transduction and Targeted Therapy 10.1038/s41392-021-00739-5, published online 03 December 2021After online publication of the letter[1], the author found two images in the supplement materials were used incorrectly. Additionally, there is an error in the chemical structure of Aiphanol in Figs. 1a and 1s that needs to be corrected. The correct data are provided as follows. The key findings of the article are not affected by these corrections. The original article has been corrected.
Fig. 1
a Structure of Aiphanol. s The schematic representation of the mechanism
a Structure of Aiphanol. s The schematic representation of the mechanismTranswell chamber analysis of SGR’s effects on the migratory and invasive abilities of HUVECs. Migrated or invaded cells were photographed and relative migration/invasion value was calculated (n = 6 per group). Scale bar, 100 μm. *P < 0.05; **P < 0.01; ***P < 0.001.Matrigel plug assay of Aiphanol’s inhibition on the newly formed vessels. Paraffin-embedded sections of Matrigel plugs were stained with Hematoxylin and eosin (HE) or probed with anti-CD31 (brown). The numbers of neovessels (red arrows) were counted and compared (n = 4 per group). Scale bar, 0.5 cm (plugs) and 50 μm (sections). Data represented mean ± SEM. *P < 0.05.Supplemental materialsSupplementary Fig S1cSupplementary Fig S4b
Fig. 1