Literature DB >> 35261119

Chronic stress promotes an immunologic inflammatory state and head and neck cancer growth in a humanized murine model.

Joseph Zenga1, Musaddiq J Awan2, Anne Frei2, Ellie Petrie2, Guru Prasad Sharma2, Aditya Shreenivas3, Monica Shukla2, Heather A Himburg2.   

Abstract

BACKGROUND: Despite the importance of immune response and environmental stress on head and neck cancer (HNC) outcomes, no current pre-clinical stress model includes a humanized immune system.
METHODS: We investigated the effects of chronic stress induced by social isolation on tumor growth and human immune response in subcutaneous HNC tumors grown in NSG-SGM3 mice engrafted with a human immune system.
RESULTS: Tumor growth (p < 0.0001) and lung metastases (p = 0.035) were increased in socially isolated versus control animals. Chronic stress increased intra-tumoral CD4+ T-cell infiltrate (p = 0.005), plasma SDF-1 (p < 0.0001) expression, and led to tumor cell dedifferentiation toward a cancer stem cell phenotype (CD44+ /ALDHhigh , p = 0.025).
CONCLUSIONS: Chronic stress induced immunophenotypic changes, increased tumor growth, and metastasis in HNC in a murine model with a humanized immune system. This model system may provide further insight into the immunologic and oncologic impact of chronic stress on patients with HNC.
© 2022 Wiley Periodicals LLC.

Entities:  

Keywords:  SDF-1; cancer stem cell; chronic stress; head and neck cancer; humanized mouse model; immunophenotype

Mesh:

Year:  2022        PMID: 35261119      PMCID: PMC9081149          DOI: 10.1002/hed.27028

Source DB:  PubMed          Journal:  Head Neck        ISSN: 1043-3074            Impact factor:   3.821


  38 in total

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