| Literature DB >> 35251486 |
Shouqian Dai1, Ting Liang1, Xiu Shi2, Zongping Luo1, Huilin Yang1.
Abstract
[This corrects the article DOI: 10.1155/2021/6672978.].Entities:
Year: 2022 PMID: 35251486 PMCID: PMC8890876 DOI: 10.1155/2022/9874240
Source DB: PubMed Journal: Oxid Med Cell Longev ISSN: 1942-0994 Impact factor: 6.543
Figure 1SAB reversed the effects on the antioxidant system induced by puncture injury in vivo that activated the JAK2/STAT3 signaling pathway. Animals were divided into three groups: control group, IDD group, or SAB group. Concentrations of (a) GSH, (b) SOD2, and (c) MDA were measured by assay kits. (d) Expression levels of phosphorylated and total JAK2 and STAT3 in IVDs were measured by Western blotting, and the relative ratios of (e) p-JAK2/JAK2 and (f) p-STAT3/STAT3 were calculated from gray-level values. ∗P < 0 : 05 compared with the control group; #P < 0 : 05 compared with the IDD group. SAB: salvianolic acid B; GSH: glutathione; SOD2: superoxide dismutase 2; MDA: malondialdehyde; GAPDH: glyceraldehyde-3-phosphate dehydrogenase; JAK2: Janus kinase 2; STAT3: signal transducer and activator of transcription 3.