Mehmet Kanbay1, Sidar Copur2, Atalay Demiray2, Kathherine R Tuttler3,4. 1. Department of Medicine, Division of Nephrology, Koc University School of Medicine, 34010, Istanbul, Turkey. mkanbay@ku.edu.tr. 2. Department of Medicine, Koc University School of Medicine, Istanbul, Turkey. 3. Division of Nephrology, University of Washington, Seattle, WA, USA. 4. Providence Medical Research Center, Providence Health Care, Spokane, WA, USA.
Abstract
PURPOSE OF REVIEW: Health consequences of nighttime eating, as a publicly discussed eating behavior type, have been speculated lately. Nighttime eating has been linked to various metabolic outcomes including hyperlipidemia, hypertriglyceridemia, hyperglycemia, weight gain, elevated blood pressure, obesity, and metabolic syndrome, and cardiorenal outcomes such as atherosclerosis, a decline in eGFR, and proteinuria. RECENT FINDINGS: Although the exact underlying pathophysiological mechanism is not yet clear, multiple hypotheses including disrupted circadian rhythm, altered hormonal levels, and decline in cellular regeneration have been proposed. In this review, we aim to evaluate the growing literature on nighttime eating behavior in terms of metabolic and cardiorenal outcomes, pathophysiological basis, and potential therapeutic alternatives.
PURPOSE OF REVIEW: Health consequences of nighttime eating, as a publicly discussed eating behavior type, have been speculated lately. Nighttime eating has been linked to various metabolic outcomes including hyperlipidemia, hypertriglyceridemia, hyperglycemia, weight gain, elevated blood pressure, obesity, and metabolic syndrome, and cardiorenal outcomes such as atherosclerosis, a decline in eGFR, and proteinuria. RECENT FINDINGS: Although the exact underlying pathophysiological mechanism is not yet clear, multiple hypotheses including disrupted circadian rhythm, altered hormonal levels, and decline in cellular regeneration have been proposed. In this review, we aim to evaluate the growing literature on nighttime eating behavior in terms of metabolic and cardiorenal outcomes, pathophysiological basis, and potential therapeutic alternatives.
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