Literature DB >> 35037467

The role of TRPC6 channel in chronic kidney disease.

Oleg Palygin1,2.   

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Year:  2022        PMID： 35037467      PMCID： PMC8816618          DOI： 10.1152/ajprenal.00455.2021

Source DB:  PubMed          Journal:  Am J Physiol Renal Physiol        ISSN： 1522-1466


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  10 in total

1.  TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function.

Authors:  Jochen Reiser; Krishna R Polu; Clemens C Möller; Peter Kenlan; Mehmet M Altintas; Changli Wei; Christian Faul; Stephanie Herbert; Ivan Villegas; Carmen Avila-Casado; Mary McGee; Hikaru Sugimoto; Dennis Brown; Raghu Kalluri; Peter Mundel; Paula L Smith; David E Clapham; Martin R Pollak
Journal:  Nat Genet       Date:  2005-05-27       Impact factor: 38.330

2.  Protective role of Trpc6 knockout in the progression of diabetic kidney disease.

Authors:  Denisha Spires; Daria V Ilatovskaya; Vladislav Levchenko; Paula E North; Aron M Geurts; Oleg Palygin; Alexander Staruschenko
Journal:  Am J Physiol Renal Physiol       Date:  2018-06-20

Review 3.  TRPC channels: Regulation, dysregulation and contributions to chronic kidney disease.

Authors:  Stuart E Dryer; Hila Roshanravan; Eun Young Kim
Journal:  Biochim Biophys Acta Mol Basis Dis       Date:  2019-04-04       Impact factor: 5.187

Review 4.  TRPC6 channel as an emerging determinant of the podocyte injury susceptibility in kidney diseases.

Authors:  Daria V Ilatovskaya; Alexander Staruschenko
Journal:  Am J Physiol Renal Physiol       Date:  2015-06-17

5.  A mutation in the TRPC6 cation channel causes familial focal segmental glomerulosclerosis.

Authors:  Michelle P Winn; Peter J Conlon; Kelvin L Lynn; Merry Kay Farrington; Tony Creazzo; April F Hawkins; Nikki Daskalakis; Shu Ying Kwan; Seth Ebersviller; James L Burchette; Margaret A Pericak-Vance; David N Howell; Jeffery M Vance; Paul B Rosenberg
Journal:  Science       Date:  2005-05-05       Impact factor: 47.728

6.  Transient receptor potential cation channel 6 contributes to kidney injury induced by diabetes and hypertension.

Authors:  Zhen Wang; Yiling Fu; Jussara M do Carmo; Alexandre A da Silva; Xuan Li; Alan Mouton; Ana Carolina M Omoto; Jaylan Sears; John E Hall
Journal:  Am J Physiol Renal Physiol       Date:  2021-12-06

7.  A mutation in TRPC6 channels abolishes their activation by hypoosmotic stretch but does not affect activation by diacylglycerol or G protein signaling cascades.

Authors:  Cory Wilson; Stuart E Dryer
Journal:  Am J Physiol Renal Physiol       Date:  2014-03-05

8.  Podocyte-specific overexpression of wild type or mutant trpc6 in mice is sufficient to cause glomerular disease.

Authors:  Paola Krall; Cesar P Canales; Pamela Kairath; Paulina Carmona-Mora; Jessica Molina; J Daniel Carpio; Phillip Ruiz; Sergio A Mezzano; Jing Li; Changli Wei; Jochen Reiser; Juan I Young; Katherina Walz
Journal:  PLoS One       Date:  2010-09-20       Impact factor: 3.240

9.  Increased glomerular filtration rate and impaired contractile function of mesangial cells in TRPC6 knockout mice.

Authors:  Weizu Li; Yanfeng Ding; Crystal Smedley; Yanxia Wang; Sarika Chaudhari; Lutz Birnbaumer; Rong Ma
Journal:  Sci Rep       Date:  2017-06-23       Impact factor: 4.379

10.  Role of opioid signaling in kidney damage during the development of salt-induced hypertension.

Authors:  Daria Golosova; Oleg Palygin; Ruslan Bohovyk; Christine A Klemens; Vladislav Levchenko; Denisha R Spires; Elena Isaeva; Ashraf El-Meanawy; Alexander Staruschenko
Journal:  Life Sci Alliance       Date:  2020-10-12
  10 in total

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