| Literature DB >> 35033074 |
Shu Liu1,2, Yewei Zhang3, Shien Cui4,5, Dajiang Song6, Bo Li3, Qian Chen3, Guangyu Yao7, Bin Gong8.
Abstract
Entities:
Year: 2022 PMID: 35033074 PMCID: PMC8760688 DOI: 10.1186/s12935-021-02437-2
Source DB: PubMed Journal: Cancer Cell Int ISSN: 1475-2867 Impact factor: 5.722
Fig. 4NAP1L1 interacts with HDGF. A, B Co-IP assay was performed to identify the interaction of NAP1L1 with HDGF. C Immunofluorescence of breast cancer cells showed subcellular localization of NAP1L1 (green) and HDGF (red) by confocal microscopy. DAPI (blue) figure showed nucleus. Merge figure showed yellow dots representing colocalization of NAP1L1 and HDGF in the cytoplasm. (scale bar: 5 μm). The data are obtained from three independent experiment
Fig. 5HDGF recruits c-Jun. A Immunofluorescence of breast cancer cells showed subcellular localization of HDGF (red) and c-Jun (green) byconfocal microscopy. DAPI (blue) figure showed nucleus. Merge figure showed yellow dots representing colocalization of HDGF and c-Jun in the cytoplasm and nucleus. (scale bar: 5 μm). B, C Co-IP assay was performed to identify the interaction of HDGF with c-Jun. The data are obtained from three independent experiment