Literature DB >> 34966070

Influence of Immune Cell Subtypes on Mitochondrial Measurements in Peripheral Blood Mononuclear Cells From Children with Sepsis.

Scott L Weiss1,2,3, Sarah E Henrickson4,5,6, Robert B Lindell1,2,6, Laura F Sartori4, Donglan Zhang1,3, Jenny Bush1, Sumera Farooqi1,3, Jonathan Starr1,3, Clifford S Deutschman7, Francis X McGowan1,3, Lance Becker8, Florin Tuluc9, E John Wherry5,10, Martin Picard11, Douglas C Wallace3,4.   

Abstract

INTRODUCTION: Peripheral blood mononuclear cells (PBMCs) are commonly used to compare mitochondrial function in patients with versus without sepsis, but how these measurements in this mixed cell population vary by composition of immune cell subtypes is not known, especially in children. We determined the effect of changing immune cell composition on PBMC mitochondrial respiration and content in children with and without sepsis.
METHODS: PBMC mitochondrial respiration and citrate synthase (CS) activity, a marker of mitochondrial content, were measured in 167 children with sepsis at three timepoints (day 1-2, 3-5, and 8-14) and once in 19 nonseptic controls. The proportion of lymphocytes and monocytes and T, B, and NK cells was measured using flow cytometry. More specific CD4+ and CD8+ T cell subsets were measured from 13 sepsis patients and 6 controls. Spearman's correlation and simple and mixed effects linear regression were used to determine the association of PBMC mitochondrial measures with proportion of immune cell subtypes.
RESULTS: PBMC mitochondrial respiration and CS activity were correlated with proportion of monocytes, lymphocytes, T B, and NK cells in controls, but not in sepsis patients. PBMC mitochondrial respiration was correlated with CD4+ and CD8+ T cell subsets in both groups. After controlling for differences in immune cell composition between groups using linear regression models, PBMC respiration and CS activity remained lower in sepsis patients than controls.
CONCLUSIONS: Mitochondrial measurements from PBMCs varied with changes in immune cell composition in children with and without sepsis. However, differences in PBMC mitochondrial measurements between sepsis patients and controls were at least partially attributable to the effects of sepsis rather than solely an epiphenomena of variable immune cell composition.
Copyright © 2021 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of the Shock Society.

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Mesh:

Year:  2021        PMID: 34966070      PMCID: PMC9117409          DOI: 10.1097/SHK.0000000000001903

Source DB:  PubMed          Journal:  Shock        ISSN: 1073-2322            Impact factor:   3.533


  24 in total

Review 1.  Cytopathic hypoxia. Is oxygen use impaired in sepsis as a result of an acquired intrinsic derangement in cellular respiration?

Authors:  Mitchell P Fink
Journal:  Crit Care Clin       Date:  2002-01       Impact factor: 3.598

2.  Mitochondrial Dysfunction is Associated With an Immune Paralysis Phenotype in Pediatric Sepsis.

Authors:  Scott L Weiss; Donglan Zhang; Jenny Bush; Kathryn Graham; Jonathan Starr; Jennifer Murray; Florin Tuluc; Sarah Henrickson; Clifford S Deutschman; Lance Becker; Francis X McGowan; Douglas C Wallace
Journal:  Shock       Date:  2020-09       Impact factor: 3.454

Review 3.  MITOCHONDRIAL FUNCTION IN SEPSIS.

Authors:  Nishkantha Arulkumaran; Clifford S Deutschman; Michael R Pinsky; Brian Zuckerbraun; Paul T Schumacker; Hernando Gomez; Alonso Gomez; Patrick Murray; John A Kellum
Journal:  Shock       Date:  2016-03       Impact factor: 3.454

4.  Cutaneous mitochondrial respirometry: non-invasive monitoring of mitochondrial function.

Authors:  Floor A Harms; Sander I A Bodmer; Nicolaas J H Raat; Egbert G Mik
Journal:  J Clin Monit Comput       Date:  2014-11-12       Impact factor: 2.502

5.  Mitochondrial membrane potential and apoptosis peripheral blood monocytes in severe human sepsis.

Authors:  C Adrie; M Bachelet; M Vayssier-Taussat; F Russo-Marie; I Bouchaert; M Adib-Conquy; J M Cavaillon; M R Pinsky; J F Dhainaut; B S Polla
Journal:  Am J Respir Crit Care Med       Date:  2001-08-01       Impact factor: 21.405

6.  Association between mitochondrial dysfunction and severity and outcome of septic shock.

Authors:  David Brealey; Michael Brand; Iain Hargreaves; Simon Heales; John Land; Ryszard Smolenski; Nathan A Davies; Chris E Cooper; Mervyn Singer
Journal:  Lancet       Date:  2002-07-20       Impact factor: 79.321

7.  Hemorrhagic shock and resuscitation are associated with peripheral blood mononuclear cell mitochondrial dysfunction and immunosuppression.

Authors:  Jose Paul Perales Villarroel; Yuxia Guan; Evan Werlin; Mary A Selak; Lance B Becker; Carrie A Sims
Journal:  J Trauma Acute Care Surg       Date:  2013-07       Impact factor: 3.313

Review 8.  Metabolic pathways in immune cell activation and quiescence.

Authors:  Erika L Pearce; Edward J Pearce
Journal:  Immunity       Date:  2013-04-18       Impact factor: 31.745

9.  NADH Fluorescence Lifetime Imaging Microscopy Reveals Selective Mitochondrial Dysfunction in Neurons Overexpressing Alzheimer's Disease-Related Proteins.

Authors:  Moritz A Niederschweiberer; Patrick M Schaefer; Larry N Singh; Ludwig Lausser; Devyani Bhosale; Raphael Hesse; Enrico Calzia; Hans A Kestler; Angelika Rueck; Douglas C Wallace; Bjoern von Einem; Christine A F von Arnim
Journal:  Front Mol Biosci       Date:  2021-06-14

Review 10.  Bench-to-bedside review: Cytopathic hypoxia.

Authors:  Mitchell P Fink
Journal:  Crit Care       Date:  2002-09-12       Impact factor: 9.097

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