Literature DB >> 34896321

Predictors of urinary polycyclic aromatic hydrocarbon metabolites in girls from the San Francisco Bay Area.

Esther M John1, Jocelyn Koo2, Sue A Ingles3, Theresa H Keegan4, Jenny T Nguyen2, Catherine Thomsen5, Mary Beth Terry6, Regina M Santella7, Khue Nguyen8, Beizhan Yan8.   

Abstract

BACKGROUND: Polycyclic aromatic hydrocarbon (PAH) exposures from tobacco smoke, automobile exhaust, grilled or smoked meat and other sources are widespread and are a public health concern, as many are classified as probable carcinogens and suspected endocrine-disrupting chemicals. PAH exposures can be quantified using urinary biomarkers.
METHODS: Seven urinary metabolites of naphthalene, fluorene, phenanthrene, and pyrene were measured in two samples collected from girls aged 6-16 years from the San Francisco Bay Area. We used Spearman correlation coefficients (SCC) to assess correlations among metabolite concentrations (corrected for specific gravity) separately in first (n = 359) and last (N = 349) samples, and to assess consistency of measurements in samples collected up to 72 months apart. Using multivariable linear regression, we assessed variation in mean metabolites across categories of participant characteristics and potential outdoor, indoor, and dietary sources of PAH exposures.
RESULTS: The detection rate of PAH metabolites was high (4 metabolites in ≥98% of first samples; 5 metabolites in ≥95% of last samples). Correlations were moderate to strong between fluorene, phenanthrene and pyrene metabolites (SCC 0.43-0.82), but weaker between naphthalene and the other metabolites (SCC 0.18-0.36). SCC between metabolites in first and last samples ranged from 0.15 to 0.49. When classifying metabolite concentrations into tertiles based on single samples (first or last samples) vs. the average of the two samples, agreement was moderate to substantial (weighted kappa statistics 0.52-0.65). For specific metabolites, concentrations varied by age, race/ethnicity, and body mass index percentile, as well as by outdoor sources (season of sample collection, street traffic), indoor sources (heating with gas, cigarette smoke), and dietary sources (frequent use of grill, consumption of smoked meat or fish) of PAH exposures.
CONCLUSIONS: Urinary PAH exposure was widespread in girls aged 6-16 years and associated with several sources of exposure. Tertile classification of a single urine sample provides reliable PAH exposure ranking.
Copyright © 2021 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Biomarkers; Children; Epidemiology; Polycyclic aromatic hydrocarbons (PAHs); Urinary metabolites

Mesh:

Substances:

Year:  2021        PMID: 34896321      PMCID: PMC8823666          DOI: 10.1016/j.envres.2021.112534

Source DB:  PubMed          Journal:  Environ Res        ISSN: 0013-9351            Impact factor:   6.498


  37 in total

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Journal:  Chem Res Toxicol       Date:  2012-06-13       Impact factor: 3.739

2.  Exposure and kinetics of polycyclic aromatic hydrocarbons (PAHs) in cigarette smokers.

Authors:  Gideon St Helen; Maciej L Goniewicz; Delia Dempsey; Margaret Wilson; Peyton Jacob; Neal L Benowitz
Journal:  Chem Res Toxicol       Date:  2012-03-29       Impact factor: 3.739

3.  Multipathway polycyclic aromatic hydrocarbon and pyrene exposure among children living in Campania (Italy).

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4.  Reference range levels of polycyclic aromatic hydrocarbons in the US population by measurement of urinary monohydroxy metabolites.

Authors:  James Grainger; Wenlin Huang; Donald G Patterson; Wayman E Turner; James Pirkle; Samuel P Caudill; Richard Y Wang; Larry L Needham; Eric J Sampson
Journal:  Environ Res       Date:  2005-10-12       Impact factor: 6.498

Review 5.  Endocrine disrupting chemicals and disease susceptibility.

Authors:  Thaddeus T Schug; Amanda Janesick; Bruce Blumberg; Jerrold J Heindel
Journal:  J Steroid Biochem Mol Biol       Date:  2011-08-27       Impact factor: 4.292

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Authors:  Pernilla Gustafson; Conny Ostman; Gerd Sällsten
Journal:  Environ Sci Technol       Date:  2008-07-15       Impact factor: 9.028

7.  The LEGACY Girls Study: Growth and Development in the Context of Breast Cancer Family History.

Authors:  Esther M John; Mary Beth Terry; Theresa H M Keegan; Angela R Bradbury; Julia A Knight; Wendy K Chung; Caren J Frost; Lothar Lilge; Linda Patrick-Miller; Lisa A Schwartz; Alice S Whittemore; Saundra S Buys; Mary B Daly; Irene L Andrulis
Journal:  Epidemiology       Date:  2016-05       Impact factor: 4.822

8.  Indoor air pollution exposure from use of indoor stoves and fireplaces in association with breast cancer: a case-control study.

Authors:  Alexandra J White; Susan L Teitelbaum; Steven D Stellman; Jan Beyea; Susan E Steck; Irina Mordukhovich; Kathleen M McCarty; Jiyoung Ahn; Pavel Rossner; Regina M Santella; Marilie D Gammon
Journal:  Environ Health       Date:  2014-12-12       Impact factor: 5.984

9.  Pubertal timing and breast cancer risk in the Sister Study cohort.

Authors:  Mandy Goldberg; Aimee A D'Aloisio; Katie M O'Brien; Shanshan Zhao; Dale P Sandler
Journal:  Breast Cancer Res       Date:  2020-10-27       Impact factor: 6.466

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  1 in total

1.  Urinary Biomarkers of Polycyclic Aromatic Hydrocarbons and Timing of Pubertal Development: The California PAH Study.

Authors:  Esther M John; Theresa H Keegan; Mary Beth Terry; Jocelyn Koo; Sue A Ingles; Jenny T Nguyen; Catherine Thomsen; Regina M Santella; Khue Nguyen; Beizhan Yan
Journal:  Epidemiology       Date:  2022-07-27       Impact factor: 4.860

  1 in total

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