Literature DB >> 34865148

Development and external validation of a diagnostic model for biopsy-proven acute interstitial nephritis using electronic health record data.

Dennis G Moledina1, Michael T Eadon2, Frida Calderon1, Yu Yamamoto1, Melissa Shaw1, Mark A Perazella1, Michael Simonov1, Randy Luciano1, Tae-Hwi Schwantes-An2, Gilbert Moeckel3, Michael Kashgarian3, Michael Kuperman4, Wassim Obeid5, Lloyd G Cantley1, Chirag R Parikh5, F Perry Wilson1.   

Abstract

BACKGROUND: Patients with acute interstitial nephritis (AIN) can present without typical clinical features, leading to a delay in diagnosis and treatment. We therefore developed and validated a diagnostic model to identify patients at risk of AIN using variables from the electronic health record.
METHODS: In patients who underwent a kidney biopsy at Yale University between 2013 and 2018, we tested the association of >150 variables with AIN, including demographics, comorbidities, vital signs and laboratory tests (training set 70%). We used least absolute shrinkage and selection operator methodology to select prebiopsy features associated with AIN. We performed area under the receiver operating characteristics curve (AUC) analysis with internal (held-out test set 30%) and external validation (Biopsy Biobank Cohort of Indiana). We tested the change in model performance after the addition of urine biomarkers in the Yale AIN study.
RESULTS: We included 393 patients (AIN 22%) in the training set, 158 patients (AIN 27%) in the test set, 1118 patients (AIN 11%) in the validation set and 265 patients (AIN 11%) in the Yale AIN study. Variables in the selected model included serum creatinine {adjusted odds ratio [aOR] 2.31 [95% confidence interval (CI) 1.42-3.76]}, blood urea nitrogen:creatinine ratio [aOR 0.40 (95% CI 0.20-0.78)] and urine dipstick specific gravity [aOR 0.95 (95% CI 0.91-0.99)] and protein [aOR 0.39 (95% CI 0.23-0.68)]. This model showed an AUC of 0.73 (95% CI 0.64-0.81) in the test set, which was similar to the AUC in the external validation cohort [0.74 (95% CI 0.69-0.79)]. The AUC improved to 0.84 (95% CI 0.76-0.91) upon the addition of urine interleukin-9 and tumor necrosis factor-α.
CONCLUSIONS: We developed and validated a statistical model that showed a modest AUC for AIN diagnosis, which improved upon the addition of urine biomarkers. Future studies could evaluate this model and biomarkers to identify unrecognized cases of AIN.
© The Author(s) 2021. Published by Oxford University Press on behalf of the ERA.

Entities:  

Keywords:  biopsy; creatinine; electronic health record; interstitial nephritis; urinalysis

Mesh:

Substances:

Year:  2022        PMID: 34865148     DOI: 10.1093/ndt/gfab346

Source DB:  PubMed          Journal:  Nephrol Dial Transplant        ISSN: 0931-0509            Impact factor:   7.186


  2 in total

1.  Mortality after acute kidney injury and acute interstitial nephritis in patients prescribed immune checkpoint inhibitor therapy.

Authors:  Megan L Baker; Yu Yamamoto; Mark A Perazella; Nazli Dizman; Anushree C Shirali; Navid Hafez; Jason Weinstein; Michael Simonov; Jeffrey M Testani; Harriet M Kluger; Lloyd G Cantley; Chirag R Parikh; F Perry Wilson; Dennis G Moledina
Journal:  J Immunother Cancer       Date:  2022-03       Impact factor: 12.469

Review 2.  The role of kidney biopsy in immune checkpoint inhibitor nephrotoxicity.

Authors:  Emily M Moss; Mark A Perazella
Journal:  Front Med (Lausanne)       Date:  2022-08-10
  2 in total

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