Literature DB >> 34611295

Placental expression of miR-21-5p, miR-210-3p and miR-141-3p: relation to human fetoplacental growth.

P Kochhar1, P Dwarkanath1, G Ravikumar2, A Thomas3, J Crasta2, T Thomas4, A V Kurpad1, A Mukhopadhyay5.   

Abstract

BACKGROUND/
OBJECTIVES: Dysregulation of microRNAs (miRNAs) and their target genes in placental tissue is associated with foetal growth restriction. We aimed to evaluate associations of placental miR-21-5p, miR-141-3p and miR-210-3p expression with maternal, placental and newborn parameters and with placental expression of their potential target genes PTEN, VEGF, FLT and ENG in a set of well-characterized small- (SGA) and appropriate- (AGA) for gestational age full-term singleton pregnancies. SUBJECTS/
METHODS: Placental samples (n = 80) from 26 SGA and 54 AGA were collected from full-term singleton pregnancies. Placental transcript abundances of miR-21-5p, miR-141-3p and miR-210-3p were assessed after normalization to a reference miRNA, mir-16-5p by real-time quantitative PCR. Placental transcript abundances of PTEN, VEGF, FLT and ENG were assessed after normalizing to a panel of reference genes.
RESULTS: Placental miR-21-5p transcript abundance was negatively associated with placental weight (n = 80, r = -0.222, P = 0.047) and this association was specific to the AGA births (n = 54, r = -0.292, P = 0.032). Placental transcript abundances of miR-210-3p and miR-141-3p were not associated with placental weight or birth weight in all 80 births. However, placental miR-210-3p transcript abundance was positively associated with birth weight specifically in the SGA births (n = 26, r = 0.449, P = 0.021). Placental transcript abundance of miR-21-5p was negatively associated with PTEN transcript abundance (Spearman's ρ = -0.245, P = 0.028) while that of miR-141-3p was positively associated with FLT (Spearman's ρ = 0.261, P = 0.019) and ENG (Spearman's ρ = 0.259, P = 0.020) transcript abundances in all 80 births.
CONCLUSION: We conclude that placental miR-21-5p and miR-210-3p may be involved in fetoplacental growth. However, this regulation is unlikely to be mediated through placental expression of PTEN, VEGF, FLT or ENG.
© 2021. The Author(s), under exclusive licence to Springer Nature Limited.

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Year:  2021        PMID: 34611295     DOI: 10.1038/s41430-021-01017-x

Source DB:  PubMed          Journal:  Eur J Clin Nutr        ISSN: 0954-3007            Impact factor:   4.884


  54 in total

Review 1.  Growth and function of the normal human placenta.

Authors:  Neil M Gude; Claire T Roberts; Bill Kalionis; Roger G King
Journal:  Thromb Res       Date:  2004       Impact factor: 3.944

2.  microRNAs: a newly described class of encoded molecules that play a role in health and disease.

Authors:  K Felekkis; E Touvana; Ch Stefanou; C Deltas
Journal:  Hippokratia       Date:  2010-10       Impact factor: 0.471

3.  Expression profile of C19MC microRNAs in placental tissue in pregnancy-related complications.

Authors:  Ilona Hromadnikova; Katerina Kotlabova; Marketa Ondrackova; Petra Pirkova; Andrea Kestlerova; Veronika Novotna; Lucie Hympanova; Ladislav Krofta
Journal:  DNA Cell Biol       Date:  2015-03-31       Impact factor: 3.311

4.  Characterization of placenta-specific microRNAs in fetal growth restriction pregnancy.

Authors:  Ai Higashijima; Kiyonori Miura; Hiroyuki Mishima; Akira Kinoshita; Ozora Jo; Shuhei Abe; Yuri Hasegawa; Shoko Miura; Kentaro Yamasaki; Atsushi Yoshida; Koh-ichiro Yoshiura; Hideaki Masuzaki
Journal:  Prenat Diagn       Date:  2013-01-27       Impact factor: 3.050

5.  miR-210 targets iron-sulfur cluster scaffold homologue in human trophoblast cell lines: siderosis of interstitial trophoblasts as a novel pathology of preterm preeclampsia and small-for-gestational-age pregnancies.

Authors:  Deug-Chan Lee; Roberto Romero; Jung-Sun Kim; Adi L Tarca; Daniel Montenegro; Beth L Pineles; Ernest Kim; JoonHo Lee; Sun Young Kim; Sorin Draghici; Pooja Mittal; Juan Pedro Kusanovic; Tinnakorn Chaiworapongsa; Sonia S Hassan; Chong Jai Kim
Journal:  Am J Pathol       Date:  2011-08       Impact factor: 4.307

Review 6.  Etiologies of fetal growth restriction.

Authors:  P S Bernstein; M Y Divon
Journal:  Clin Obstet Gynecol       Date:  1997-12       Impact factor: 2.190

7.  Differentially expressed microRNAs and affected biological pathways revealed by modulated modularity clustering (MMC) analysis of human preeclamptic and IUGR placentas.

Authors:  L Guo; S Q Tsai; N E Hardison; A H James; A A Motsinger-Reif; B Thames; E A Stone; C Deng; J A Piedrahita
Journal:  Placenta       Date:  2013-04-30       Impact factor: 3.481

8.  Reduced cystathionine γ-lyase and increased miR-21 expression are associated with increased vascular resistance in growth-restricted pregnancies: hydrogen sulfide as a placental vasodilator.

Authors:  Tereza Cindrova-Davies; Emilio A Herrera; Youguo Niu; John Kingdom; Dino A Giussani; Graham J Burton
Journal:  Am J Pathol       Date:  2013-02-12       Impact factor: 4.307

9.  miR-141 contributes to fetal growth restriction by regulating PLAG1 expression.

Authors:  Qiuqin Tang; Wei Wu; Xia Xu; Lu Huang; Qiong Gao; Huijuan Chen; Hong Sun; Yankai Xia; Jiahao Sha; Xinru Wang; Daozhen Chen; Qian Xu
Journal:  PLoS One       Date:  2013-03-15       Impact factor: 3.240

Review 10.  Small for gestational age and obesity related comorbidities.

Authors:  Yong Hee Hong; Sochung Chung
Journal:  Ann Pediatr Endocrinol Metab       Date:  2018-03-22
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  1 in total

1.  Characteristics of tRNA-Derived Small RNAs and microRNAs Associated with Immunocompromise in an Intrauterine Growth-Restricted Pig Model.

Authors:  Jianfeng Ma; Mailin Gan; Jingyun Chen; Lei Chen; Ye Zhao; Yan Zhu; Lili Niu; Shunhua Zhang; Yanzhi Jiang; Zongyi Guo; Jinyong Wang; Li Zhu; Linyuan Shen
Journal:  Animals (Basel)       Date:  2022-08-17       Impact factor: 3.231

  1 in total

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