| Literature DB >> 34545332 |
Shaolan Zhou1,2, Jing Zhang1,3,4, Pengfei Luan1,3, Zhanbing Ma1,3, Jie Dang1,3, Hong Zhu2, Qian Ma1,3, Yanfeng Wang1,3, Zhenghao Huo1,3,5.
Abstract
[This corrects the article DOI: 10.1155/2021/5547635.].Entities:
Year: 2021 PMID: 34545332 PMCID: PMC8449717 DOI: 10.1155/2021/9818203
Source DB: PubMed Journal: J Immunol Res ISSN: 2314-7156 Impact factor: 4.818
Figure 1miR-183-5p directly targets Foxo1 3′UTR. (a) The binding site of miR-183-5p and the position 226-242 of Foxo1 3′UTR wild type (WT) and mutant type (mut). (b) miR-183-5p ectopic expression significantly inhibited luciferase activity of the wild-type Foxo1 3′UTR reporter plasmid in comparison with the mutated counterpart. Groups labelled with different letters are statistically different from each other (∗p < 0.05). Differences between groups were analyzed for statistical significance by ANOVA with Fischer's probable least-square difference post hoc test.