Literature DB >> 34515147

Downregulation of Arntl mRNA Expression in Women with Hypertension: A Case-Control Study.

Zhengmei Fang1, Lijun Zhu1, Yuelong Jin1, Yan Chen1, Weiwei Chang1, Yingshui Yao1,2.   

Abstract

BACKGROUND: Previous studies have reported that disturbance of endogenous circadian rhythms enhances the chance of hypertension and suggested that circadian clock genes could have a crucial function in the onset of the disease. This case-control study was aimed to investigate the association of the mRNA expression of aryl hydrocarbon receptor nuclear translocator like (Arntl), clock circadian regulator (Clock), and period circadian regulators 1 and 2 (Per1 and Per2) with hypertension and blood pressure levels.
METHODS: A total of 172 subjects were recruited in this study, including 86 hypertension and 86 nonhypertension controls. The mRNA expression levels in peripheral blood mononuclear cells were determined by real-time quantitative polymerase chain reaction. The differences in Arntl, Clock, Per1, and Per2 mRNA expression were compared between the 2 groups, and the relationship between mRNA expression and cardiometabolic risk profiles was also assessed.
RESULTS: We found that the mRNA expression of Arntl was downregulated in the hypertension cases compared with controls in women (1.10 [0.66, 1.71] vs. 1.30 [0.99, 2.06], p = 0.031). There was a significant negative correlation between the Arntl mRNA expression and SBP (r = -0.301, p = 0.004) and DBP (r = -0.222, p = 0.034) in women. In men, a negative correlation between the Per1 mRNA expression and SBP (r = -0.247, p = 0.026) was found.
CONCLUSIONS: The Arntl mRNA expression may play an important role in progression of hypertension in women.
© 2021 The Author(s) Published by S. Karger AG, Basel.

Entities:  

Keywords:  Aryl hydrocarbon receptor nuclear translocator like; Clock circadian regulator; Essential hypertension; Period circadian regulator 1; Period circadian regulator 2

Mesh:

Substances:

Year:  2021        PMID: 34515147      PMCID: PMC8743905          DOI: 10.1159/000518669

Source DB:  PubMed          Journal:  Kidney Blood Press Res        ISSN: 1420-4096            Impact factor:   2.687


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