Literature DB >> 34372921

Adipose-derived stem cells alleviate radiation-induced dermatitis by suppressing apoptosis and downregulating cathepsin F expression.

Chaoling Yao1, Yue Zhou2, Hui Wang2, Feiyan Deng3, Yongyi Chen4, Xiaomei Zhu4, Yu Kong5, Lijun Pan5, Lei Xue6, Xiao Zhou1, Chunmeng Shi7, Xiaowu Sheng8.   

Abstract

BACKGROUND: Radiation-induced dermatitis is a serious side effect of radiotherapy, and very few effective treatments are currently available for this condition. We previously demonstrated that apoptosis is an important feature of radiation-induced dermatitis and adipose-derived stem cells (ADSCs) are one of the most promising types of stem cells that have a protective effect on acute radiation-induced dermatitis. Cathepsin F (CTSF) is a recently discovered protein that plays an important role in apoptosis. In this study, we investigated whether ADSCs affect chronic radiation-induced dermatitis, and the underlying mechanisms involved.
METHODS: ADSCs were isolated from male Sprague-Dawley (SD) rats and characterized. For in vivo studies, rats were randomly divided into control and ADSC-treated groups, and cultured ADSCs were transplanted into radiation-induced dermatitis model rats. The effects of ADSC transplantation were determined by skin damage scoring, histopathological analysis, electron microscopy, immunohistochemical staining, and western blotting analysis of apoptosis-related proteins. To evaluate the effects of ADSCs in vitro, radiation-induced apoptotic cells were treated with ADSC culture supernatant, and apoptosis-related protein expression was investigated by TUNEL staining, flow cytometry, and western blotting.
RESULTS: In the in vivo studies, skin damage, inflammation, fibrosis, and apoptosis were reduced and hair follicle and sebaceous gland regeneration were enhanced in the ADSC group compared with the control group. Further, CTSF and downstream pro-apoptotic proteins (Bid, BAX, and caspase 9) were downregulated, while anti-apoptotic proteins (Bcl-2 and Bcl-XL) were upregulated. In vitro, ADSCs markedly attenuated radiation-induced apoptosis, downregulated CTSF and downstream pro-apoptotic proteins, and upregulated anti-apoptotic proteins.
CONCLUSION: ADSCs protect against radiation-induced dermatitis by exerting an anti-apoptotic effect through inhibition of CTSF expression. ADSCs may be a good therapeutic candidate to prevent the development of radiation-induced dermatitis.
© 2021. The Author(s).

Entities:  

Keywords:  Adipose-derived stem cells; Apoptosis; Cathepsin F; Radiation-induced dermatitis

Year:  2021        PMID: 34372921     DOI: 10.1186/s13287-021-02516-1

Source DB:  PubMed          Journal:  Stem Cell Res Ther        ISSN: 1757-6512            Impact factor:   6.832


  36 in total

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Authors:  Fanni Hegedus; Laju M Mathew; Robert A Schwartz
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7.  Differential contextual responses of normal human breast epithelium to ionizing radiation in a mouse xenograft model.

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Review 8.  Skin toxicity during breast irradiation: pathophysiology and management.

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10.  Establishment and characterization of a radiation-induced dermatitis rat model.

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Journal:  J Cell Mol Med       Date:  2019-02-28       Impact factor: 5.310

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