Literature DB >> 34350492

Craniocervical abnormalities in osteogenesis imperfecta type V.

K Ludwig1,2, C Seiltgens1,2, A Ibba1,2, N Saran1,3,4, J A Ouellet1,3,4, F Glorieux1,2,4, F Rauch5,6.   

Abstract

Craniocervical abnormalities in osteogenesis imperfecta (OI) such as basilar invagination or cervical kyphosis can cause severe neurological morbidity. These abnormalities may be more frequent in OI type V compared with other OI subtypes of similar disease severity, underlining the importance of screening in this group.
INTRODUCTION: Craniocervical abnormalities in osteogenesis imperfecta (OI) can cause severe neurological morbidity. Although radiological cranial base abnormalities in OI have been well described in the literature, there are limited data on these abnormalities in OI type V and their association with clinical sequelae.
METHODS: A retrospective case series on patients with craniocervical abnormalities in OI type V at our institution.
RESULTS: Craniocervical abnormalities were present in 7 of 37 patients with OI type V (19%). For 5 patients (age at last follow-up: 5 to 26 years; 2 females), sufficient information was available for inclusion in the case series. All had genetically confirmed OI type V. Age range at diagnosis of the craniocervical abnormality was 1 day to 18 years. Basilar invagination was present in 3 patients; 2 had cervical kyphosis. Dysplasia of upper cervical vertebrae or base of skull was seen in 3 patients. The severity of the craniocervical abnormality did not clearly correlate with the severity of the OI phenotype. Three patients required surgical intervention (ages 7, 11, and 26 years) due to compression of the spinal cord or brainstem. Craniocervical abnormalities were detected incidentally or on screening in 3 patients, and only 2 had significant positive findings on neurological examination.
CONCLUSION: A variety of craniocervical abnormalities are seen in OI type V including dysplasia of the cervical vertebrae. These cases highlight the importance of screening patients with OI type V with lateral skull and cervical spine x-rays throughout childhood and after skeletal maturity.
© 2021. International Osteoporosis Foundation and National Osteoporosis Foundation.

Entities:  

Keywords:  Basilar invagination; Cervical spine; Kyphosis; Osteogenesis imperfect

Mesh:

Year:  2021        PMID: 34350492     DOI: 10.1007/s00198-021-06088-x

Source DB:  PubMed          Journal:  Osteoporos Int        ISSN: 0937-941X            Impact factor:   4.507


  18 in total

1.  Cranial base abnormalities in osteogenesis imperfecta: phenotypic and genotypic determinants.

Authors:  Moira S Cheung; Heidi Arponen; Peter Roughley; Michel E Azouz; Francis H Glorieux; Janna Waltimo-Sirén; Frank Rauch
Journal:  J Bone Miner Res       Date:  2011-02       Impact factor: 6.741

2.  DNA sequence analysis in 598 individuals with a clinical diagnosis of osteogenesis imperfecta: diagnostic yield and mutation spectrum.

Authors:  G Bardai; P Moffatt; F H Glorieux; F Rauch
Journal:  Osteoporos Int       Date:  2016-08-11       Impact factor: 4.507

Review 3.  Craniocervical abnormalities in osteogenesis imperfecta: genetic and molecular correlation.

Authors:  D O Sillence
Journal:  Pediatr Radiol       Date:  1994

4.  Skull base abnormalities in osteogenesis imperfecta: a cephalometric evaluation of 54 patients and 108 control volunteers.

Authors:  Outi Kovero; Seppo Pynnönen; Kaija Kuurila-Svahn; Ilkka Kaitila; Janna Waltimo-Sirén
Journal:  J Neurosurg       Date:  2006-09       Impact factor: 5.115

5.  Type V osteogenesis imperfecta: a new form of brittle bone disease.

Authors:  F H Glorieux; F Rauch; H Plotkin; L Ward; R Travers; P Roughley; L Lalic; D F Glorieux; F Fassier; N J Bishop
Journal:  J Bone Miner Res       Date:  2000-09       Impact factor: 6.741

6.  Topological mapping of BRIL reveals a type II orientation and effects of osteogenesis imperfecta mutations on its cellular destination.

Authors:  Alexa Patoine; Marie-Hélène Gaumond; Prashant K Jaiswal; François Fassier; Frank Rauch; Pierre Moffatt
Journal:  J Bone Miner Res       Date:  2014-09       Impact factor: 6.741

7.  Osteogenesis imperfecta type V: marked phenotypic variability despite the presence of the IFITM5 c.-14C>T mutation in all patients.

Authors:  Frank Rauch; Pierre Moffatt; Moira Cheung; Peter Roughley; Liljana Lalic; Allan M Lund; Norman Ramirez; Somayyeh Fahiminiya; Jacek Majewski; Francis H Glorieux
Journal:  J Med Genet       Date:  2013-01       Impact factor: 6.318

8.  A single recurrent mutation in the 5'-UTR of IFITM5 causes osteogenesis imperfecta type V.

Authors:  Tae-Joon Cho; Kyung-Eun Lee; Sook-Kyung Lee; Su Jeong Song; Kyung Jin Kim; Daehyun Jeon; Gene Lee; Ha-Neui Kim; Hye Ran Lee; Hye-Hyun Eom; Zang Hee Lee; Ok-Hwa Kim; Woong-Yang Park; Sung Sup Park; Shiro Ikegawa; Won Joon Yoo; In Ho Choi; Jung-Wook Kim
Journal:  Am J Hum Genet       Date:  2012-08-02       Impact factor: 11.025

Review 9.  Osteogenesis imperfecta.

Authors:  Antonella Forlino; Joan C Marini
Journal:  Lancet       Date:  2015-11-03       Impact factor: 79.321

10.  A mutation in the 5'-UTR of IFITM5 creates an in-frame start codon and causes autosomal-dominant osteogenesis imperfecta type V with hyperplastic callus.

Authors:  Oliver Semler; Lutz Garbes; Katharina Keupp; Daniel Swan; Katharina Zimmermann; Jutta Becker; Sandra Iden; Brunhilde Wirth; Peer Eysel; Friederike Koerber; Eckhard Schoenau; Stefan K Bohlander; Bernd Wollnik; Christian Netzer
Journal:  Am J Hum Genet       Date:  2012-08-02       Impact factor: 11.043

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