Literature DB >> 34255918

Unraveling the substrates of cognitive impairment in multiple sclerosis: A multiparametric structural and functional MRI study.

Lorenzo Conti1, Paolo Preziosa1,2, Alessandro Meani1, Elisabetta Pagani1, Paola Valsasina1, Olga Marchesi1, Carmen Vizzino1, Maria A Rocca1,2,3, Massimo Filippi1,2,4,5,3.   

Abstract

BACKGROUND: Cognitive impairment frequently affects multiple sclerosis (MS) patients. However, its neuroanatomical correlates still need to be fully explored. We investigated the contribution of structural and functional magnetic resonance imaging (MRI) abnormalities in explaining cognitive impairment in MS.
METHODS: Brain dual-echo, diffusion tensor, 3D T1-weighted and resting-state (RS) MRI sequences were acquired from 276 MS patients and 102 healthy controls. Using random forest analysis, the contribution of regional white matter (WM) lesions, WM fractional anisotropy (FA) abnormalities, gray matter (GM) atrophy and RS functional connectivity (FC) alterations to cognitive impairment in MS patients was investigated.
RESULTS: Eighty-four MS patients (30.4%) were cognitively impaired. The best MRI predictors of cognitive impairment (relative importance [%]) (out-of-bag area under the curve [AUC]=0.795) were (a) WM lesions in the right superior longitudinal fasciculus (100%), left anterior thalamic radiation (93.4%), left posterior corona radiata (78.5%), left medial lemniscus (74.2%), left inferior longitudinal fasciculus (70.4%), left optic radiation (68.7%), right middle cerebellar peduncle (60.6%) and right optic radiation (53.5%); (b) decreased FA in the splenium of the corpus callosum (64.3%), left optic radiation (61.0%), body of the corpus callosum (51.9%), and fornix (50.9%); (c) atrophy of the left precuneus (91.4%), right cerebellum crus I (84.4%), right caudate nucleus (78.6%), left thalamus (76.2%) and left supplementary motor area (59.8%). The relevance of these MRI measures in explaining cognitive impairment was confirmed in a cross-validation analysis (AUC=0.765).
CONCLUSIONS: Structural damage in strategic WM and GM regions more than RS FC abnormalities explains cognitive impairment in MS patients. This article is protected by copyright. All rights reserved.

Entities:  

Keywords:  MRI; cognitive impairment; gray matter atrophy; multiple sclerosis; resting state functional connectivity

Year:  2021        PMID: 34255918     DOI: 10.1111/ene.15023

Source DB:  PubMed          Journal:  Eur J Neurol        ISSN: 1351-5101            Impact factor:   6.089


  2 in total

1.  NODDI, diffusion tensor microstructural abnormalities and atrophy of brain white matter and gray matter contribute to cognitive impairment in multiple sclerosis.

Authors:  Paolo Preziosa; Elisabetta Pagani; Alessandro Meani; Olga Marchesi; Lorenzo Conti; Andrea Falini; Maria A Rocca; Massimo Filippi
Journal:  J Neurol       Date:  2022-10-06       Impact factor: 6.682

2.  Somatosensory evoked potentials and their relation to microstructural damage in patients with multiple sclerosis-A whole brain DTI study.

Authors:  Jan Hamann; Barbara Ettrich; Karl Titus Hoffman; Florian Then Bergh; Donald Lobsien
Journal:  Front Neurol       Date:  2022-08-29       Impact factor: 4.086

  2 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.