Literature DB >> 34237250

Cabozantinib for radioiodine-refractory differentiated thyroid cancer (COSMIC-311): a randomised, double-blind, placebo-controlled, phase 3 trial.

Marcia S Brose1, Bruce Robinson2, Steven I Sherman3, Jolanta Krajewska4, Chia-Chi Lin5, Fernanda Vaisman6, Ana O Hoff7, Erika Hitre8, Daniel W Bowles9, Jorge Hernando10, Leonardo Faoro11, Kamalika Banerjee11, Jennifer W Oliver11, Bhumsuk Keam12, Jaume Capdevila10.   

Abstract

BACKGROUND: Patients with radioiodine-refractory differentiated thyroid cancer (DTC) previously treated with vascular endothelial growth factor receptor (VEGFR)-targeted therapy have aggressive disease and no available standard of care. The aim of this study was to evaluate the tyrosine kinase inhibitor cabozantinib in this patient population.
METHODS: In this global, randomised, double-blind, placebo-controlled, phase 3 trial, patients aged 16 years and older with radioiodine-refractory DTC (papillary or follicular and their variants) and an Eastern Cooperative Oncology Group performance status of 0 or 1 were randomly assigned (2:1) to oral cabozantinib (60 mg once daily) or matching placebo, stratified by previous lenvatinib treatment and age. The randomisation scheme used stratified permuted blocks of block size six and an interactive voice-web response system; both patients and investigators were masked to study treatment. Patients must have received previous lenvatinib or sorafenib and progressed during or after treatment with up to two VEGFR tyrosine kinase inhibitors. Patients receiving placebo could cross over to open-label cabozantinib on disease progression confirmed by blinded independent radiology committee (BIRC). The primary endpoints were objective response rate (confirmed response per Response Evaluation Criteria in Solid Tumours [RECIST] version 1.1) in the first 100 randomly assigned patients (objective response rate intention-to-treat [OITT] population) and progression-free survival (time to earlier of disease progression per RECIST version 1.1 or death) in all patients (intention-to-treat [ITT] population), both assessed by BIRC. This report presents the primary objective response rate analysis and a concurrent preplanned interim progression-free survival analysis. The study is registered with ClinicalTrials.gov, NCT03690388, and is no longer enrolling patients.
FINDINGS: Between Feb 27, 2019, and Aug 18, 2020, 227 patients were assessed for eligibility, of whom 187 were enrolled from 164 clinics in 25 countries and randomly assigned to cabozantinib (n=125) or placebo (n=62). At data cutoff (Aug 19, 2020) for the primary objective response rate and interim progression-free survival analyses, median follow-up was 6·2 months (IQR 3·4-9·2) for the ITT population and 8·9 months (7·1-10·5) for the OITT population. An objective response in the OITT population was achieved in ten (15%; 99% CI 5·8-29·3) of 67 patients in the cabozantinib group versus 0 (0%; 0-14·8) of 33 in the placebo (p=0·028) but did not meet the prespecified significance level (α=0·01). At interim analysis, the primary endpoint of progression-free survival was met in the ITT population; cabozantinib showed significant improvement in progression-free survival over placebo: median not reached (96% CI 5·7-not estimable [NE]) versus 1·9 months (1·8-3·6); hazard ratio 0·22 (96% CI 0·13-0·36; p<0·0001). Grade 3 or 4 adverse events occurred in 71 (57%) of 125 patients receiving cabozantinib and 16 (26%) of 62 receiving placebo, the most frequent of which were palmar-plantar erythrodysaesthesia (13 [10%] vs 0), hypertension (11 [9%] vs 2 [3%]), and fatigue (ten [8%] vs 0). Serious treatment-related adverse events occurred in 20 (16%) of 125 patients in the cabozantinib group and one (2%) of 62 in the placebo group. There were no treatment-related deaths.
INTERPRETATION: Our results show that cabozantinib significantly prolongs progression-free survival and might provide a new treatment option for patients with radioiodine-refractory DTC who have no available standard of care. FUNDING: Exelixis.
Copyright © 2021 Elsevier Ltd. All rights reserved.

Entities:  

Year:  2021        PMID: 34237250     DOI: 10.1016/S1470-2045(21)00332-6

Source DB:  PubMed          Journal:  Lancet Oncol        ISSN: 1470-2045            Impact factor:   41.316


  24 in total

Review 1.  Aggressive variants of follicular cell-derived thyroid carcinoma: an overview.

Authors:  Cristina Pizzimenti; Vincenzo Fiorentino; Antonio Ieni; Maurizio Martini; Giovanni Tuccari; Maria Lentini; Guido Fadda
Journal:  Endocrine       Date:  2022-07-21       Impact factor: 3.925

Review 2.  Molecular mechanisms of resistance to kinase inhibitors and redifferentiation in thyroid cancers.

Authors:  Marie-Claude Hofmann; Muthusamy Kunnimalaiyaan; Jennifer R Wang; Naifa L Busaidy; Steven I Sherman; Stephen Y Lai; Mark Zafereo; Maria E Cabanillas
Journal:  Endocr Relat Cancer       Date:  2022-09-14       Impact factor: 5.900

3.  Mitochondrial Respiration Inhibition Suppresses Papillary Thyroid Carcinoma Via PI3K/Akt/FoxO1/Cyclin D1 Pathway.

Authors:  Bojie Chen; Shuwen Lei; Xinlu Yin; Mengjia Fei; Yixin Hu; Yuan Shi; Yanan Xu; Lei Fu
Journal:  Front Oncol       Date:  2022-07-05       Impact factor: 5.738

Review 4.  Small molecule inhibitors targeting the cancers.

Authors:  Gui-Hong Liu; Tao Chen; Xin Zhang; Xue-Lei Ma; Hua-Shan Shi
Journal:  MedComm (2020)       Date:  2022-10-13

5.  Assessment of Treatment Response to Lenvatinib in Thyroid Cancer Monitored by F-18 FDG PET/CT Using PERCIST 1.0, Modified PERCIST and EORTC Criteria-Which One Is Most Suitable?

Authors:  Gundula Rendl; Gregor Schweighofer-Zwink; Stefan Sorko; Hans-Jürgen Gallowitsch; Wolfgang Hitzl; Diana Reisinger; Christian Pirich
Journal:  Cancers (Basel)       Date:  2022-04-07       Impact factor: 6.575

Review 6.  Trial Watch: combination of tyrosine kinase inhibitors (TKIs) and immunotherapy.

Authors:  Adriana Petrazzuolo; M Chiara Maiuri; Laurence Zitvogel; Guido Kroemer; Oliver Kepp
Journal:  Oncoimmunology       Date:  2022-05-26       Impact factor: 7.723

Review 7.  Novel Inhibitor-Based Therapies for Thyroid Cancer-An Update.

Authors:  Maciej Ratajczak; Damian Gaweł; Marlena Godlewska
Journal:  Int J Mol Sci       Date:  2021-10-31       Impact factor: 5.923

Review 8.  State of the Art in the Current Management and Future Directions of Targeted Therapy for Differentiated Thyroid Cancer.

Authors:  Horatiu Silaghi; Vera Lozovanu; Carmen Emanuela Georgescu; Cristina Pop; Bogdana Adriana Nasui; Adriana Florinela Cătoi; Cristina Alina Silaghi
Journal:  Int J Mol Sci       Date:  2022-03-23       Impact factor: 5.923

Review 9.  Kinase-Inhibitors in Iodine-Refractory Differentiated Thyroid Cancer-Focus on Occurrence, Mechanisms, and Management of Treatment-Related Hypertension.

Authors:  Anne Christine Kaae; Michael C Kreissl; Marcus Krüger; Manfred Infanger; Daniela Grimm; Markus Wehland
Journal:  Int J Mol Sci       Date:  2021-11-12       Impact factor: 5.923

10.  In silico Analysis of Publicly Available Transcriptomics Data Identifies Putative Prognostic and Therapeutic Molecular Targets for Papillary Thyroid Carcinoma.

Authors:  Asma Almansoori; Poorna Manasa Bhamidimarri; Riyad Bendardaf; Rifat Hamoudi
Journal:  Int J Gen Med       Date:  2022-03-18
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