Literature DB >> 34157100

High-throughput Molecular Analysis of Pseudohypoparathyroidism 1b Patients Reveals Novel Genetic and Epigenetic Defects.

Jennifer Danzig1, Dong Li2, Suzanne Jan de Beur3, Michael A Levine1.   

Abstract

CONTEXT: Patients with pseudohypoparathyroidism type 1b (PHP1b) show disordered imprinting of the maternal GNAS allele or paternal uniparental disomy (UPD). Genetic deletions in STX16 or in upstream exons of GNAS are present in many familial but not sporadic cases.
OBJECTIVE: Characterization of epigenetic and genetic defects in patients with PHP1b. DESIGN AND PATIENTS: DNA from 84 subjects, including 26 subjects with sporadic PHP1b, 27 affected subjects and 17 unaffected and/or obligate gene carriers from 12 PHP1b families, 11 healthy individuals, and 3 subjects with PHP1a was subjected to quantitative pyrosequencing of GNAS differentially methylated regions (DMRs), microarray analysis, and microsatellite haplotype analysis.
SETTING: Academic medical center. MAIN OUTCOME MEASUREMENTS: Molecular pathology of PHP1b.
RESULTS: Healthy subjects, unaffected family members and obligate carriers of paternal PHP1b alleles, and subjects with PHP1a showed normal methylation of all DMRs. All PHP1b subjects showed loss of methylation (LOM) at the exon A/B DMR. Affected members of 9 PHP1b kindreds showed LOM only at the exon A/B DMR, which was associated with a 3-kb deletion of STX16 exons 4 through 6 in 7 families and a novel deletion of STX16 and adjacent NEPEPL1 in 1 family. A novel NESP deletion was found in 1 of 2 other families with more extensive methylation defects. One sporadic PHP1b had UPD of 20q, 2 had 3-kb STX16 deletions, and 5 had apparent epigenetic mosaicism.
CONCLUSIONS: We found diverse patterns of defective methylation and identified novel or previously known mutations in 9 of 12 PHP1b families.
© The Author(s) 2021. Published by Oxford University Press on behalf of the Endocrine Society. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com.

Entities:  

Keywords:  zzm321990 GNASzzm321990 ; epigenetics; imprinting; parathyroid hormone; pseudohypoparathyroidism

Mesh:

Year:  2021        PMID: 34157100      PMCID: PMC8677598          DOI: 10.1210/clinem/dgab460

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   6.134


  87 in total

1.  A Large Inversion Involving GNAS Exon A/B and All Exons Encoding Gsα Is Associated With Autosomal Dominant Pseudohypoparathyroidism Type Ib (PHP1B).

Authors:  Giedre Grigelioniene; Pasi I Nevalainen; Monica Reyes; Susanne Thiele; Olta Tafaj; Angelo Molinaro; Rieko Takatani; Marja Ala-Houhala; Daniel Nilsson; Jesper Eisfeldt; Anna Lindstrand; Marie-Laure Kottler; Outi Mäkitie; Harald Jüppner
Journal:  J Bone Miner Res       Date:  2017-02-24       Impact factor: 6.741

2.  Bidirectional imprinting of a single gene: GNAS1 encodes maternally, paternally, and biallelically derived proteins.

Authors:  B E Hayward; V Moran; L Strain; D T Bonthron
Journal:  Proc Natl Acad Sci U S A       Date:  1998-12-22       Impact factor: 11.205

3.  Identification of a methylation imprint mark within the mouse Gnas locus.

Authors:  J Liu; S Yu; D Litman; W Chen; L S Weinstein
Journal:  Mol Cell Biol       Date:  2000-08       Impact factor: 4.272

4.  Deletion of the NESP55 differentially methylated region causes loss of maternal GNAS imprints and pseudohypoparathyroidism type Ib.

Authors:  Murat Bastepe; Leopold F Fröhlich; Agnès Linglart; Hilal S Abu-Zahra; Katsuyoshi Tojo; Leanne M Ward; Harald Jüppner
Journal:  Nat Genet       Date:  2004-12-12       Impact factor: 38.330

5.  A GNAS1 imprinting defect in pseudohypoparathyroidism type IB.

Authors:  J Liu; D Litman; M J Rosenberg; S Yu; L G Biesecker; L S Weinstein
Journal:  J Clin Invest       Date:  2000-11       Impact factor: 14.808

Review 6.  GNAS epigenetic defects and pseudohypoparathyroidism: time for a new classification?

Authors:  G Mantovani; F M Elli; A Spada
Journal:  Horm Metab Res       Date:  2012-06-06       Impact factor: 2.936

7.  Autosomal dominant pseudohypoparathyroidism type Ib: a novel inherited deletion ablating STX16 causes loss of imprinting at the A/B DMR.

Authors:  Francesca M Elli; Luisa de Sanctis; Erika Peverelli; Paolo Bordogna; Barbara Pivetta; Gianmaria Miolo; Paolo Beck-Peccoz; Anna Spada; Giovanna Mantovani
Journal:  J Clin Endocrinol Metab       Date:  2014-01-17       Impact factor: 5.958

8.  The pseudohypoparathyroidism type lb locus is linked to a region including GNAS1 at 20q13.3.

Authors:  Suzanne M Jan De Beur; Jeffery R O'Connell; Rita Peila; Justin Cho; Zhichao Deng; Stephen Kam; Michael A Levine
Journal:  J Bone Miner Res       Date:  2003-03       Impact factor: 6.741

Review 9.  Genomic Imprinting and Physiological Processes in Mammals.

Authors:  Valter Tucci; Anthony R Isles; Gavin Kelsey; Anne C Ferguson-Smith
Journal:  Cell       Date:  2019-02-21       Impact factor: 41.582

10.  DNA methylation changes during preimplantation development reveal inter-species differences and reprogramming events at imprinted genes.

Authors:  Elena Ivanova; Sebastian Canovas; Soledad Garcia-Martínez; Raquel Romar; Jordana S Lopes; Dimitrios Rizos; Maria J Sanchez-Calabuig; Felix Krueger; Simon Andrews; Fernando Perez-Sanz; Gavin Kelsey; Pilar Coy
Journal:  Clin Epigenetics       Date:  2020-05-11       Impact factor: 6.551

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  5 in total

1.  Lack of GNAS Remethylation During Oogenesis May Be a Cause of Sporadic Pseudohypoparathyroidism Type Ib.

Authors:  Angelo Milioto; Monica Reyes; Patrick Hanna; Zentaro Kiuchi; Serap Turan; Daniel Zeve; Chhavi Agarwal; Giedre Grigelioniene; Ang Chen; Veronica Mericq; Myrto Frangos; Svetlana Ten; Giovanna Mantovani; Isidro B Salusky; Peter Tebben; Harald Jüppner
Journal:  J Clin Endocrinol Metab       Date:  2022-03-24       Impact factor: 5.958

2.  Targeted Long-Read Sequencing Identifies a Retrotransposon Insertion as a Cause of Altered GNAS Exon A/B Methylation in a Family With Autosomal Dominant Pseudohypoparathyroidism Type 1b (PHP1B).

Authors:  Danny E Miller; Patrick Hanna; Miranda Galey; Monica Reyes; Agnès Linglart; Evan E Eichler; Harald Jüppner
Journal:  J Bone Miner Res       Date:  2022-08-03       Impact factor: 6.390

3.  Novel PTH Gene Mutations Causing Isolated Hypoparathyroidism.

Authors:  Colin P Hawkes; Jamal M Al Jubeh; Dong Li; Susan E Tucker; Tara Rajiyah; Michael A Levine
Journal:  J Clin Endocrinol Metab       Date:  2022-05-17       Impact factor: 6.134

4.  Progression of PTH Resistance in Autosomal Dominant Pseudohypoparathyroidism Type Ib Due to Maternal STX16 Deletions.

Authors:  Zentaro Kiuchi; Monica Reyes; Patrick Hanna; Anu Sharma; Terry DeClue; Robert C Olney; Peter Tebben; Harald Jüppner
Journal:  J Clin Endocrinol Metab       Date:  2022-01-18       Impact factor: 6.134

5.  A novel deletion involving the first GNAS exon encoding Gsα causes PHP1A without methylation changes at exon A/B.

Authors:  Devon Campbell; Monica Reyes; Sare Betul Kaygusuz; Saygın Abali; Tulay Guran; Abdullah Bereket; Masayo Kagami; Serap Turan; Harald Jüppner
Journal:  Bone       Date:  2022-01-29       Impact factor: 4.626

  5 in total

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