Literature DB >> 34111452

Therapeutic options for CTLA-4 insufficiency.

David Egg1, Ina Caroline Rump1, Noriko Mitsuiki1, Jessica Rojas-Restrepo1, Maria-Elena Maccari2, Charlotte Schwab1, Annemarie Gabrysch1, Klaus Warnatz3, Sigune Goldacker3, Virginia Patiño4, Daniel Wolff5, Satoshi Okada6, Seiichi Hayakawa6, Yoshiaki Shikama7, Kenji Kanda8, Kohsuke Imai9, Manabu Sotomatsu10, Makoto Kuwashima11, Takahiro Kamiya12, Tomohiro Morio13, Kazuaki Matsumoto13, Takeshi Mori14, Yuri Yoshimoto15, Ingunn Dybedal16, Maria Kanariou17, Zeynep Yesim Kucuk18, Hugo Chapdelaine19, Lenka Petruzelkova20, Hanns-Martin Lorenz21, Kathleen E Sullivan22, Jennifer Heimall22, Michel Moutschen23, Jiri Litzman24, Mike Recher25, Michael H Albert26, Fabian Hauck26, Suranjith Seneviratne27, Jana Pachlopnik Schmid28, Antonios Kolios29, Gary Unglik30, Christian Klemann2, Scott Snapper31, Lisa Giulino-Roth32, Michael Svaton33, Craig D Platt34, Sophie Hambleton35, Olaf Neth36, Geraldine Gosse37, Steffen Reinsch38, Dirk Holzinger39, Yae-Jean Kim40, Shahrzad Bakhtiar41, Faranaz Atschekzei42, Reinhold Schmidt42, Georgios Sogkas42, Shanmuganathan Chandrakasan43, William Rae44, Beata Derfalvi45, Hanne Vibeke Marquart46, Ahmet Ozen47, Ayca Kiykim47, Elif Karakoc-Aydiner47, Pavlína Králíčková48, Godelieve de Bree49, Dimitra Kiritsi50, Markus G Seidel51, Robin Kobbe52, Jennifer Dantzer53, Laia Alsina54, Thais Armangue55, Vassilios Lougaris56, Philipp Agyeman57, Sofia Nyström58, David Buchbinder59, Peter D Arkwright60, Bodo Grimbacher61.   

Abstract

BACKGROUND: Heterozygous germline mutations in cytotoxic T lymphocyte-associated antigen-4 (CTLA4) impair the immunomodulatory function of regulatory T cells. Affected individuals are prone to life-threatening autoimmune and lymphoproliferative complications. A number of therapeutic options are currently being used with variable effectiveness.
OBJECTIVE: Our aim was to characterize the responsiveness of patients with CTLA-4 insufficiency to specific therapies and provide recommendations for the diagnostic workup and therapy at an organ-specific level.
METHODS: Clinical features, laboratory findings, and response to treatment were reviewed retrospectively in an international cohort of 173 carriers of CTLA4 mutation. Patients were followed between 2014 and 2020 for a total of 2624 months from diagnosis. Clinical manifestations were grouped on the basis of organ-specific involvement. Medication use and response were recorded and evaluated.
RESULTS: Among the 173 CTLA4 mutation carriers, 123 (71%) had been treated for immune complications. Abatacept, rituximab, sirolimus, and corticosteroids ameliorated disease severity, especially in cases of cytopenias and lymphocytic organ infiltration of the gut, lungs, and central nervous system. Immunoglobulin replacement was effective in prevention of infection. Only 4 of 16 patients (25%) with cytopenia who underwent splenectomy had a sustained clinical response. Cure was achieved with stem cell transplantation in 13 of 18 patients (72%). As a result of the aforementioned methods, organ-specific treatment pathways were developed.
CONCLUSION: Systemic immunosuppressants and abatacept may provide partial control but require ongoing administration. Allogeneic hematopoietic stem cell transplantation offers a possible cure for patients with CTLA-4 insufficiency.
Copyright © 2021. Published by Elsevier Inc.

Entities:  

Keywords:  CTLA-4; HSCT; LRBA; abatacept; common variable immunodeficiency; diagnosis; primary immunodeficiency; rituximab; sirolimus; treatment

Mesh:

Substances:

Year:  2021        PMID: 34111452     DOI: 10.1016/j.jaci.2021.04.039

Source DB:  PubMed          Journal:  J Allergy Clin Immunol        ISSN: 0091-6749            Impact factor:   10.793


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