Literature DB >> 34027625

The biologic importance of the vitamin D binding protein polymorphism in pediatric COVID-19 patients.

Marijn M Speeckaert1,2, Reinhart Speeckaert3,4, Joris R Delanghe5.   

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Year:  2021        PMID: 34027625      PMCID: PMC8141358          DOI: 10.1007/s00431-021-04110-2

Source DB:  PubMed          Journal:  Eur J Pediatr        ISSN: 0340-6199            Impact factor:   3.183


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With interest, we read the paper of Bayramoğlu et al. [1], which showed an association between vitamin D deficiency and clinical severity in children and adolescents with COVID-19. Besides the investigated parameters, we would like to highlight the potential influence of vitamin D binding protein (DBP) polymorphism on the reported findings. Being one of the most abundant serum proteins, DBP transports 85–90% of vitamin D metabolites, whereas 10–15% is more loosely bound to albumin and <0.1% circulates in an unbound form. DBP is characterized by three major alleles, determined by two SNPs (rs7041 and rs4588): DBP1F (rs7041-T/rs4588-C), DBP1S (rs7041-G/rs4588-C), and DBP2 (rs7041-T/rs4588-A), yielding six allelic combinations and corresponding phenotypes: 3 homozygous (DBP1F/1F, DBP 1S/1S, DBP 2/2) and 3 heterozygous (DBP 1F/2, DBP 1F/1S, DBP1S/2) [2]. The DBP gene is one of the genes involved in the variation of 25-hydroxy vitamin D (25OHD) concentrations. In several studies with adults and children, minor alleles of rs7041 and rs4588, but also of other DBP SNPs (rs2282679 and rs1155563), have been linked with lower 25OHD concentrations [3]. 25OHD and DBP concentrations are highest in DBP1-1, intermediate in DBP1-2, and lowest in DBP2-2 individuals [2]. In infants, the DBP1S/1S, DBP1S/1F, and DBP1F/1F group showed higher serum 25OHD concentrations from birth onward and a significantly better response to 24-month high-dose supplementation of 30 μg/day vitamin D3 in comparison with the DBP1S/2, DBP 1F/2, and DBP 2/2 group [3]. Moreover, the DBP1 allele frequency has been associated with a lower prevalence and mortality due to a SARS-CoV-2 infection, which could be partly explained by potential protective effects of vitamin D and DBP (actin scavenging, hemotaxis, influence on T cell response, …) [4]. In conclusion, the DBP polymorphism could influence the reported vitamin D concentrations in children with COVID-19 and should be taken into account in future studies.
  4 in total

1.  Genetic Variation of the Vitamin D Binding Protein Affects Vitamin D Status and Response to Supplementation in Infants.

Authors:  Maria Enlund-Cerullo; Laura Koljonen; Elisa Holmlund-Suila; Helena Hauta-Alus; Jenni Rosendahl; Saara Valkama; Otto Helve; Timo Hytinantti; Heli Viljakainen; Sture Andersson; Outi Mäkitie; Minna Pekkinen
Journal:  J Clin Endocrinol Metab       Date:  2019-11-01       Impact factor: 5.958

Review 2.  Vitamin D binding protein: a multifunctional protein of clinical importance.

Authors:  Marijn M Speeckaert; Reinhart Speeckaert; Nanja van Geel; Joris R Delanghe
Journal:  Adv Clin Chem       Date:  2014       Impact factor: 5.394

3.  Vitamin D binding protein polymorphism and COVID-19.

Authors:  Marijn M Speeckaert; Marc L De Buyzere; Joris R Delanghe
Journal:  J Med Virol       Date:  2020-09-28       Impact factor: 2.327

4.  The association between vitamin D levels and the clinical severity and inflammation markers in pediatric COVID-19 patients: single-center experience from a pandemic hospital.

Authors:  Elvan Bayramoğlu; Gülşen Akkoç; Ayşe Ağbaş; Özlem Akgün; Kamer Yurdakul; Hatice Nilgün Selçuk Duru; Murat Elevli
Journal:  Eur J Pediatr       Date:  2021-03-31       Impact factor: 3.183

  4 in total

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