Literature DB >> 33970334

Slow recovery from fast inactivation of Nav1.3 channels: a common gating mechanism shared in sweet- and sour-sensing cells.

Christopher J Lingle1.   

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Year:  2021        PMID： 33970334      PMCID： PMC8240190          DOI： 10.1007/s00424-021-02575-6

Source DB:  PubMed          Journal:  Pflugers Arch        ISSN： 0031-6768            Impact factor:   4.458


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  15 in total

Review 1.  Progress and renewal in gustation: new insights into taste bud development.

Authors:  Linda A Barlow
Journal:  Development       Date:  2015-11-01       Impact factor: 6.868

2.  Fast-onset long-term open-state block of sodium channels by A-type FHFs mediates classical spike accommodation in hippocampal pyramidal neurons.

Authors:  Kumar Venkatesan; Yue Liu; Mitchell Goldfarb
Journal:  J Neurosci       Date:  2014-11-26       Impact factor: 6.167

3.  Long-term inactivation particle for voltage-gated sodium channels.

Authors:  Katarzyna Dover; Sergio Solinas; Egidio D'Angelo; Mitchell Goldfarb
Journal:  J Physiol       Date:  2010-08-02       Impact factor: 5.182

Review 4.  Taste buds: cells, signals and synapses.

Authors:  Stephen D Roper; Nirupa Chaudhari
Journal:  Nat Rev Neurosci       Date:  2017-06-29       Impact factor: 34.870

5.  Nav1.3 sodium channels: rapid repriming and slow closed-state inactivation display quantitative differences after expression in a mammalian cell line and in spinal sensory neurons.

Authors:  T R Cummins; F Aglieco; M Renganathan; R I Herzog; S D Dib-Hajj; S G Waxman
Journal:  J Neurosci       Date:  2001-08-15       Impact factor: 6.167

6.  Sour taste: receptors, cells and circuits.

Authors:  Emily R Liman; Sue C Kinnamon
Journal:  Curr Opin Physiol       Date:  2021-01-13

7.  Nerve injury induces robust allodynia and ectopic discharges in Nav1.3 null mutant mice.

Authors:  Mohammed A Nassar; Mark D Baker; Alessandra Levato; Rachel Ingram; Giovanna Mallucci; Stephen B McMahon; John N Wood
Journal:  Mol Pain       Date:  2006-10-19       Impact factor: 3.395

8.  Sodium channel NaV1.3 is important for enterochromaffin cell excitability and serotonin release.

Authors:  Peter R Strege; Kaitlyn Knutson; Samuel J Eggers; Joyce H Li; Fan Wang; David Linden; Joseph H Szurszewski; Lorin Milescu; Andrew B Leiter; Gianrico Farrugia; Arthur Beyder
Journal:  Sci Rep       Date:  2017-11-15       Impact factor: 4.379

9.  Nav1.3 and FGF14 are primary determinants of the TTX-sensitive sodium current in mouse adrenal chromaffin cells.

Authors:  Pedro L Martinez-Espinosa; Chengtao Yang; Xiao-Ming Xia; Christopher J Lingle
Journal:  J Gen Physiol       Date:  2021-04-05       Impact factor: 4.000

10.  Fast inactivation of Nav1.3 channels by FGF14 proteins: An unconventional way to regulate the slow firing of adrenal chromaffin cells.

Authors:  Emilio Carbone
Journal:  J Gen Physiol       Date:  2021-05-03       Impact factor: 4.086
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