Literature DB >> 33853082

Brain Transcriptome Responses to Dexamethasone Depending on Dose and Sex Reveal Factors Contributing to Sex-Specific Vulnerability to Stress-Induced Disorders.

Eduard Murani1, Nares Trakooljul1, Frieder Hadlich1, Siriluck Ponsuksili1, Klaus Wimmers1.   

Abstract

BACKGROUND: Glucocorticoid (GC) receptor (GR) signaling in the hypothalamus (Hyp) and in the superordinate limbic structures, such as the hippocampus (Hip), conveys feedback regulation of the neuroendocrine stress response and acts upon other neurobiological functions that ultimately influence mental health. These responses are strongly influenced by sex, but the molecular causes are still largely unexplored.
METHODS: To investigate GR targets and their GC sensitivity in the Hyp and Hip, we treated juvenile male and female piglets with 10 (D10) or 60 (D60) µg/kg dexamethasone (DEX), a selective GR agonist, and analyzed transcriptome responses compared to a saline control group using RNA sequencing.
RESULTS: Both doses influenced similar biological functions, including cellular response to lipid and immune cell-related functions, but the transcriptional response to D10 was considerably weaker, particularly in the Hip. Weighted Gene Co-expression Network Analysis revealed a network of genes coordinately regulated by DEX in both structures, among which the alpha-arrestin ARRDC2 takes a central position. Distinct functional groups of genes were differentially regulated by DEX between sexes depending on the dose; at D10, these included particularly mitochondrial genes, whereas at D60 interferon signaling and lipid homeostasis genes were enriched. The general and sex-specific transcriptional responses to DEX highlight microglia as the prominent target. Several key marker genes of disease-associated microglia were regulated by DEX depending on sex, such as TREM2 and LPL.
CONCLUSION: The discovered expression signatures suggest that DEX induced a dysfunctional state of microglia in males, while in females microglia were primed, which could entail predisposition for different mental disorders. The Author(s). Published by S. Karger AG, Basel.

Entities:  

Keywords:  Glucocorticoid receptor; Hypothalamus-pituitary-adrenal axis; Mental disorder; Sex differences; Stress; Transcriptome

Mesh:

Substances:

Year:  2021        PMID: 33853082      PMCID: PMC8985051          DOI: 10.1159/000516500

Source DB:  PubMed          Journal:  Neuroendocrinology        ISSN: 0028-3835            Impact factor:   4.914


  55 in total

Review 1.  How do glucocorticoids influence stress responses? Integrating permissive, suppressive, stimulatory, and preparative actions.

Authors:  R M Sapolsky; L M Romero; A U Munck
Journal:  Endocr Rev       Date:  2000-02       Impact factor: 19.871

2.  Glucocorticoid exposure during hippocampal neurogenesis primes future stress response by inducing changes in DNA methylation.

Authors:  Nadine Provençal; Janine Arloth; Annamaria Cattaneo; Christoph Anacker; Nadia Cattane; Tobias Wiechmann; Simone Röh; Maik Ködel; Torsten Klengel; Darina Czamara; Nikola S Müller; Jari Lahti; Katri Räikkönen; Carmine M Pariante; Elisabeth B Binder
Journal:  Proc Natl Acad Sci U S A       Date:  2019-08-09       Impact factor: 11.205

3.  Opposite microglial activation stages upon loss of PGRN or TREM2 result in reduced cerebral glucose metabolism.

Authors:  Julia K Götzl; Matthias Brendel; Georg Werner; Samira Parhizkar; Laura Sebastian Monasor; Gernot Kleinberger; Alessio-Vittorio Colombo; Maximilian Deussing; Matias Wagner; Juliane Winkelmann; Janine Diehl-Schmid; Johannes Levin; Katrin Fellerer; Anika Reifschneider; Sebastian Bultmann; Peter Bartenstein; Axel Rominger; Sabina Tahirovic; Scott T Smith; Charlotte Madore; Oleg Butovsky; Anja Capell; Christian Haass
Journal:  EMBO Mol Med       Date:  2019-06       Impact factor: 12.137

4.  Malate-aspartate shuttle inhibitor aminooxyacetic acid blocks lipopolysaccharides-induced activation of BV2 microglia.

Authors:  Wangsong Shang; Xunbin Wei; Weihai Ying
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Review 5.  Genetic Approaches to Hypothalamic-Pituitary-Adrenal Axis Regulation.

Authors:  Melinda G Arnett; Lisa M Muglia; Gloria Laryea; Louis J Muglia
Journal:  Neuropsychopharmacology       Date:  2015-07-20       Impact factor: 7.853

Review 6.  Microglia: Neuroimmune-sensors of stress.

Authors:  Matthew G Frank; Laura K Fonken; Linda R Watkins; Steven F Maier
Journal:  Semin Cell Dev Biol       Date:  2019-01-09       Impact factor: 7.727

Review 7.  Brain microglia in psychiatric disorders.

Authors:  Valeria Mondelli; Anthony C Vernon; Federico Turkheimer; Paola Dazzan; Carmine M Pariante
Journal:  Lancet Psychiatry       Date:  2017-04-25       Impact factor: 77.056

8.  Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2.

Authors:  Michael I Love; Wolfgang Huber; Simon Anders
Journal:  Genome Biol       Date:  2014       Impact factor: 13.583

9.  Stress-induced microglial activation occurs through β-adrenergic receptor: noradrenaline as a key neurotransmitter in microglial activation.

Authors:  Shuei Sugama; Takato Takenouchi; Makoto Hashimoto; Hisayuki Ohata; Yasuhiro Takenaka; Yoshihiko Kakinuma
Journal:  J Neuroinflammation       Date:  2019-12-17       Impact factor: 8.322

10.  TREM2 deficiency attenuates neuroinflammation and protects against neurodegeneration in a mouse model of tauopathy.

Authors:  Cheryl E G Leyns; Jason D Ulrich; Mary B Finn; Floy R Stewart; Lauren J Koscal; Javier Remolina Serrano; Grace O Robinson; Elise Anderson; Marco Colonna; David M Holtzman
Journal:  Proc Natl Acad Sci U S A       Date:  2017-10-09       Impact factor: 11.205

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  2 in total

Review 1.  α-Arrestins and Their Functions: From Yeast to Human Health.

Authors:  Kacper Zbieralski; Donata Wawrzycka
Journal:  Int J Mol Sci       Date:  2022-04-30       Impact factor: 6.208

2.  Wnt-Signaling Regulated by Glucocorticoid-Induced miRNAs.

Authors:  Henriett Butz; Katalin Mészáros; István Likó; Attila Patocs
Journal:  Int J Mol Sci       Date:  2021-10-29       Impact factor: 5.923

  2 in total

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