Literature DB >> 33834579

High turnover of types III and VI collagen in progressive idiopathic pulmonary fibrosis.

Nils Hoyer1, Henrik Jessen2, Thomas S Prior3, Jannie M B Sand2, Diana J Leeming2, Morten A Karsdal2, Emilia K A Åttingsberg1, Gustav K M Vangsgaard1, Elisabeth Bendstrup3, Saher B Shaker1.   

Abstract

BACKGROUND AND
OBJECTIVE: Prediction of idiopathic pulmonary fibrosis (IPF) progression is vital for the choice and timing of treatment and patient follow-up. This could potentially be achieved by prognostic blood biomarkers of extracellular matrix (ECM) remodelling.
METHODS: Neoepitope biomarkers of types III and VI collagen turnover (C3M, C6M, PRO-C3 and PRO-C6) were measured in 185 patients with newly diagnosed IPF. Disease severity at baseline and progression over 6 months was assessed by lung function tests and 6-min walk tests. All-cause mortality was assessed over a 3-year follow-up period.
RESULTS: High baseline levels of C3M, C6M, PRO-C3 and PRO-C6 were associated with more advanced disease at the time of diagnosis. Baseline levels of C6M and PRO-C3 were also associated with mortality over 3 years of follow-up (hazard ratio [HR]: 2.3, 95% CI: 1.3-3.9, p = 0.002 and HR: 1.8, 95% CI: 1.1-3.0, p = 0.03). Patients with several increased biomarkers at baseline, representing a high ECM remodelling phenotype, had more advanced disease at baseline, higher risk of progression or death at 6 months (OR: 1.4, 95% CI: 1.1-1.8, p = 0.002) and higher mortality over 3 years of follow-up (HR: 2.4, 95% CI: 1.3-4.5, p = 0.007).
CONCLUSION: Blood biomarkers of types III and VI collagen turnover, assessed at the time of diagnosis, are associated with several indices of disease severity, short-term progression and long-term mortality. These biomarkers can help to identify patients with a high ECM remodelling phenotype at high risk of disease progression and death.
© 2021 Asian Pacific Society of Respirology.

Entities:  

Keywords:  biomarkers; cohort study; collagen; extracellular matrix; idiopathic pulmonary fibrosis; prognosis

Year:  2021        PMID: 33834579     DOI: 10.1111/resp.14056

Source DB:  PubMed          Journal:  Respirology        ISSN: 1323-7799            Impact factor:   6.424


  4 in total

1.  LPA1 antagonist BMS-986020 changes collagen dynamics and exerts antifibrotic effects in vitro and in patients with idiopathic pulmonary fibrosis.

Authors:  Benjamin E Decato; Diana Julie Leeming; Jannie Marie Bülow Sand; Aryeh Fischer; Shuyan Du; Scott M Palmer; Morten Karsdal; Yi Luo; Anne Minnich
Journal:  Respir Res       Date:  2022-03-18

2.  Nintedanib modulates type III collagen turnover in viable precision-cut lung slices from bleomycin-treated rats and patients with pulmonary fibrosis.

Authors:  Christina Hesse; Valerie Beneke; Sebastian Konzok; Claudia Diefenbach; Jannie Marie Bülow Sand; Sarah Rank Rønnow; Morten Asser Karsdal; Danny Jonigk; Katherina Sewald; Armin Braun; Diana Julie Leeming; Lutz Wollin
Journal:  Respir Res       Date:  2022-08-04

Review 3.  Contemporary Concise Review 2021: Interstitial lung disease.

Authors:  Cormac McCarthy; Michael P Keane
Journal:  Respirology       Date:  2022-05-05       Impact factor: 6.175

4.  Turnover of type I and III collagen predicts progression of idiopathic pulmonary fibrosis.

Authors:  H Jessen; N Hoyer; T S Prior; P Frederiksen; M A Karsdal; D J Leeming; E Bendstrup; J M B Sand; S B Shaker
Journal:  Respir Res       Date:  2021-07-15
  4 in total

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