Literature DB >> 33763482

Upregulation of CDC7 Associated with Cervical Cancer Incidence and Development.

Qingwei Wang1, Weiping Zheng1.   

Abstract

BACKGROUND: Cervical cancer is a common malignant tumor of women. Using integrated bioinformatics, this study identified key disease-causing genes in cervical cancer that may provide effective biomarkers or therapeutic targets for early diagnosis and treatment.
RESULTS: We used high-throughput sequencing data from the Gene Expression Omnibus (GEO) to identify new cervical cancer biomarkers. The GSE63678 dataset was downloaded. The data was analyzed via bioinformatics methods, and 61 differentially expressed genes were obtained. These differential genes were analyzed by the Gene Ontology (GO) and Kyoto Encyclopedia of Genes and Genomes (KEGG) pathway enrichments analyses. GO analysis demonstrated that the basic biological functions of differential genes were mostly regulating cell division, mitotic nuclear division, and immune response. Analysis of the KEGG pathway showed the primary involved in the cell cycle, p53 signaling pathway, and cytokine-cytokine receptor interactions. Using TCGA database to query differential expression of differential genes in cervical cancer, the CDC7 gene was found to be highly expressed. In silico analysis of protein interactions using the STRING database revealed that CDC7 interacts with many proteins. These findings were then validated in vitro with immunohistochemistry and qRt-PCR to confirm that CDC7 is highly expressed in cervical cancer tissues. Cell function tests demonstrated that inhibition of CDC7 expression could inhibit the proliferation and migration of cervical cancer HeLa and SiHa cells and promote apoptosis.
CONCLUSION: With comprehensive bioinformatics combined with clinical and cellular function analysis, CDC7 is important to the development of cervical cancer. Targeting of this biomarker may improve the early diagnosis and treatment of cervical cancer.
Copyright © 2021 Qingwei Wang and Weiping Zheng.

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Year:  2021        PMID: 33763482      PMCID: PMC7952153          DOI: 10.1155/2021/6663367

Source DB:  PubMed          Journal:  Biomed Res Int            Impact factor:   3.411


  23 in total

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Journal:  Genes Dev       Date:  2010-06-15       Impact factor: 11.361

2.  Cdc7 is an active kinase in human cancer cells undergoing replication stress.

Authors:  Pierluigi Tenca; Deborah Brotherton; Alessia Montagnoli; Sonia Rainoldi; Clara Albanese; Corrado Santocanale
Journal:  J Biol Chem       Date:  2006-10-24       Impact factor: 5.157

3.  ATR-dependent activation of p38 MAP kinase is responsible for apoptotic cell death in cells depleted of Cdc7.

Authors:  Jun-Sub Im; Joon-Kyu Lee
Journal:  J Biol Chem       Date:  2008-07-14       Impact factor: 5.157

4.  Dual Inhibition of Cdc7 and Cdk9 by PHA-767491 Suppresses Hepatocarcinoma Synergistically with 5-Fluorouracil.

Authors:  Wei Li; Xiao-Le Zhao; Shi-Qiang Shang; Hong-Qiang Shen; Xi Chen
Journal:  Curr Cancer Drug Targets       Date:  2015       Impact factor: 3.428

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6.  Cdc7 inhibition reveals a p53-dependent replication checkpoint that is defective in cancer cells.

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Journal:  Cancer Res       Date:  2004-10-01       Impact factor: 12.701

7.  p53 gain-of-function mutations increase Cdc7-dependent replication initiation.

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Journal:  Diagn Pathol       Date:  2015-07-25       Impact factor: 2.644

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Journal:  Oncotarget       Date:  2016-06-21

10.  DAVID Knowledgebase: a gene-centered database integrating heterogeneous gene annotation resources to facilitate high-throughput gene functional analysis.

Authors:  Brad T Sherman; Da Wei Huang; Qina Tan; Yongjian Guo; Stephan Bour; David Liu; Robert Stephens; Michael W Baseler; H Clifford Lane; Richard A Lempicki
Journal:  BMC Bioinformatics       Date:  2007-11-02       Impact factor: 3.169

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  1 in total

1.  Evaluation of the Synergistic Potential of Simultaneous Pan- or Isoform-Specific BET and SYK Inhibition in B-Cell Lymphoma: An In Vitro Approach.

Authors:  Sina Sender; Ahmad Wael Sultan; Daniel Palmer; Dirk Koczan; Anett Sekora; Julia Beck; Ekkehard Schuetz; Leila Taher; Bertram Brenig; Georg Fuellen; Christian Junghanss; Hugo Murua Escobar
Journal:  Cancers (Basel)       Date:  2022-09-27       Impact factor: 6.575

  1 in total

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