| Literature DB >> 33746454 |
Divya C Ragate1, Sunil Taneja2, Akash Roy2, Ajay K Duseja2, Radha K Dhiman2, Virendra Singh2.
Abstract
Hypercalcemia is a rare metabolic abnormality seen in patients with cirrhosis and is usually considered a paraneoplastic manifestation of hepatocellular carcinoma. Idiopathic hypercalcemia in cirrhosis is a diagnosis of exclusion, which is considered when all the causes of hypercalcemia have been ruled out. Here, we report a rare case of idiopathic hypercalcemia presenting as acute kidney injury in a case of decompensated cirrhosis, managed with adequate hydration and injection of ibandronate and intranasal calcitonin, leading to the normalization of serum calcium and resolution of acute kidney injury.Entities:
Keywords: AFP, Alpha Fetoprotein; ALT, Alanine Transaminase; AST, Aspartate Transaminase; CEA, Carcinoembryonic Antigen; INR, International Normalization Ratio; PET, Positron Emission Technology; PSA, Prostate-Specific Antigen; PTH rp, Parathyroid hormone-related Peptide; PTH, Parathyroid Hormone; PTI, Prothrombin Index; SAP, Serum Alkaline Phosphatase; SFLC, Serum Free Light Chain Assay; SIFE, Serum Immunofixation; SPEP, Serum Protein Electrophoresis; TPCT, Triple Phase CT; UPEP, Urine Protein Electrophoresis; cirrhosis; diagnosis; idiopathic hypercalcemia
Year: 2020 PMID： 33746454 PMCID： PMC7953001 DOI： 10.1016/j.jceh.2020.05.004
Source DB: PubMed Journal: J Clin Exp Hepatol ISSN： 0973-6883