Literature DB >> 33738103

Hymenolepis diminuta-based helminth therapy in C3(1)-TAg mice does not alter breast tumor onset or progression.

Scott Sauer1, Dylan Beinart1, Sade M B Finn1, Sereena L Kumar1, Qing Cheng1, Shelley E Hwang1, William Parker1, Gayathri R Devi1.   

Abstract

BACKGROUND AND OBJECTIVES: An individual's risk of breast cancer is profoundly affected by evolutionary mismatch. Mismatches in Western society known to increase the risk of breast cancer include a sedentary lifestyle and reproductive factors. Biota alteration, characterized by a loss of biodiversity from the ecosystem of the human body as a result of Western society, is a mismatch known to increase the risk of a variety of inflammation-related diseases, including colitis-associated colon cancer. However, the effect of biota alteration on breast cancer has not been evaluated.
METHODOLOGY: In this study, we utilized the C3(1)-TAg mouse model of breast cancer to evaluate the role of biota alteration in the development of breast cancer. This model has been used to recapitulate the role of exercise and pregnancy in reducing the risk of breast cancer. C3(1)-TAg mice were treated with Hymenolepis diminuta, a benign helminth that has been shown to reverse the effects of biota alteration in animal models.
RESULTS: No effect of the helminth H. diminuta was observed. Neither the latency nor tumor growth was affected by the therapy, and no significant effects on tumor transcriptome were observed based on RNAseq analysis. CONCLUSIONS AND IMPLICATIONS: These findings suggest that biota alteration, although known to affect a variety of Western-associated diseases, might not be a significant factor in the high rate of breast cancer observed in Western societies. LAY
SUMMARY: An almost complete loss of intestinal worms in high-income countries has led to increases in allergic disorders, autoimmune conditions, and perhaps colon cancer. However, in this study, results using laboratory mice suggest that loss of intestinal worms might not be associated with breast cancer.
© The Author(s) 2021. Published by Oxford University Press on behalf of the Foundation for Evolution, Medicine, and Public Health.

Entities:  

Keywords:  breast cancer; evolutionary mismatch; helminth therapy; hygiene hypothesis; mouse model

Year:  2021        PMID: 33738103      PMCID: PMC7953836          DOI: 10.1093/emph/eoab007

Source DB:  PubMed          Journal:  Evol Med Public Health        ISSN: 2050-6201


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5.  XIAP Regulation by MNK Links MAPK and NFκB Signaling to Determine an Aggressive Breast Cancer Phenotype.

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Journal:  Cancer Res       Date:  2018-01-19       Impact factor: 12.701

6.  Risk factors for inflammatory breast cancer and other invasive breast cancers.

Authors:  Catherine Schairer; Yan Li; Peter Frawley; Barry I Graubard; Robert D Wellman; Diana S M Buist; Karla Kerlikowske; Tracy L Onega; William F Anderson; Diana L Miglioretti
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Review 7.  Impact of reproductive evolutionary mismatch on women's health and the need for action and research.

Authors:  Mahmoud F Fathalla
Journal:  Int J Gynaecol Obstet       Date:  2018-11-02       Impact factor: 3.561

8.  Consumption of red and processed meat and breast cancer incidence: A systematic review and meta-analysis of prospective studies.

Authors:  Maryam S Farvid; Mariana C Stern; Teresa Norat; Shizuka Sasazuki; Paolo Vineis; Matty P Weijenberg; Alicja Wolk; Kana Wu; Bernard W Stewart; Eunyoung Cho
Journal:  Int J Cancer       Date:  2018-10-03       Impact factor: 7.396

9.  Human papillomavirus infection and risk of breast cancer: a meta-analysis of case-control studies.

Authors:  Jong-Myon Bae; Eun Hee Kim
Journal:  Infect Agent Cancer       Date:  2016-03-14       Impact factor: 2.965

10.  Association between meat consumption and risk of breast cancer: Findings from the Sister Study.

Authors:  Jamie J Lo; Yong-Moon Mark Park; Rashmi Sinha; Dale P Sandler
Journal:  Int J Cancer       Date:  2019-08-06       Impact factor: 7.316

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