Literature DB >> 33691726

Growth in ataxia telangiectasia.

Valerie A I Natale1, Tim J Cole2, Cynthia Rothblum-Oviatt3, Jennifer Wright4, Thomas O Crawford5, Maureen A Lefton-Greif6, Sharon A McGrath-Morrow7,8, Haley Schlechter8, Howard M Lederman4.   

Abstract

BACKGROUND: Ataxia telangiectasia (A-T) is a DNA repair disorder that affects multiple body systems. Neurological problems and immunodeficiency are two important features of this disease. At this time, two main severity groups are defined in A-T: classic (the more severe form) and mild. Poor growth is a common problem in classic A-T. An objective of this study was to develop growth references for classic A-T. Another objective was to compare growth patterns in classic A-T and mild A-T with each other and with the general population, using the CDC growth references. A final objective was to examine the effects of chronic infection on height.
RESULTS: We found that classic A-T patients were smaller overall, and suffered from height and weight faltering that continued throughout childhood and adolescence. When compared to the CDC growth references, the median heights and weights for both male and female patients eventually fell to or below the 3rd centile on the CDC charts. Height faltering was more pronounced in females. Birthweight was lower in the classic A-T group compared to mild A-T and the general population, whereas birth length was not. Finally, we investigated height and BMI faltering in relation to number of infections and found no association.
CONCLUSIONS: Classic A-T appears to affect growth in utero. Although children appear to grow well in very early life, faltering begins early, and is unrelenting.

Entities:  

Keywords:  Ataxia telangiectasia; Growth; Growth charts; Infections

Mesh:

Year:  2021        PMID: 33691726      PMCID: PMC7945359          DOI: 10.1186/s13023-021-01716-5

Source DB:  PubMed          Journal:  Orphanet J Rare Dis        ISSN: 1750-1172            Impact factor:   4.123


  3 in total

1.  Twenty-year follow-up of 160 patients with ataxia-telangiectasia.

Authors:  F Ersoy; A I Berkel; O Sanal; H Oktay
Journal:  Turk J Pediatr       Date:  1991 Oct-Dec       Impact factor: 0.552

2.  The incidence and gene frequency of ataxia-telangiectasia in the United States.

Authors:  M Swift; D Morrell; E Cromartie; A R Chamberlin; M H Skolnick; D T Bishop
Journal:  Am J Hum Genet       Date:  1986-11       Impact factor: 11.025

3.  Genotype, extrapyramidal features, and severity of variant ataxia-telangiectasia.

Authors:  Katherine Schon; Nienke J H van Os; Nicholas Oscroft; Helen Baxendale; Daniel Scoffings; Julian Ray; Mohnish Suri; William P Whitehouse; Puja R Mehta; Natasha Everett; Leonardo Bottolo; Bart P van de Warrenburg; Philip J Byrd; Corry Weemaes; Michel A Willemsen; Marc Tischkowitz; A Malcolm Taylor; Anke E Hensiek
Journal:  Ann Neurol       Date:  2019-02       Impact factor: 10.422

  3 in total
  2 in total

1.  Analysis of Clinical and Genetic Characterization of Three Ataxia-Telangiectasia Pedigrees With Novel ATM Gene Mutations.

Authors:  Peng Huang; Lu Zhang; Li Tang; Yi Ren; Hong Peng; Jie Xiong; Lingjuan Liu; Jie Xu; Yangyang Xiao; Jian Li; Dingan Mao; Liqun Liu
Journal:  Front Pediatr       Date:  2022-05-02       Impact factor: 3.418

2.  Correction to: Growth in ataxia telangiectasia.

Authors:  Valerie A I Natale; Tim J Cole; Cynthia Rothblum-Oviatt; Jennifer Wright; Thomas O Crawford; Maureen A Lefton-Greif; Sharon A McGrath-Morrow; Haley Schlechter; Howard M Lederman
Journal:  Orphanet J Rare Dis       Date:  2021-06-01       Impact factor: 4.123

  2 in total

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