Literature DB >> 33663571

Pseudohypoparathyroidism: application of the Italian common healthcare-pathway for a homogeneous clinical approach and a shared follow up.

Daniele Tessaris1,2, Elisa Bonino3, Giovanna Weber4, Malgorzata Wasniewska5, Domenico Corica5, Marco Pitea4, Giuseppe Scirè6, Manuela Caruso-Nicoletti7, Danilo Fintini6, Luisa de Sanctis3.   

Abstract

BACKGROUND: Pseudohypoparathyroidism (PHP) represents a heterogeneous group of rare endocrine disorders caused by (epi) genetic abnormalities affecting the GNAS locus. It is mainly characterized by resistance to PTH and TSH, and by peculiar clinical features such as short stature, obesity, cognitive impairment, subcutaneous ossifications and brachydactyly. Delayed puberty, GHRH and calcitonin resistances have also been described. The healthcare-pathway recently proposed by the Italian Society of Pediatric Endocrinology and Diabetology (ISPED) has provided a standardized clinical approach to these conditions. The purpose of the present study was to evaluate its application in clinical practice, and to collect data for setting future specific studies.
METHODS: Through a semi-structured survey, based on the indications of the care-pathway, data on PHP clinical management were collected. The compilation of each data in the survey was read as an index of the adoption of the healthcare-pathway in clinical practice.
RESULTS: In addition to the proposing Center, 4 Centers joined the study, thus obtaining a large collection of data on 48 PHP patients. Highest rates in the completion of data were obtained for diagnostic history, auxological measurements and subcutaneous ossifications evaluation. As expected, the availability of data for the other investigated fields was lower, coming from recent research studies. More information has been obtained on hormonal resistance classically involved in PHP (PTH, TSH, GHRH and GnRH) and on cognitive impairment, while a few data has been collected on bone mineral status, calcitonin levels and glucolipid metabolism.
CONCLUSIONS: The presented data show that the ISPED healthcare-pathway could represent a valid tool both to confirm the clinical approach to PHP patients and to allow homogeneous data collection; however, it has not yet been fully adopted. The strengthening of the network among the major Italian Endocrine Centers will contribute to improve its application in clinical practice, optimizing the follow-up of these patients and increasing knowledge on PHP.

Entities:  

Keywords:  GNAS locus; Healthcare pathway; PHP; Pseudohypoparathyroidism; iPPSDs

Year:  2021        PMID: 33663571      PMCID: PMC7934261          DOI: 10.1186/s13052-021-01000-z

Source DB:  PubMed          Journal:  Ital J Pediatr        ISSN: 1720-8424            Impact factor:   2.638


  24 in total

Review 1.  From pseudohypoparathyroidism to inactivating PTH/PTHrP signalling disorder (iPPSD), a novel classification proposed by the EuroPHP network.

Authors:  Susanne Thiele; Giovanna Mantovani; Anne Barlier; Valentina Boldrin; Paolo Bordogna; Luisa De Sanctis; Francesca M Elli; Kathleen Freson; Intza Garin; Virginie Grybek; Patrick Hanna; Benedetta Izzi; Olaf Hiort; Beatriz Lecumberri; Arrate Pereda; Vrinda Saraff; Caroline Silve; Serap Turan; Alessia Usardi; Ralf Werner; Guiomar Perez de Nanclares; Agnès Linglart
Journal:  Eur J Endocrinol       Date:  2016-07-11       Impact factor: 6.664

2.  Progressive Development of PTH Resistance in Patients With Inactivating Mutations on the Maternal Allele of GNAS.

Authors:  Alessia Usardi; Asmaa Mamoune; Elodie Nattes; Jean-Claude Carel; Anya Rothenbuhler; Agnès Linglart
Journal:  J Clin Endocrinol Metab       Date:  2017-06-01       Impact factor: 5.958

3.  Reproductive dysfunction in women with Albright's hereditary osteodystrophy.

Authors:  A B Namnoum; G R Merriam; A M Moses; M A Levine
Journal:  J Clin Endocrinol Metab       Date:  1998-03       Impact factor: 5.958

Review 4.  GNAS epigenetic defects and pseudohypoparathyroidism: time for a new classification?

Authors:  G Mantovani; F M Elli; A Spada
Journal:  Horm Metab Res       Date:  2012-06-06       Impact factor: 2.936

5.  Growth hormone-releasing hormone resistance in pseudohypoparathyroidism type ia: new evidence for imprinting of the Gs alpha gene.

Authors:  Giovanna Mantovani; Mohamad Maghnie; Giovanna Weber; Ernesto De Menis; Valeria Brunelli; Marco Cappa; Paola Loli; Paolo Beck-Peccoz; Anna Spada
Journal:  J Clin Endocrinol Metab       Date:  2003-09       Impact factor: 5.958

Review 6.  An update on the clinical and molecular characteristics of pseudohypoparathyroidism.

Authors:  Michael A Levine
Journal:  Curr Opin Endocrinol Diabetes Obes       Date:  2012-12       Impact factor: 3.243

7.  Cognitive impairment is prevalent in pseudohypoparathyroidism type Ia, but not in pseudopseudohypoparathyroidism: possible cerebral imprinting of Gsalpha.

Authors:  M Mouallem; M Shaharabany; N Weintrob; S Shalitin; N Nagelberg; H Shapira; Z Zadik; Z Farfel
Journal:  Clin Endocrinol (Oxf)       Date:  2007-09-04       Impact factor: 3.478

8.  Pseudohypoparathyroidism: defective excretion of 3',5'-AMP in response to parathyroid hormone.

Authors:  L R Chase; G L Melson; G D Aurbach
Journal:  J Clin Invest       Date:  1969-10       Impact factor: 14.808

9.  Pseudohypoparathyroidism and GNAS epigenetic defects: clinical evaluation of albright hereditary osteodystrophy and molecular analysis in 40 patients.

Authors:  Giovanna Mantovani; Luisa de Sanctis; Anna Maria Barbieri; Francesca M Elli; Valentina Bollati; Valentina Vaira; Pamela Labarile; Sara Bondioni; Erika Peverelli; Andrea G Lania; Paolo Beck-Peccoz; Anna Spada
Journal:  J Clin Endocrinol Metab       Date:  2010-01-08       Impact factor: 5.958

10.  The Prevalence of GNAS Deficiency-Related Diseases in a Large Cohort of Patients Characterized by the EuroPHP Network.

Authors:  Francesca Marta Elli; Agnès Linglart; Intza Garin; Luisa de Sanctis; Paolo Bordogna; Virginie Grybek; Arrate Pereda; Federica Giachero; Elisa Verrua; Patrick Hanna; Giovanna Mantovani; Guiomar Perez de Nanclares
Journal:  J Clin Endocrinol Metab       Date:  2016-07-18       Impact factor: 5.958

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