Literature DB >> 33648569

Towards robust and replicable sex differences in the intrinsic brain function of autism.

Dorothea L Floris1,2, José O A Filho3, Meng-Chuan Lai4,5,6,7,8, Steve Giavasis3, Marianne Oldehinkel2, Maarten Mennes1, Tony Charman9, Julian Tillmann9,10, Guillaume Dumas11,12, Christine Ecker13,14, Flavio Dell'Acqua13,15, Tobias Banaschewski16, Carolin Moessnang17, Simon Baron-Cohen7, Sarah Durston18, Eva Loth13,15, Declan G M Murphy13,15, Jan K Buitelaar1,2,19, Christian F Beckmann1,2,20, Michael P Milham3,21, Adriana Di Martino22.   

Abstract

BACKGROUND: Marked sex differences in autism prevalence accentuate the need to understand the role of biological sex-related factors in autism. Efforts to unravel sex differences in the brain organization of autism have, however, been challenged by the limited availability of female data.
METHODS: We addressed this gap by using a large sample of males and females with autism and neurotypical (NT) control individuals (ABIDE; Autism: 362 males, 82 females; NT: 409 males, 166 females; 7-18 years). Discovery analyses examined main effects of diagnosis, sex and their interaction across five resting-state fMRI (R-fMRI) metrics (voxel-level Z > 3.1, cluster-level P < 0.01, gaussian random field corrected). Secondary analyses assessed the robustness of the results to different pre-processing approaches and their replicability in two independent samples: the EU-AIMS Longitudinal European Autism Project (LEAP) and the Gender Explorations of Neurogenetics and Development to Advance Autism Research.
RESULTS: Discovery analyses in ABIDE revealed significant main effects of diagnosis and sex across the intrinsic functional connectivity of the posterior cingulate cortex, regional homogeneity and voxel-mirrored homotopic connectivity (VMHC) in several cortical regions, largely converging in the default network midline. Sex-by-diagnosis interactions were confined to the dorsolateral occipital cortex, with reduced VMHC in females with autism. All findings were robust to different pre-processing steps. Replicability in independent samples varied by R-fMRI measures and effects with the targeted sex-by-diagnosis interaction being replicated in the larger of the two replication samples-EU-AIMS LEAP. LIMITATIONS: Given the lack of a priori harmonization among the discovery and replication datasets available to date, sample-related variation remained and may have affected replicability.
CONCLUSIONS: Atypical cross-hemispheric interactions are neurobiologically relevant to autism. They likely result from the combination of sex-dependent and sex-independent factors with a differential effect across functional cortical networks. Systematic assessments of the factors contributing to replicability are needed and necessitate coordinated large-scale data collection across studies.

Entities:  

Keywords:  Autism spectrum disorder; Replication; Resting-state functional connectivity; Robustness; Sex differences; Voxel-mirrored homotopic connectivity

Mesh:

Year:  2021        PMID: 33648569      PMCID: PMC7923310          DOI: 10.1186/s13229-021-00415-z

Source DB:  PubMed          Journal:  Mol Autism            Impact factor:   6.476


  111 in total

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