Literature DB >> 33602320

MicroRNAs overexpressed in Crohn's disease and their interactions with mechanisms of epigenetic regulation explain novel aspects of Crohn's disease pathogenesis.

Cecilia Fernández-Ponce1, Roberto Navarro Quiroz2, Anderson Díaz Perez3,4, Gustavo Aroca Martinez3,5, Andrés Cadena Bonfanti3,5, Antonio Acosta Hoyos3, Lorena Gómez Escorcia3,4, Sandra Hernández Agudelo3,5, Christian Orozco Sánchez3, José Villarreal Camacho6, Linda Atencio Ibarra7, Jose Consuegra Machado7, Alberto Espinoza Garavito3, Francisco García-Cózar1, Elkin Navarro Quiroz8,9.   

Abstract

BACKGROUND: In this review, we were interested to identify the wide universe of enzymes associated with epigenetic modifications, whose gene expression is regulated by miRNAs with a high relative abundance in Crohn's disease (CD) affected tissues, with the aim to determine their impact in the pathogenesis and evolution of the disease.
METHODS: We used HMDD and Bibliometrix R-package in order to identify the miRNAs overexpressed in CD. The identified enzymes associated with epigenetic mechanisms and post-translational modifications, regulated by miRNAs upregulated in CD, were analyzed using String v11 database.
RESULTS: We found 190 miRNAs with great abundance in patients with CD, of which 26 miRNAs regulate the gene expression of enzymes known to catalyze epigenetic modifications involved in essentials pathophysiological processes, such as chromatin architecture reorganization, immune response regulation including CD4+ T cells polarization, integrity of gut mucosa, gut microbiota composition and tumorigenesis.
CONCLUSION: The integrated analysis of miRNAs with a high relative abundance in patients with CD showed a combined and superimposed gene expression regulation of enzymes associated with relevant epigenetic mechanisms and that could explain, in part, the pathogenesis of CD.

Entities:  

Keywords:  Crohn’s disease; Epigenetic mechanisms; MicroRNA; Post-translational modifications; T cells

Mesh:

Substances:

Year:  2021        PMID: 33602320      PMCID: PMC7890887          DOI: 10.1186/s13148-021-01022-8

Source DB:  PubMed          Journal:  Clin Epigenetics        ISSN: 1868-7075            Impact factor:   6.551


  86 in total

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4.  Reduced Histone H3 Lysine 9 Methylation Contributes to the Pathogenesis of Latent Autoimmune Diabetes in Adults via Regulation of SUV39H2 and KDM4C.

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5.  Host-derived fecal microRNAs can indicate gut microbiota healthiness and ability to induce inflammation.

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Journal:  Theranostics       Date:  2019-06-09       Impact factor: 11.556

Review 6.  Aberrant DNA methylation profile exacerbates inflammation and neurodegeneration in multiple sclerosis patients.

Authors:  Naiara Celarain; Jordi Tomas-Roig
Journal:  J Neuroinflammation       Date:  2020-01-14       Impact factor: 8.322

7.  KMT2D Deficiency Impairs Super-Enhancers to Confer a Glycolytic Vulnerability in Lung Cancer.

Authors:  Hunain Alam; Ming Tang; Mayinuer Maitituoheti; Shilpa S Dhar; Manish Kumar; Chae Young Han; Chandrashekar R Ambati; Samir B Amin; Bingnan Gu; Tsai-Yu Chen; Yu-Hsi Lin; Jichao Chen; Florian L Muller; Nagireddy Putluri; Elsa R Flores; Francesco J DeMayo; Laura Baseler; Kunal Rai; Min Gyu Lee
Journal:  Cancer Cell       Date:  2020-04-02       Impact factor: 38.585

8.  TET3 inhibits TGF-β1-induced epithelial-mesenchymal transition by demethylating miR-30d precursor gene in ovarian cancer cells.

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10.  Comprehensive analysis of gene expression profiles provides insight into the pathogenesis of Crohn's disease.

Authors:  Lumin Bo; Hongyu Fu; Junchi Yang
Journal:  Mol Med Rep       Date:  2018-07-09       Impact factor: 2.952

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  1 in total

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Authors:  Giacomo Caio; Lisa Lungaro; Fabio Caputo; Eleonora Zoli; Fiorella Giancola; Giuseppe Chiarioni; Roberto De Giorgio; Giorgio Zoli
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  1 in total

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