Literature DB >> 33592069

Gene expression profiling identifies FLT3 mutation-like cases in wild-type FLT3 acute myeloid leukemia.

Adrián Mosquera Orgueira1,2,3, Andrés Peleteiro Raíndo1,2,3, Miguel Cid López1,2,3, Beatriz Antelo Rodríguez1,2,3, José Ángel Díaz Arias1,2, Roi Ferreiro Ferro1,2, Natalia Alonso Vence1,2, Ángeles Bendaña López1,2, Aitor Abuín Blanco1,2, Laura Bao Pérez1,2, Paula Melero Valentín1, Marta Sonia González Pérez1,2, Claudio Cerchione4, Giovanni Martinelli5, Pau Montesinos Fernández6, Manuel Mateo Pérez Encinas1,2,3, José Luis Bello López1,2,3.   

Abstract

BACKGROUND: FLT3 mutation is present in 25-30% of all acute myeloid leukemias (AML), and it is associated with adverse outcome. FLT3 inhibitors have shown improved survival results in AML both as upfront treatment and in relapsed/refractory disease. Curiously, a variable proportion of wild-type FLT3 patients also responded to these drugs.
METHODS: We analyzed 6 different transcriptomic datasets of AML cases. Differential expression between mutated and wild-type FLT3 AMLs was performed with the Wilcoxon-rank sum test. Hierarchical clustering was used to identify FLT3-mutation like AMLs. Finally, enrichment in recurrent mutations was performed with the Fisher's test.
RESULTS: A FLT3 mutation-like gene expression pattern was identified among wild-type FLT3 AMLs. This pattern was highly enriched in NPM1 and DNMT3A mutants, and particularly in combined NPM1/DNMT3A mutants.
CONCLUSIONS: We identified a FLT3 mutation-like gene expression pattern in AML which was highly enriched in NPM1 and DNMT3A mutations. Future analysis about the predictive role of this biomarker among wild-type FLT3 patients treated with FLT3 inhibitors is envisaged.

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Year:  2021        PMID: 33592069      PMCID: PMC7886212          DOI: 10.1371/journal.pone.0247093

Source DB:  PubMed          Journal:  PLoS One        ISSN: 1932-6203            Impact factor:   3.752


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Review 7.  Molecular Mutations and Their Cooccurrences in Cytogenetically Normal Acute Myeloid Leukemia.

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Review 9.  Targeting FLT3 mutations in AML: review of current knowledge and evidence.

Authors:  Naval Daver; Richard F Schlenk; Nigel H Russell; Mark J Levis
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10.  Gilteritinib or Chemotherapy for Relapsed or Refractory FLT3-Mutated AML.

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  2 in total

1.  Correction: Gene expression profiling identifies FLT3 mutation-like cases in wild-type FLT3 acute myeloid leukemia.

Authors:  Adrián Mosquera Orgueira; Andrés Peleteiro Raíndo; Miguel Cid López; Beatriz Antelo Rodríguez; José Ángel Díaz Arias; Roi Ferreiro Ferro; Natalia Alonso Vence; Ángeles Bendaña López; Aitor Abuín Blanco; Laura Bao Pérez; Paula Melero Valentín; Marta Sonia González Pérez; Claudio Cerchione; Giovanni Martinelli; Pau Montesinos Fernández; Manuel Mateo Pérez Encinas; José Luis Bello López
Journal:  PLoS One       Date:  2022-09-15       Impact factor: 3.752

Review 2.  Role of Biomarkers in FLT3 AML.

Authors:  Jiao Wei; Ai-Min Hui
Journal:  Cancers (Basel)       Date:  2022-02-24       Impact factor: 6.639

  2 in total

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