Literature DB >> 33584347

Voltage/Calcium Uncoupling Underlies Sustained Torsade de Pointes Ventricular Tachyarrhythmia in an Experimental Model of Long QT Syndrome.

Herman D Himel1, Michael Cupelli1,2, Mohamed Boutjdir1,2,3, Nabil El-Sherif1,2.   

Abstract

BACKGROUND: Clinical experience showed that the majority of Torsade de Pointes (TdP) ventricular tachyarrhythmia (VT) in patients with long QT syndrome (LQTS) are self-terminating (ST), but the few that are non-self-terminating (NST) are potentially fatal. A paramount issue in clinical arrhythmology is to understand the electrophysiological mechanism of ST vs. NST TdP VT.
METHODS: We investigated the electrophysiological mechanism of ST vs. NST TdP VT in the guinea pig Anthopleurin-A experimental model of LQTS, a close surrogate model of congenital LQT3. We utilized simultaneous optical recordings of membrane voltage (V m ) and intracellular calcium (Ca i ) and a robust analytical method based on spatiotemporal entropy difference (E d ) to investigate the hypothesis that early V m /Ca i uncoupling during TdP VT can play a primary role in perpetuation of VT episodes.
RESULTS: We analyzed a total of 35 episodes of TdP VT from 14 guinea pig surrogate models of LQTS, including 23 ST and 12 NST VTs. E d values for NST VT were significantly higher than E d values for ST VT. Analysis of wave front topology during the early phase of ST VT showed the Ca i wave front following closely V m wave front consistent with a lower degree of E d . In contrast, NST VT was associated with uncoupling of V m /Ca i wave fronts during the first 2 or 3 cycles of VT associated with early wave break propagation pattern.
CONCLUSIONS: Utilizing a robust analytical method we showed that, in comparison to ST TdP VT, NST VT was consistently predated by early uncoupling of V m /Ca i that destabilized wave front propagation and can explain a sustained complex reentrant excitation pattern.
Copyright © 2021 Himel, Cupelli, Boutjdir and El-Sherif.

Entities:  

Keywords:  Torsade de Pointes tachyarrhythmias; long QT syndrome; optical mapping; spatiotemporal entropy; voltage/intracellular calcium uncoupling

Year:  2021        PMID: 33584347      PMCID: PMC7876465          DOI: 10.3389/fphys.2021.617847

Source DB:  PubMed          Journal:  Front Physiol        ISSN: 1664-042X            Impact factor:   4.566


  27 in total

1.  Twisting and Turning to Find an Explanation for Torsades de Pointes.

Authors:  Sachin Nayyar; Andreu Porta-Sánchez; Kumaraswamy Nanthakumar
Journal:  JACC Clin Electrophysiol       Date:  2017-11-06

Review 2.  Microvolt T-wave alternans physiological basis, methods of measurement, and clinical utility--consensus guideline by International Society for Holter and Noninvasive Electrocardiology.

Authors:  Richard L Verrier; Thomas Klingenheben; Marek Malik; Nabil El-Sherif; Derek V Exner; Stefan H Hohnloser; Takanori Ikeda; Juan Pablo Martínez; Sanjiv M Narayan; Tuomo Nieminen; David S Rosenbaum
Journal:  J Am Coll Cardiol       Date:  2011-09-20       Impact factor: 24.094

3.  The electrophysiological mechanism of ventricular arrhythmias in the long QT syndrome. Tridimensional mapping of activation and recovery patterns.

Authors:  N el-Sherif; E B Caref; H Yin; M Restivo
Journal:  Circ Res       Date:  1996-09       Impact factor: 17.367

4.  Enhanced susceptibility for acquired torsade de pointes arrhythmias in the dog with chronic, complete AV block is related to cardiac hypertrophy and electrical remodeling.

Authors:  M A Vos; S H de Groot; S C Verduyn; J van der Zande; H D Leunissen; J P Cleutjens; M van Bilsen; M J Daemen; J J Schreuder; M A Allessie; H J Wellens
Journal:  Circulation       Date:  1998-09-15       Impact factor: 29.690

5.  Electrophysiological mechanism of the characteristic electrocardiographic morphology of torsade de pointes tachyarrhythmias in the long-QT syndrome: detailed analysis of ventricular tridimensional activation patterns.

Authors:  N El-Sherif; M Chinushi; E B Caref; M Restivo
Journal:  Circulation       Date:  1997-12-16       Impact factor: 29.690

6.  Short-Lasting Episodes of Torsade de Pointes in the Chronic Atrioventricular Block Dog Model Have a Focal Mechanism, While Longer-Lasting Episodes Are Maintained by Re-Entry.

Authors:  Nele Vandersickel; Alexandre Bossu; Jan De Neve; Albert Dunnink; Veronique M F Meijborg; Marcel A G van der Heyden; Jet D M Beekman; Jacques M T De Bakker; Marc A Vos; Alexander V Panfilov
Journal:  JACC Clin Electrophysiol       Date:  2017-12-18

7.  Early voltage/calcium uncoupling predestinates the duration of ventricular tachyarrhythmias during ischemia/reperfusion.

Authors:  Herman D Himel; Gil Bub; Yuankun Yue; Nabil El-Sherif
Journal:  Heart Rhythm       Date:  2009-06-27       Impact factor: 6.343

8.  Modification of cardiac Na+ channels by anthopleurin-A: effects on gating and kinetics.

Authors:  J A Wasserstrom; J E Kelly; K N Liberty
Journal:  Pflugers Arch       Date:  1993-06       Impact factor: 3.657

9.  Contrasting effects of ischemia on the kinetics of membrane voltage and intracellular calcium transient underlie electrical alternans.

Authors:  Vikram Lakireddy; Paramdeep Baweja; Asma Syed; Gil Bub; Mohamed Boutjdir; Nabil El-Sherif
Journal:  Am J Physiol Heart Circ Physiol       Date:  2004-09-02       Impact factor: 4.733

10.  Perpetuation of torsade de pointes in heterogeneous hearts: competing foci or re-entry?

Authors:  Nele Vandersickel; Teun P de Boer; Marc A Vos; Alexander V Panfilov
Journal:  J Physiol       Date:  2016-03-04       Impact factor: 5.182

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