Literature DB >> 33532862

Genome-wide analysis identifies novel susceptibility loci for myocardial infarction.

Jaana A Hartiala1, Yi Han1,2, Qiong Jia1,2, James R Hilser1,2, Pin Huang1,2, Janet Gukasyan1,2, William S Schwartzman1,2, Zhiheng Cai1,2, Subarna Biswas1, David-Alexandre Trégouët3, Nicholas L Smith4,5,6, Marcus Seldin7, Calvin Pan8, Margarete Mehrabian9, Aldons J Lusis8,9,10, Peter Bazeley11,12,13, Yan V Sun14,15, Chang Liu14,16, Arshed A Quyyumi16, Markus Scholz17,18, Joachim Thiery18,19, Graciela E Delgado20, Marcus E Kleber20, Winfried März20,21,22, Laurence J Howe23, Folkert W Asselbergs23,24,25, Marion van Vugt25, Georgios J Vlachojannis25, Riyaz S Patel23,26, Leo-Pekka Lyytikäinen27,28,29, Mika Kähönen30,31, Terho Lehtimäki27,28, Tuomo V M Nieminen32, Pekka Kuukasjärvi33, Jari O Laurikka33,34, Xuling Chang35,36, Chew-Kiat Heng35,36, Rong Jiang37, William E Kraus38,39, Elizabeth R Hauser38,40, Jane F Ferguson41, Muredach P Reilly42,43, Kaoru Ito44, Satoshi Koyama44, Yoichiro Kamatani45,46,47, Issei Komuro48, Lindsey K Stolze49, Casey E Romanoski49, Mohammad Daud Khan50,51,52,53, Adam W Turner50,51,52,53, Clint L Miller50,51,52,53, Redouane Aherrahrou50,51, Mete Civelek50,51, Lijiang Ma54, Johan L M Björkegren54,55, S Ram Kumar56, W H Wilson Tang11,12,13, Stanley L Hazen12,13, Hooman Allayee1,2.   

Abstract

AIMS: While most patients with myocardial infarction (MI) have underlying coronary atherosclerosis, not all patients with coronary artery disease (CAD) develop MI. We sought to address the hypothesis that some of the genetic factors which establish atherosclerosis may be distinct from those that predispose to vulnerable plaques and thrombus formation. METHODS AND
RESULTS: We carried out a genome-wide association study for MI in the UK Biobank (n∼472 000), followed by a meta-analysis with summary statistics from the CARDIoGRAMplusC4D Consortium (n∼167 000). Multiple independent replication analyses and functional approaches were used to prioritize loci and evaluate positional candidate genes. Eight novel regions were identified for MI at the genome wide significance level, of which effect sizes at six loci were more robust for MI than for CAD without the presence of MI. Confirmatory evidence for association of a locus on chromosome 1p21.3 harbouring choline-like transporter 3 (SLC44A3) with MI in the context of CAD, but not with coronary atherosclerosis itself, was obtained in Biobank Japan (n∼165 000) and 16 independent angiography-based cohorts (n∼27 000). Follow-up analyses did not reveal association of the SLC44A3 locus with CAD risk factors, biomarkers of coagulation, other thrombotic diseases, or plasma levels of a broad array of metabolites, including choline, trimethylamine N-oxide, and betaine. However, aortic expression of SLC44A3 was increased in carriers of the MI risk allele at chromosome 1p21.3, increased in ischaemic (vs. non-diseased) coronary arteries, up-regulated in human aortic endothelial cells treated with interleukin-1β (vs. vehicle), and associated with smooth muscle cell migration in vitro.
CONCLUSIONS: A large-scale analysis comprising ∼831 000 subjects revealed novel genetic determinants of MI and implicated SLC44A3 in the pathophysiology of vulnerable plaques. Published on behalf of the European Society of Cardiology. All rights reserved.
© The Author(s) 2021. For permissions, please email: journals.permissions@oup.com.

Entities:  

Keywords:  Genetic factors; Genome-wide association study; Meta-analysis; Myocardial infarction; SLC44A3

Mesh:

Year:  2021        PMID: 33532862      PMCID: PMC7936531          DOI: 10.1093/eurheartj/ehaa1040

Source DB:  PubMed          Journal:  Eur Heart J        ISSN: 0195-668X            Impact factor:   29.983


  39 in total

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Authors:  Heribert Schunkert; Jeanette Erdmann; Nilesh J Samani
Journal:  Eur Heart J       Date:  2010-03-10       Impact factor: 29.983

2.  An electric lobe suppressor for a yeast choline transport mutation belongs to a new family of transporter-like proteins.

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3.  Rosuvastatin to prevent vascular events in men and women with elevated C-reactive protein.

Authors:  Paul M Ridker; Eleanor Danielson; Francisco A H Fonseca; Jacques Genest; Antonio M Gotto; John J P Kastelein; Wolfgang Koenig; Peter Libby; Alberto J Lorenzatti; Jean G MacFadyen; Børge G Nordestgaard; James Shepherd; James T Willerson; Robert J Glynn
Journal:  N Engl J Med       Date:  2008-11-09       Impact factor: 91.245

4.  Cardiometabolic risk loci share downstream cis- and trans-gene regulation across tissues and diseases.

Authors:  Oscar Franzén; Raili Ermel; Ariella Cohain; Nicholas K Akers; Antonio Di Narzo; Husain A Talukdar; Hassan Foroughi-Asl; Claudia Giambartolomei; John F Fullard; Katyayani Sukhavasi; Sulev Köks; Li-Ming Gan; Chiara Giannarelli; Jason C Kovacic; Christer Betsholtz; Bojan Losic; Tom Michoel; Ke Hao; Panos Roussos; Josefin Skogsberg; Arno Ruusalepp; Eric E Schadt; Johan L M Björkegren
Journal:  Science       Date:  2016-08-19       Impact factor: 47.728

5.  Fifteen new risk loci for coronary artery disease highlight arterial-wall-specific mechanisms.

Authors:  Joanna M M Howson; Wei Zhao; Daniel R Barnes; Weang-Kee Ho; Robin Young; Dirk S Paul; Lindsay L Waite; Daniel F Freitag; Eric B Fauman; Elias L Salfati; Benjamin B Sun; John D Eicher; Andrew D Johnson; Wayne H H Sheu; Sune F Nielsen; Wei-Yu Lin; Praveen Surendran; Anders Malarstig; Jemma B Wilk; Anne Tybjærg-Hansen; Katrine L Rasmussen; Pia R Kamstrup; Panos Deloukas; Jeanette Erdmann; Sekar Kathiresan; Nilesh J Samani; Heribert Schunkert; Hugh Watkins; Ron Do; Daniel J Rader; Julie A Johnson; Stanley L Hazen; Arshed A Quyyumi; John A Spertus; Carl J Pepine; Nora Franceschini; Anne Justice; Alex P Reiner; Steven Buyske; Lucia A Hindorff; Cara L Carty; Kari E North; Charles Kooperberg; Eric Boerwinkle; Kristin Young; Mariaelisa Graff; Ulrike Peters; Devin Absher; Chao A Hsiung; Wen-Jane Lee; Kent D Taylor; Ying-Hsiang Chen; I-Te Lee; Xiuqing Guo; Ren-Hua Chung; Yi-Jen Hung; Jerome I Rotter; Jyh-Ming J Juang; Thomas Quertermous; Tzung-Dau Wang; Asif Rasheed; Philippe Frossard; Dewan S Alam; Abdulla Al Shafi Majumder; Emanuele Di Angelantonio; Rajiv Chowdhury; Yii-Der Ida Chen; Børge G Nordestgaard; Themistocles L Assimes; John Danesh; Adam S Butterworth; Danish Saleheen
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7.  Rationale and design of the Leipzig (LIFE) Heart Study: phenotyping and cardiovascular characteristics of patients with coronary artery disease.

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8.  Genetic analysis in UK Biobank links insulin resistance and transendothelial migration pathways to coronary artery disease.

Authors:  Derek Klarin; Qiuyu Martin Zhu; Connor A Emdin; Mark Chaffin; Steven Horner; Brian J McMillan; Alison Leed; Michael E Weale; Chris C A Spencer; François Aguet; Ayellet V Segrè; Kristin G Ardlie; Amit V Khera; Virendar K Kaushik; Pradeep Natarajan; Sekar Kathiresan
Journal:  Nat Genet       Date:  2017-07-17       Impact factor: 38.330

9.  Dysfunctional nitric oxide signalling increases risk of myocardial infarction.

Authors:  Jeanette Erdmann; Klaus Stark; Ulrike B Esslinger; Philipp Moritz Rumpf; Doris Koesling; Cor de Wit; Frank J Kaiser; Diana Braunholz; Anja Medack; Marcus Fischer; Martina E Zimmermann; Stephanie Tennstedt; Elisabeth Graf; Sebastian Eck; Zouhair Aherrahrou; Janja Nahrstaedt; Christina Willenborg; Petra Bruse; Ingrid Brænne; Markus M Nöthen; Per Hofmann; Peter S Braund; Evanthia Mergia; Wibke Reinhard; Christof Burgdorf; Stefan Schreiber; Anthony J Balmforth; Alistair S Hall; Lars Bertram; Elisabeth Steinhagen-Thiessen; Shu-Chen Li; Winfried März; Muredach Reilly; Sekar Kathiresan; Ruth McPherson; Ulrich Walter; Jurg Ott; Nilesh J Samani; Tim M Strom; Thomas Meitinger; Christian Hengstenberg; Heribert Schunkert
Journal:  Nature       Date:  2013-11-10       Impact factor: 49.962

Review 10.  Shared genetic susceptibility to ischemic stroke and coronary artery disease: a genome-wide analysis of common variants.

Authors:  Martin Dichgans; Rainer Malik; Inke R König; Jonathan Rosand; Robert Clarke; Solveig Gretarsdottir; Gudmar Thorleifsson; Braxton D Mitchell; Themistocles L Assimes; Christopher Levi; Christopher J O'Donnell; Myriam Fornage; Unnur Thorsteinsdottir; Bruce M Psaty; Christian Hengstenberg; Sudha Seshadri; Jeanette Erdmann; Joshua C Bis; Annette Peters; Giorgio B Boncoraglio; Winfried März; James F Meschia; Sekar Kathiresan; M Arfan Ikram; Ruth McPherson; Kari Stefansson; Cathie Sudlow; Muredach P Reilly; John R Thompson; Pankaj Sharma; Jemma C Hopewell; John C Chambers; Hugh Watkins; Peter M Rothwell; Robert Roberts; Hugh S Markus; Nilesh J Samani; Martin Farrall; Heribert Schunkert
Journal:  Stroke       Date:  2013-11-21       Impact factor: 7.914

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3.  Monogenic and Polygenic Contributions to QTc Prolongation in the Population.

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Review 5.  Common and Rare 5'UTR Variants Altering Upstream Open Reading Frames in Cardiovascular Genomics.

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6.  The year in cardiovascular medicine 2021: acute cardiovascular care and ischaemic heart disease.

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7.  FAM114A1 influences cardiac pathological remodeling by regulating angiotensin II signaling.

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8.  Association of MMP1 and MMP3 haplotypes with myocardial infarction and echocardiographic parameters of the left ventricle.

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Journal:  BMC Cardiovasc Disord       Date:  2021-06-08       Impact factor: 2.298

  10 in total

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