Literature DB >> 33528645

Disease stability and extended dosing under anti-VEGF treatment of exudative age-related macular degeneration (AMD) - a meta-analysis.

Justus G Garweg1,2, Christin Gerhardt3.   

Abstract

PURPOSE: To assess disease stability (absence of intra- and/or subretinal fluid) and the portion of eyes being capable to extend their treatment interval to ≥ 12 weeks in exudative age-related macular degeneration (AMD).
METHODS: A systematic literature search was performed in NCBI, PubMed, CENTRAL, and ClinicalTrials.gov to identify clinical studies reporting treatment outcomes for ranibizumab, aflibercept, and brolucizumab in exudative AMD under a treat-and-extend protocol and a follow-up of ≥ 12 months. Weighted mean differences and subgroup comparisons were used to integrate the different studies.
RESULTS: This meta-analysis refers to 29 published series, including 27 independent samples and 5629 patients. In the pooled group, disease stability was reported in 62.9% and 56.0%, respectively, after 12 and 24 months of treatment, whereas treatment intervals were extended to ≥ 12 weeks in 37.7% and 42.6%, respectively. Ranibizumab, aflibercept, and brolucizumab differed regarding their potential to achieve disease stability (56.3%, 64.5%, and 71.5% after 12, and 50.0%, 52.7% and 75.7% after 24 months; p = < 0.001) and to allow an interval extension to ≥ 12 weeks (28.6%, 34.2%, and 53.3% after 12, and 34.2%, 47.7%, and 41.7% after 24 months; p = < 0.001).
CONCLUSION: The portion of eyes achieving disease stability regressed in the second year, whereas the portion of eyes under a ≥ 12-week interval increased. This discrepancy may reflect the challenges in balancing between under-treatment and a reduced treatment burden.
© 2021. The Author(s).

Entities:  

Keywords:  Aflibercept; Age-related macular degeneration (AMD); Brolucizumab; Neovascular AMD; Ranibizumab; Treat-and-extend

Year:  2021        PMID: 33528645     DOI: 10.1007/s00417-020-05048-1

Source DB:  PubMed          Journal:  Graefes Arch Clin Exp Ophthalmol        ISSN: 0721-832X            Impact factor:   3.117


  63 in total

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4.  Ranibizumab versus verteporfin for neovascular age-related macular degeneration.

Authors:  David M Brown; Peter K Kaiser; Mark Michels; Gisele Soubrane; Jeffrey S Heier; Robert Y Kim; Judy P Sy; Susan Schneider
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5.  Ranibizumab for neovascular age-related macular degeneration.

Authors:  Philip J Rosenfeld; David M Brown; Jeffrey S Heier; David S Boyer; Peter K Kaiser; Carol Y Chung; Robert Y Kim
Journal:  N Engl J Med       Date:  2006-10-05       Impact factor: 91.245

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Authors:  Stephan Michels; Ursula Schmidt-Erfurth; Philip J Rosenfeld
Journal:  Expert Opin Investig Drugs       Date:  2006-07       Impact factor: 6.206

7.  Ranibizumab for exudative age-related macular degeneration: A five year study of adherence to follow-up in a real-life setting.

Authors:  E Boulanger-Scemama; G Querques; F About; N Puche; M Srour; V Mane; N Massamba; F Canoui-Poitrine; E H Souied
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Review 8.  The evolving role of vascular endothelial growth factor inhibitors in the treatment of neovascular age-related macular degeneration.

Authors:  H Dadgostar; N Waheed
Journal:  Eye (Lond)       Date:  2008-04-04       Impact factor: 3.775

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Authors:  Salvatore Grisanti; Focke Ziemssen
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10.  Mortality in patients treated with intravitreal bevacizumab for age-related macular degeneration.

Authors:  Joel Hanhart; Doron S Comaneshter; Yossi Freier Dror; Shlomo Vinker
Journal:  BMC Ophthalmol       Date:  2017-10-10       Impact factor: 2.209

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1.  Cytochrome P450 oxidase 2J inhibition suppresses choroidal neovascularization in mice.

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2.  A Randomized, Controlled Trial of Treat-and-Extend vs. Pro Re Nata Regimen for Neovascular Age-Related Macular Degeneration.

Authors:  Huixun Jia; Bing Lu; Yuanzhi Yuan; Fei Yuan; Lei Li; Yanping Song; Ao Rong; Minwen Zhou; Fenghua Wang; Xiaodong Sun
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3.  Brolucizumab in Neovascular Age-Related Macular Degeneration - Indian Real-World Experience: The BRAILLE Study.

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Review 4.  Long-Chain Polyunsaturated Fatty Acids and Their Metabolites Regulate Inflammation in Age-Related Macular Degeneration.

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