Literature DB >> 33512014

Inflammation of the periodontium associates with risk of future cardiovascular events.

Thomas E Van Dyke1,2, Karim El Kholy1,2, Amorina Ishai3, Richard A P Takx3, Kene Mezue3,4,5, Shady M Abohashem3,4,5, Abdelrahman Ali3, Neal Yuan6, Priscilla Hsue6, Michael T Osborne3,4,5, Ahmed Tawakol3,4,5.   

Abstract

BACKGROUND: While growing evidence suggests a link between periodontal disease (PD) and cardiovascular disease (CVD), the independence of this association and the pathway remain unclear. Herein, we tested the hypotheses that: (1) inflammation of the periodontium (PDinflammation ) predicts future CVD independently of disease risk factors shared between CVD and PD, and (2) the mechanism linking the two diseases involves heightened arterial inflammation.
METHODS: 18 F-fluorodeoxyglucose positron emission tomography/computed tomography (18 F-FDG-PET/CT) imaging was performed in 304 individuals (median age 54 years; 42.4% male) largely for cancer screening; individuals without active cancer were included. PDinflammation and arterial inflammation were quantified using validated 18 F-FDG-PET/CT methods. Additionally, we evaluated the relationship between PDinflammation and subsequent major adverse cardiovascular events (MACE) using Cox models and log-rank tests.
RESULTS: Thirteen individuals developed MACE during follow-up (median 4.1 years). PDinflammation associated with arterial inflammation, remaining significant after adjusting for PD and CVD risk factors (standardized β [95% CI]: 0.30 [0.20-0.40], P < 0.001). PDinflammation predicted subsequent MACE (standardized HR [95% CI]: 2.25 [1.47 to 3.44], P <0.001, remaining significant in multivariable models), while periodontal bone loss did not. Furthermore, mediation analysis suggested that arterial inflammation accounts for 80% of the relationship between PDinflammation and MACE (standardized log odds ratio [95% CI]: 0.438 [0.019-0.880], P = 0.022).
CONCLUSION: PDinflammation is independently associated with MACE via a mechanism that may involve increased arterial inflammation. These findings provide important support for an independent relationship between PDinflammation and CVD.
© 2021 American Academy of Periodontology.

Entities:  

Keywords:  atherosclerosis; cardiovascular disease; positron emission tomography;  periodontal disease

Mesh:

Substances:

Year:  2021        PMID: 33512014      PMCID: PMC8080258          DOI: 10.1002/JPER.19-0441

Source DB:  PubMed          Journal:  J Periodontol        ISSN: 0022-3492            Impact factor:   6.993


  53 in total

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5.  Characteristics and Outcome in Acute Coronary Syndrome Patients with and without Established Modifiable Cardiovascular Risk Factors: Insights from the Nationwide AMIS Plus Registry 1997-2010.

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Review 6.  Macrophage death and defective inflammation resolution in atherosclerosis.

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Journal:  Nat Rev Immunol       Date:  2009-12-04       Impact factor: 53.106

7.  Periodontitis-induced lipid peroxidation in rat descending aorta is involved in the initiation of atherosclerosis.

Authors:  D Ekuni; T Tomofuji; T Sanbe; K Irie; T Azuma; T Maruyama; N Tamaki; J Murakami; S Kokeguchi; T Yamamoto
Journal:  J Periodontal Res       Date:  2009-02-06       Impact factor: 4.419

8.  Periodontitis is associated with platelet activation.

Authors:  Dimitris Papapanagiotou; Elena A Nicu; Sergio Bizzarro; Victor E A Gerdes; Joost C Meijers; Rienk Nieuwland; Ubele van der Velden; Bruno G Loos
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9.  Platelet activation and platelet-leukocyte interaction in generalized aggressive periodontitis.

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10.  Innate immunity and monocyte-macrophage activation in atherosclerosis.

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Review 6.  The Roles of Neutrophils Linking Periodontitis and Atherosclerotic Cardiovascular Diseases.

Authors:  Rizky A Irwandi; Scott T Chiesa; George Hajishengallis; Venizelos Papayannopoulos; John E Deanfield; Francesco D'Aiuto
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Review 10.  What Are the Clinical and Systemic Results of Periodontitis Treatment in Obese Individuals?

Authors:  Carina M Silva-Boghossian; Romulo S Dezonne
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