Literature DB >> 33492535

Polyphosphate, Derived from Lactobacillus brevis, Modulates the Intestinal Microbiome and Attenuates Acute Pancreatitis.

Shuhei Takauji1,2, Hiroaki Konishi3, Mikihiro Fujiya4,5, Nobuhiro Ueno2, Hiroki Tanaka6, Hiroki Sato2, Shotaro Isozaki2, Shin Kashima2, Kentaro Moriichi2, Yusuke Mizukami7, Toshikatsu Okumura8.   

Abstract

BACKGROUND: We previously showed that Lactobacillus brevis-derived polyphosphate (poly P) exerts a curative effect on intestinal inflammation. However, whether or not poly P improves the inflammation and injury of distant organs remains unclear. AIMS: We aimed to investigate the change in the intestinal microbiome and to evaluate the protective effect of poly P on injuries in a cerulein-induced acute pancreatitis (AP) mouse.
METHODS: Poly P was orally administered to BALB/C mice every day for 24 days, and then mice were intraperitoneally injected with cerulein. Before cerulein injection, stool samples were collected and analyzed by 16S rRNA gene sequencing. Mice were sacrificed at 24 h after the last cerulein injection; subsequently, the serum, pancreas, and colon were collected.
RESULTS: The microbial profile differed markedly between poly P and control group. Notably, the levels of beneficial bacteria, including Alistipes and Candidatus_Saccharimonas, were significantly increased, while those of the virulent bacteria Desulfovibrio were decreased in the poly P group. The elevations of the serum amylase and lipase levels by cerulein treatment were suppressed by the pre-administration of poly P for 24 days, but not for 7 days. The numbers of cells MPO-positive by immunohistology were decreased and the levels of MCP-1 significantly reduced in the AP + Poly P group. An immunofluorescence analysis showed that the ZO-1 and occludin in the colon was strongly augmented in the epithelial cell membrane layer in the AP + Poly P group.
CONCLUSIONS: Poly P attenuates AP through both modification of the intestinal microbiome and enhancement of the intestinal barrier integrity.

Entities:  

Keywords:  16S rRNA gene sequencing; Acute pancreatitis; Intestinal microbiome; Polyphosphate; Probiotics

Year:  2021        PMID: 33492535     DOI: 10.1007/s10620-020-06747-9

Source DB:  PubMed          Journal:  Dig Dis Sci        ISSN: 0163-2116            Impact factor:   3.199


  1 in total

Review 1.  Colonic sulfide in pathogenesis and treatment of ulcerative colitis.

Authors:  W E Roediger; J Moore; W Babidge
Journal:  Dig Dis Sci       Date:  1997-08       Impact factor: 3.199

  1 in total
  5 in total

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Authors:  Xiao-Yu Dai; Qian Hu; Jia-Qi Yao; Xiao-Jia Wu; Yi-Fan Miao; Juan Li; Mei-Hua Wan; Wen-Fu Tang
Journal:  Evid Based Complement Alternat Med       Date:  2022-05-09       Impact factor: 2.650

Review 2.  Gut Dysbiosis in Pancreatic Diseases: A Causative Factor and a Novel Therapeutic Target.

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Journal:  Front Nutr       Date:  2022-02-15

Review 3.  Intestinal Microbiota - An Unmissable Bridge to Severe Acute Pancreatitis-Associated Acute Lung Injury.

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Journal:  Front Immunol       Date:  2022-06-14       Impact factor: 8.786

Review 4.  Microbiota in Pancreatic Diseases: A Review of the Literature.

Authors:  Tommaso Schepis; Sara S De Lucia; Enrico C Nista; Vittoria Manilla; Giulia Pignataro; Veronica Ojetti; Andrea Piccioni; Antonio Gasbarrini; Francesco Franceschi; Marcello Candelli
Journal:  J Clin Med       Date:  2021-12-17       Impact factor: 4.241

5.  Anti-Osteoporotic Effect of Lactobacillus brevis AR281 in an Ovariectomized Mouse Model Mediated by Inhibition of Osteoclast Differentiation.

Authors:  Jing Yu; Yiling Hang; Wenni Sun; Guangqiang Wang; Zhiqiang Xiong; Lianzhong Ai; Yongjun Xia
Journal:  Biology (Basel)       Date:  2022-02-24
  5 in total

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