Literature DB >> 33468134

Correlation analysis of IL-11 polymorphisms and Hirschsprung disease subtype susceptibility in Southern Chinese Children.

Hong Zhang1, Jing-Lu Zhao1, Yi Zheng1, Xiao-Li Xie1, Li-Hua Huang1, Le Li1, Yun Zhu1, Li-Feng Lu1, Tu-Qun Hu1, Wei Zhong2, Qiu-Ming He3.   

Abstract

BACKGROUND: Hirschsprung disease (HSCR) is a hereditary defect, which is characterized by the absence of enteric ganglia and is frequently concurrent with Hirschsprung-associated enterocolitis (HAEC). However, the pathogenesis for HSCR is complicated and remains unclear. Recent studies have shown that pro-inflammatory cytokines such as interleukin-11 (IL-11) are involved in the enteric nervous system's progress. It was found that IL-11 SNPs (rs8104023 and rs4252546) are associated with HSCR in the Korean population waiting for replication in an independent cohort. This study evaluated the relationship between IL-11 and the susceptibility of patients to HSCR by performing subphenotype interaction examination, HAEC pre-/post-surgical patient-only association analysis, and independence testing.
METHODS: In this study, a cohort consisting of children from Southern China, comprising 1470 cases and 1473 controls, was chosen to examine the relationship between two polymorphisms (rs8104023 and rs4252546 in IL-11) and susceptibility to HSCR by replication research, subphenotype association analysis, and independence testing.
RESULTS: The results showed that IL-11 gene polymorphisms (rs8104023 and rs4252546) are not associated with the risk of HSCR in the Chinese population. The results of both short-segment and long-segment (S-HSCR and L-HSCR) surgery (3.34 ≤ OR ≤ 4.05, 0.02 ≤ P ≤ 0.04) showed that single nucleotide polymorphisms (SNP) rs8104023 is associated with susceptibility to HAEC.
CONCLUSIONS: This study explored the relationship between genetic polymorphisms and susceptibility to HAEC in HSCR subtypes for the first time. These findings should be replicated in a larger and multicentre study.

Entities:  

Keywords:  Hirschsprung's disease; Hirschsprung's disease-associated enterocolitis; IL-11; Polymorphisms

Year:  2021        PMID: 33468134      PMCID: PMC7814452          DOI: 10.1186/s12920-020-00867-x

Source DB:  PubMed          Journal:  BMC Med Genomics        ISSN: 1755-8794            Impact factor:   3.063


  17 in total

Review 1.  Hirschsprung-associated enterocolitis: pathogenesis, treatment and prevention.

Authors:  Farokh R Demehri; Ihab F Halaweish; Arnold G Coran; Daniel H Teitelbaum
Journal:  Pediatr Surg Int       Date:  2013-09       Impact factor: 1.827

2.  Genetic variants of IL-11 associated with risk of Hirschsprung disease.

Authors:  L H Kim; H S Cheong; J-G Shin; J-M Seo; D-Y Kim; J-T Oh; H-Y Kim; K Jung; I Koh; J-H Kim; H D Shin
Journal:  Neurogastroenterol Motil       Date:  2015-07-14       Impact factor: 3.598

3.  Altered expression of IL36γ and IL36 receptor (IL1RL2) in the colon of patients with Hirschsprung's disease.

Authors:  Christian Tomuschat; Anne Marie O'Donnell; David Coyle; Prem Puri
Journal:  Pediatr Surg Int       Date:  2016-11-16       Impact factor: 1.827

4.  GT-repeat extension in the IL11 promoter is associated with Hirschsprung's disease (HSCR).

Authors:  Michael G Haase; Annekatrin Schulze; Sandeep Grover; Ivonne Kemnitz; Inke R König; Guido Fitze
Journal:  Gene       Date:  2018-07-26       Impact factor: 3.688

5.  Recombinant human interleukin-11 restores smooth muscle function in the jejunum and colon of human leukocyte antigen-B27 rats with intestinal inflammation.

Authors:  B Greenwood-Van Meerveld; K Venkova; J C Keith
Journal:  J Pharmacol Exp Ther       Date:  2001-10       Impact factor: 4.030

6.  Molecular cloning of a cDNA encoding interleukin 11, a stromal cell-derived lymphopoietic and hematopoietic cytokine.

Authors:  S R Paul; F Bennett; J A Calvetti; K Kelleher; C R Wood; R M O'Hara; A C Leary; B Sibley; S C Clark; D A Williams
Journal:  Proc Natl Acad Sci U S A       Date:  1990-10       Impact factor: 11.205

7.  Increased Act1/IL-17R expression in Hirschsprung's disease.

Authors:  Christian Tomuschat; Anne Marie O'Donnell; David Coyle; Prem Puri
Journal:  Pediatr Surg Int       Date:  2016-09-22       Impact factor: 1.827

8.  Age-dependent changes in the gut environment restrict the invasion of the hindgut by enteric neural progenitors.

Authors:  Noah R Druckenbrod; Miles L Epstein
Journal:  Development       Date:  2009-09       Impact factor: 6.868

9.  Multiple functional effects of RET kinase domain sequence variants in Hirschsprung disease.

Authors:  Brandy D Hyndman; Taranjit S Gujral; Jonathan R Krieger; Jessica G Cockburn; Lois M Mulligan
Journal:  Hum Mutat       Date:  2012-08-23       Impact factor: 4.878

10.  Contribution of interaction between genetic variants of interleukin-11 and Helicobacter pylori infection to the susceptibility of gastric cancer.

Authors:  Chuanwen Liao; Shuqin Hu; Zihan Zheng; Huazhang Tong
Journal:  Onco Targets Ther       Date:  2019-09-11       Impact factor: 4.147

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