Literature DB >> 33415786

CaV α2δ Autoimmune Encephalitis: A Novel Antibody and its Characteristics.

Soon-Tae Lee1, Byoung Joo Lee2, Ji-Yeon Bae1, Young Sook Kim1, Do-Hyun Han3,4, Hyun-Sook Shin3, Soyun Kim1, Dong-Kyu Park1, Sang Won Seo5, Kon Chu1, Sang Kun Lee1, Won-Kyung Ho2,6.   

Abstract

OBJECTIVE: Discovery of a novel antibody would enable diagnosis and early treatment of autoimmune encephalitis. The aim was to discover a novel antibody targeting a synaptic receptor and characterize the pathogenic mechanism.
METHOD: We screened for unknown antibodies in serum and cerebrospinal fluid samples from autoimmune encephalitis patients. Samples with reactivity to rat brain sections and no reactivity to conventional antibody tests underwent further processing for antibody discovery, using immunoprecipitation to primary neuronal cells, mass-spectrometry analysis, an antigen-binding assay on an antigen-overexpressing cell line, and an electrophysiological assay with cultured hippocampal neurons.
RESULTS: Two patients had a novel antibody against CaV α2δ (voltage-gated calcium channel alpha-2/delta subunit). The patient samples stained neuropils of the hippocampus, basal ganglia, and cortex in rat brain sections and bound to a CaV α2δ-overexpressing cell line. Knockdown of CaV α2δ expression in cultured neurons turned off the immunoreactivity of the antibody from the patients to the neurons. The patients were associated with preceding meningitis or neuroendocrine carcinoma and responded to immunotherapy. In cultured neurons, the antibody reduced neurotransmitter release from presynaptic nerve terminals by interfering with tight coupling of calcium channels and exocytosis.
INTERPRETATION: Here, we discovered a novel autoimmune encephalitis associated with anti-CaV α2δ antibody. Further analysis of the antibody in autoimmune encephalitis might promote early diagnosis and treatment. ANN NEUROL 2021;89:740-752.
© 2021 American Neurological Association.

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Year:  2021        PMID: 33415786     DOI: 10.1002/ana.26017

Source DB:  PubMed          Journal:  Ann Neurol        ISSN: 0364-5134            Impact factor:   10.422


  2 in total

1.  Biallelic CACNA2D1 loss-of-function variants cause early-onset developmental epileptic encephalopathy.

Authors:  Shehrazade Dahimene; Leonie von Elsner; Tess Holling; Lauren S Mattas; Jess Pickard; Davor Lessel; Kjara S Pilch; Ivan Kadurin; Wendy S Pratt; Igor B Zhulin; Hongzheng Dai; Maja Hempel; Maura R Z Ruzhnikov; Kerstin Kutsche; Annette C Dolphin
Journal:  Brain       Date:  2022-08-27       Impact factor: 15.255

2.  Identification of Anti-Collapsin Response Mediator Protein 2 Antibodies in Patients With Encephalitis or Encephalomyelitis.

Authors:  Kaibiao Xu; Dongmei Wang; Yan He; Shengnan Wang; Guanghui Liu; Yue Pan; Haishan Jiang; Yu Peng; Fenliang Xiao; Yihua Huang; Qiqi Wang; Yongming Wu; Suyue Pan; Yafang Hu
Journal:  Front Immunol       Date:  2022-04-11       Impact factor: 8.786

  2 in total

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