Literature DB >> 33360425

Guillain-Barré syndrome during the Zika virus outbreak in Northeast Brazil: An observational cohort study.

Sonja E Leonhard1, Susan Halstead2, Suzannah B Lant3, Maria de Fatima Pessoa Militão de Albuquerque4, Carlos Alexandre Antunes de Brito5, Lívia Brito Bezerra de Albuquerque6, Mark A Ellul7, Rafael Freitas de Oliveira França8, Dawn Gourlay2, Michael J Griffiths9, Adélia Maria de Miranda Henriques-Souza10, Maria Í de Morais Machado10, Raquel Medialdea-Carrera3, Ravi Mehta3, Roberta da Paz Melo11, Solange D Mesquita11, Álvaro J P Moreira11, Lindomar J Pena8, Marcela Lopes Santos4, Lance Turtle12, Tom Solomon7, Hugh J Willison2, Bart C Jacobs13, Maria L Brito Ferreira11.   

Abstract

OBJECTIVE: To determine the clinical phenotype of Guillain-Barré syndrome (GBS) after Zika virus (ZIKV) infection, the anti-glycolipid antibody signature, and the role of other circulating arthropod-borne viruses, we describe a cohort of GBS patients identified during ZIKV and chikungunya virus (CHIKV) outbreaks in Northeast Brazil.
METHODS: We prospectively recruited GBS patients from a regional neurology center in Northeast Brazil between December 2014 and February 2017. Serum and CSF were tested for ZIKV, CHIKV, and dengue virus (DENV), by RT-PCR and antibodies, and serum was tested for GBS-associated antibodies to glycolipids.
RESULTS: Seventy-one patients were identified. Forty-eight (68%) had laboratory evidence of a recent arbovirus infection; 25 (52%) ZIKV, 8 (17%) CHIKV, 1 (2%) DENV, and 14 (29%) ZIKV and CHIKV. Most patients with a recent arbovirus infection had motor and sensory symptoms (72%), a demyelinating electrophysiological subtype (67%) and a facial palsy (58%). Patients with a recent infection with ZIKV and CHIKV had a longer hospital admission and more frequent mechanical ventilation compared to the other patients. No specific anti-glycolipid antibody signature was identified in association with arbovirus infection, although significant antibody titres to GM1, GalC, LM1, and GalNAc-GD1a were found infrequently.
CONCLUSION: A large proportion of cases had laboratory evidence of a recent infection with ZIKV or CHIKV, and recent infection with both viruses was found in almost one third of patients. Most patients with a recent arbovirus infection had a sensorimotor, demyelinating GBS. We did not find a specific anti-glycolipid antibody signature in association with arbovirus-related GBS.
Copyright © 2020 The Author(s). Published by Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Arbovirus; Chikungunya virus; Guillain-Barré syndrome; Neuroinflammatory disease; Zika virus

Mesh:

Year:  2020        PMID: 33360425     DOI: 10.1016/j.jns.2020.117272

Source DB:  PubMed          Journal:  J Neurol Sci        ISSN: 0022-510X            Impact factor:   3.181


  7 in total

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2.  Presence of SARS-CoV-2 in the CSF of Guillain-Barré Syndrome Patients Requires Validation.

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5.  Antecedent infections in Guillain-Barré syndrome in endemic areas of arbovirus transmission: A multinational case-control study.

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