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Literature DB >> 3334781

Transthyretin (prealbumin) in familial amyloidotic polyneuropathy: genetic and functional aspects.

M J Saraiva¹, P P Costa, D S Goodman.

Abstract

Entities: Disease Gene

Mesh：

Substances：
Prealbumin

Year: 1988 PMID： 3334781

Source DB: PubMed Journal: Adv Neurol ISSN： 0091-3952

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7 in total

1. The molecular interaction of 4'-iodo-4'-deoxydoxorubicin with Leu-55Pro transthyretin 'amyloid-like' oligomer leading to disaggregation.

Authors: M P Sebastião; G Merlini; M J Saraiva; A M Damas
Journal: Biochem J Date: 2000-10-01 Impact factor: 3.857

2. FRET studies of various conformational states adopted by transthyretin.

Authors: Seyyed Abolghasem Ghadami; Francesco Bemporad; Benedetta Maria Sala; Guido Tiana; Stefano Ricagno; Fabrizio Chiti
Journal: Cell Mol Life Sci Date: 2017-05-06 Impact factor: 9.261

3. Evaluating the binding selectivity of transthyretin amyloid fibril inhibitors in blood plasma.

Authors: H E Purkey; M I Dorrell; J W Kelly
Journal: Proc Natl Acad Sci U S A Date: 2001-05-08 Impact factor: 11.205

4. Selective binding to transthyretin and tetramer stabilization in serum from patients with familial amyloidotic polyneuropathy by an iodinated diflunisal derivative.

Authors: Maria Rosário Almeida; Bárbara Macedo; Isabel Cardoso; Isabel Alves; Gregorio Valencia; Gemma Arsequell; Antoni Planas; Maria João Saraiva
Journal: Biochem J Date: 2004-07-15 Impact factor: 3.857

Transthyretin (prealbumin) in familial amyloidotic polyneuropathy: genetic and functional aspects.

1. The molecular interaction of 4'-iodo-4'-deoxydoxorubicin with Leu-55Pro transthyretin 'amyloid-like' oligomer leading to disaggregation.

2. FRET studies of various conformational states adopted by transthyretin.

3. Evaluating the binding selectivity of transthyretin amyloid fibril inhibitors in blood plasma.

4. Selective binding to transthyretin and tetramer stabilization in serum from patients with familial amyloidotic polyneuropathy by an iodinated diflunisal derivative.

5. Transthyretin gene mutations in British and French patients with amyloid neuropathy.

6. Probing conformational changes of monomeric transthyretin with second derivative fluorescence.

7. Haplotype analysis of common transthyretin mutations.