Literature DB >> 33341911

Targeted Use of Prednisolone with Intravenous Immunoglobulin for Kawasaki Disease.

Hidemasa Sakai1,2, Satoru Iwashima3,4, Shinichiro Sano1,5, Naoe Akiyama1,6, Eiko Nagata1,7, Masashi Harazaki1,8, Tetuya Fukuoka1,9.   

Abstract

BACKGROUND AND OBJECTIVES: Intravenous immunoglobulin (IVIG) therapy for acute-stage Kawasaki disease (KD) is the first-line treatment for preventing the development of coronary artery aneurysms (CAA). Corticosteroids (prednisolone) and infliximab are often used in patients at a high risk of CAA or those with CAA at diagnosis; however, there are only a few reports of non-responders to corticosteroids as an adjuvant therapy or rescue alternative to IVIG. In this study, we compared the therapeutic effects of primary and secondary prednisolone with IVIG for KD.
METHODS: We established the following three protocols: A was a secondary rescue prednisolone protocol; B was no prednisolone and second-line infliximab protocol, and C was the primary prednisolone protocol. The indication for prednisolone administration was based on the following: primary prednisolone administration, Kobayashi score; and secondary administration, Shizuoka score.
RESULTS: Four hundred and sixty-nine patients were enrolled in the three protocols. A comparison between primary and secondary prednisolone and IVIG, as the first-line therapy revealed that the number of first non-responders in C group was 7 (8.3%), which was significantly lower than the 50 (20.9%) in A group. There was a significant difference in the first and second non-responders among the three groups, and the number of non-responders in A group was 6 (2.5%), which was significantly lower than the 13 (9.9%) in B group (p < 0.001, by Bonferroni test). The multivariate logistic regression analysis showed that IVIG non-responders among the protocol groups had an adjusted odds ratio of 6.47. Fifteen IVIG non-responders were administered infliximab as a second-line therapy, and of them, 9 (60%) showed therapy resistance. CAA occurred in 21 patients (4.6%). There was no significant difference among each protocol group.
CONCLUSIONS: The number of IVIG non-responders in the group with prednisolone administration was lower than that in the group without prednisolone administration. Secondary rescue infliximab therapy for IVIG non-responders resulted in a lower defervescence effect than the secondary rescue IVIG with prednisolone administration. Further prospective randomized studies are needed to identify factors useful for preventing IVIG non-responders and determine the optimal rescue therapy for preventing CAA.

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Year:  2020        PMID: 33341911     DOI: 10.1007/s40261-020-00984-6

Source DB:  PubMed          Journal:  Clin Drug Investig        ISSN: 1173-2563            Impact factor:   2.859


  44 in total

1.  Initial intravenous gammaglobulin treatment failure in Kawasaki disease.

Authors:  C A Wallace; J W French; S J Kahn; D D Sherry
Journal:  Pediatrics       Date:  2000-06       Impact factor: 7.124

2.  The treatment of Kawasaki syndrome with intravenous gamma globulin.

Authors:  J W Newburger; M Takahashi; J C Burns; A S Beiser; K J Chung; C E Duffy; M P Glode; W H Mason; V Reddy; S P Sanders
Journal:  N Engl J Med       Date:  1986-08-07       Impact factor: 91.245

3.  Management and outcome of persistent or recurrent fever after initial intravenous gamma globulin therapy in acute Kawasaki disease.

Authors:  R K Han; E D Silverman; A Newman; B W McCrindle
Journal:  Arch Pediatr Adolesc Med       Date:  2000-07

4.  Immunoglobulin failure and retreatment in Kawasaki disease.

Authors:  K Durongpisitkul; J Soongswang; D Laohaprasitiporn; A Nana; C Prachuabmoh; C Kangkagate
Journal:  Pediatr Cardiol       Date:  2002-12-04       Impact factor: 1.655

Review 5.  Diagnosis, Treatment, and Long-Term Management of Kawasaki Disease: A Scientific Statement for Health Professionals From the American Heart Association.

Authors:  Brian W McCrindle; Anne H Rowley; Jane W Newburger; Jane C Burns; Anne F Bolger; Michael Gewitz; Annette L Baker; Mary Anne Jackson; Masato Takahashi; Pinak B Shah; Tohru Kobayashi; Mei-Hwan Wu; Tsutomu T Saji; Elfriede Pahl
Journal:  Circulation       Date:  2017-03-29       Impact factor: 29.690

6.  Prevalence of coronary artery abnormalities in Kawasaki disease is highly dependent on gamma globulin dose but independent of salicylate dose.

Authors:  M Terai; S T Shulman
Journal:  J Pediatr       Date:  1997-12       Impact factor: 4.406

7.  Corticosteroids in the initial treatment of Kawasaki disease: report of a randomized trial.

Authors:  Robert P Sundel; Annette L Baker; David R Fulton; Jane W Newburger
Journal:  J Pediatr       Date:  2003-06       Impact factor: 4.406

8.  Intravenous gamma-globulin treatment and retreatment in Kawasaki disease. US/Canadian Kawasaki Syndrome Study Group.

Authors:  J C Burns; E V Capparelli; J A Brown; J W Newburger; M P Glode
Journal:  Pediatr Infect Dis J       Date:  1998-12       Impact factor: 2.129

9.  Efficacy of intravenous immune globulin therapy combined with dexamethasone for the initial treatment of acute Kawasaki disease.

Authors:  Toshiaki Jibiki; Masaru Terai; Tomomichi Kurosaki; Hiromichi Nakajima; Kazuhiro Suzuki; Hiroaki Inomata; Itaru Terashima; Takafumi Honda; Kumi Yasukawa; Hiromichi Hamada; Yoichi Kohno
Journal:  Eur J Pediatr       Date:  2004-02-13       Impact factor: 3.183

10.  The prevention of coronary artery aneurysm in Kawasaki disease: a meta-analysis on the efficacy of aspirin and immunoglobulin treatment.

Authors:  K Durongpisitkul; V J Gururaj; J M Park; C F Martin
Journal:  Pediatrics       Date:  1995-12       Impact factor: 7.124

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