Literature DB >> 33221815

Teprotumumab for non-inflammatory thyroid eye disease (TED): evidence for increased IGF-1R expression.

Shoaib Ugradar1, Lu Shi2, Yao Wang3, Tunde Mester3, Huasheng Yang2, Raymond S Douglas3.   

Abstract

PURPOSE: Teprotumumab, a blocking antibody to the insulin like growth factor 1 receptor (IGF-1R) has been shown to significantly reduce proptosis in recent phase 2 and 3 trials in patients with inflammatory thyroid eye disease (TED). Herein, we investigate the impact of teprotumumab on patients with non-inflammatory TED. We also investigate the expression of the IGF-1R on orbital tissues from patients with inflammatory and non-inflammatory TED compared to controls.
METHODS: Consecutive patients with non-inflammatory TED (clinical activity score, CAS ≤ 1, for at least 4 months, were treated with teprotumumab. They received a complete course (total eight infusions) of teprotumumab (10 mg/kg for the first infusion and 20 mg/kg for subsequent infusions every 3 weeks). The primary outcome was a proptosis response at week 24. Further, IGF-1R α and β expression was evaluated on orbital tissue from patients with inflammatory and non-inflammatory TED, as well as healthy controls. Non-biased histological analysis of IGF-1R expression was performed using ImageJ.
RESULTS: Four patients met eligibility criteria for the clinical study, with a mean (SD) CAS of 0 (0). Following teprotumumab treatment, there was a mean (SD) reduction in proptosis of 2.6 mm (1.2). Five patients were included for each group of the histological study; inflammatory TED, non-inflammatory TED and controls. IGF-1Rα and IGF-1Rβ expression was significantly greater in the orbital tissues of patients with inflammatory TED and non-inflammatory TED, when compared to controls.
CONCLUSION: Our findings demonstrate for the first time, that teprotumumab, a blocking antibody to the IGF-1R reduces proptosis in a series of patients with non-inflammatory TED. Overexpression of the IGF-1R in orbital tissue from patients with non-inflammatory disease compared to controls may be an important consideration for effect.
© 2020. The Author(s), under exclusive licence to The Royal College of Ophthalmologists.

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Year:  2020        PMID: 33221815      PMCID: PMC8376861          DOI: 10.1038/s41433-020-01297-w

Source DB:  PubMed          Journal:  Eye (Lond)        ISSN: 0950-222X            Impact factor:   4.456


  6 in total

1.  The role of teprotumumab in chronic, clinically active thyroid eye disease.

Authors:  Caroline Y Yu; Brittany A Simmons; Chau M Pham; Erin M Shriver
Journal:  Eye (Lond)       Date:  2022-01-04       Impact factor: 4.456

2.  Dihydroartemisinin Exerts Antifibrotic and Anti-Inflammatory Effects in Graves' Ophthalmopathy by Targeting Orbital Fibroblasts.

Authors:  Shenglan Yang; Xing Wang; Wei Xiao; Zhihui Xu; Huijing Ye; Xiaotong Sha; Huasheng Yang
Journal:  Front Endocrinol (Lausanne)       Date:  2022-05-17       Impact factor: 6.055

3.  Facial and Eyelid Changes in Thyroid Eye Disease Are Reversed by Teprotumumab.

Authors:  Shoaib Ugradar; Jenna Braun; Yao Wang; Erin Zimmerman; Raymond S Douglas
Journal:  Plast Reconstr Surg Glob Open       Date:  2021-09-15

Review 4.  Thyroid eye disease: From pathogenesis to targeted therapies.

Authors:  Jin Sook Yoon; Don O Kikkawa
Journal:  Taiwan J Ophthalmol       Date:  2022-01-21

Review 5.  Update on thyroid eye disease: Regional variations in prevalence, diagnosis, and management.

Authors:  Caroline Y Yu; Rebecca L Ford; Sara T Wester; Erin M Shriver
Journal:  Indian J Ophthalmol       Date:  2022-07       Impact factor: 2.969

6.  Teprotumumab for the treatment of chronic thyroid eye disease.

Authors:  Shoaib Ugradar; Julia Kang; Andrea L Kossler; Erin Zimmerman; Jenna Braun; Andrew R Harrison; Swaraj Bose; Kimberly Cockerham; Raymond S Douglas
Journal:  Eye (Lond)       Date:  2021-07-09       Impact factor: 4.456

  6 in total

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