Literature DB >> 33138691

Ischemic Cerebral Endothelial Cell-Derived Exosomes Promote Axonal Growth.

Yi Zhang1, Yi Qin1, Michael Chopp1,2, Chao Li1, Amy Kemper3, Xianshuang Liu1, Xinli Wang1, Li Zhang1, Zheng Gang Zhang1.   

Abstract

BACKGROUND AND
PURPOSE: Cerebral endothelial cells (CECs) and axons of neurons interact to maintain vascular and neuronal homeostasis and axonal remodeling in normal and ischemic brain, respectively. However, the role of exosomes in the interaction of CECs and axons in brain under normal conditions and after stroke is unknown.
METHODS: Exosomes were isolated from CECs of nonischemic rats and is chemic rats (nCEC-exos and isCEC-exos), respectively. A multicompartmental cell culture system was used to separate axons from neuronal cell bodies.
RESULTS: Axonal application of nCEC-exos promotes axonal growth of cortical neurons, whereas isCEC-exos further enhance axonal growth than nCEC-exos. Ultrastructural analysis revealed that CEC-exos applied into distal axons were internalized by axons and reached to their parent somata. Bioinformatic analysis revealed that both nCEC-exos and isCEC-exos contain abundant mature miRNAs; however, isCEC-exos exhibit more robust elevation of select miRNAs than nCEC-exos. Mechanistically, axonal application of nCEC-exos and isCEC-exos significantly elevated miRNAs and reduced proteins in distal axons and their parent somata that are involved in inhibiting axonal outgrowth. Blockage of axonal transport suppressed isCEC-exo-altered miRNAs and proteins in somata but not in distal axons.
CONCLUSIONS: nCEC-exos and isCEC-exos facilitate axonal growth by altering miRNAs and their target protein profiles in recipient neurons.

Entities:  

Keywords:  axons; cell body; endothelial cells; exosomes; microRNAs

Mesh:

Substances:

Year:  2020        PMID: 33138691      PMCID: PMC7686085          DOI: 10.1161/STROKEAHA.120.031728

Source DB:  PubMed          Journal:  Stroke        ISSN: 0039-2499            Impact factor:   7.914


  56 in total

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