Literature DB >> 33079326

Follicle inhibition at the primordial stage without increasing apoptosis, with a combination of everolimus, verapamil.

Michail Pargianas1, Ioannis Kosmas2, Kyriaki Papageorgiou3,4, Chrysoula Kitsou5, Alexandra Papoudou-Bai6, Anna Batistatou6, Sofia Markoula5, Styliani Salta5, Alexandros Dalkalitsis5, Stratis Kolibianakis7, Basil C Tarlatzis7, Ioannis Georgiou5, Theologos M Michaelidis3,4.   

Abstract

The aim of the present study was to test whether inhibition of ovarian primordial follicles and subsequent activation can be achieved by transient mTOR inhibition. In this preclinical investigation, forty-five female immature Wistar rats were randomized in 5 groups. The control group received subcutaneous saline injections. The other groups received Everolimus, Everolimus plus Verapamil, Everolimus plus Fisetin, and Fisetin alone. Primary and secondary outcomes were measured in the left ovary after a treatment period of 8 weeks. Ten days later, animals received 35 IU FSH for 4 days and 35 IU of hCG on the 5th day. The same parameters were examined in the right ovary. AMH, estradiol, and progesterone levels were assessed at the end of both interventions. Significantly, more primordial and less atretic follicles were observed in the Everolimus plus Verapamil group. AMH and progesterone levels were substantially lower in the Everolimus group. Interestingly, after ovarian stimulation higher levels of AMH and progesterone were observed in the Everolimus plus Verapamil group. Immunoblot analysis of ovarian extracts revealed that the administration of Everolimus led to a significant reduction in the mTORC1-mediated phosphorylation of the 70-kDa ribosomal protein S6 kinase 1. This decrease was reversed in the presence of FSH after stopping drug administration. The expression of the anti-apoptotic molecule Bcl2 as well as of LC3-II and ATG12 was increased after removal of the Everolimus plus Verapamil combination, indicating reduced apoptosis and increased autophagy, whereas the levels of the proliferation marker PCNA in the granulosa cells were elevated, consistent with initiation of follicular growth.Thus, the combination of Everolimus plus Verapamil is capable of increasing the number of competent primordial follicles while reducing atresia.

Entities:  

Keywords:  Apoptosis; Autophagy; Everolimus plus verapamil; Primordial follicles; mTORC1; mTORC2

Mesh:

Substances:

Year:  2020        PMID: 33079326     DOI: 10.1007/s11033-020-05917-2

Source DB:  PubMed          Journal:  Mol Biol Rep        ISSN: 0301-4851            Impact factor:   2.316


  68 in total

1.  mTOR signaling in the control of activation of primordial follicles.

Authors:  Deepak Adhikari; Kui Liu
Journal:  Cell Cycle       Date:  2010-05-25       Impact factor: 4.534

2.  Verapamil treatment induces cytoprotective autophagy by modulating cellular metabolism.

Authors:  Elżbieta Kania; Beata Pająk; Jim O'Prey; Pablo Sierra Gonzalez; Anna Litwiniuk; Kaja Urbańska; Kevin M Ryan; Arkadiusz Orzechowski
Journal:  FEBS J       Date:  2017-04-18       Impact factor: 5.542

3.  Pharmacokinetic interaction between verapamil and everolimus in healthy subjects.

Authors:  J M Kovarik; D Beyer; M N Bizot; Q Jiang; M J Allison; R L Schmouder
Journal:  Br J Clin Pharmacol       Date:  2005-10       Impact factor: 4.335

4.  Everolimus drug interactions: application of a classification system for clinical decision making.

Authors:  John M Kovarik; Doris Beyer; Robert L Schmouder
Journal:  Biopharm Drug Dispos       Date:  2006-12       Impact factor: 1.627

5.  Phosphatidylinositol 3-kinase/Akt pathway regulates tuberous sclerosis tumor suppressor complex by phosphorylation of tuberin.

Authors:  Han C Dan; Mei Sun; Lin Yang; Richard I Feldman; Xue-Mei Sui; Chien Chen Ou; Mark Nellist; Raymond S Yeung; Dicky J J Halley; Santo V Nicosia; Warren J Pledger; Jin Q Cheng
Journal:  J Biol Chem       Date:  2002-07-11       Impact factor: 5.157

Review 6.  Calcium channel blockers as potential therapeutics for obesity-associated autophagy defects and fatty liver pathologies.

Authors:  Hwan-Woo Park; Jun Hee Lee
Journal:  Autophagy       Date:  2014       Impact factor: 16.016

7.  Disruption of Tsc2 in oocytes leads to overactivation of the entire pool of primordial follicles.

Authors:  Deepak Adhikari; Gilian Flohr; Nagaraju Gorre; Yan Shen; Hairu Yang; Eva Lundin; Zijian Lan; Michael J Gambello; Kui Liu
Journal:  Mol Hum Reprod       Date:  2009-10-20       Impact factor: 4.025

8.  Fisetin inhibits cellular proliferation and induces mitochondria-dependent apoptosis in human gastric cancer cells.

Authors:  Akash Sabarwal; Rajesh Agarwal; Rana P Singh
Journal:  Mol Carcinog       Date:  2016-06-21       Impact factor: 4.784

9.  Identification of the tuberous sclerosis complex-2 tumor suppressor gene product tuberin as a target of the phosphoinositide 3-kinase/akt pathway.

Authors:  Brendan D Manning; Andrew R Tee; M Nicole Logsdon; John Blenis; Lewis C Cantley
Journal:  Mol Cell       Date:  2002-07       Impact factor: 17.970

10.  Tsc/mTORC1 signaling in oocytes governs the quiescence and activation of primordial follicles.

Authors:  Deepak Adhikari; Wenjing Zheng; Yan Shen; Nagaraju Gorre; Tuula Hämäläinen; Austin J Cooney; Ilpo Huhtaniemi; Zi-Jian Lan; Kui Liu
Journal:  Hum Mol Genet       Date:  2009-10-20       Impact factor: 6.150

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  2 in total

1.  Amphetamine-Decreased Progesterone and Estradiol Release in Rat Granulosa Cells: The Regulatory Role of cAMP- and Ca2+-Mediated Signaling Pathways.

Authors:  Chung-Yu Chen; Chien-Rung Chen; Chiao-Nan Chen; Paulus S Wang; Toby Mündel; Yi-Hung Liao; Shiow-Chwen Tsai
Journal:  Biomedicines       Date:  2021-04-29

Review 2.  Interplay Between mTOR and Hippo Signaling in the Ovary: Clinical Choice Guidance Between Different Gonadotropin Preparations for Better IVF.

Authors:  Kyriaki Papageorgiou; Eirini Mastora; Athanasios Zikopoulos; Maria E Grigoriou; Ioannis Georgiou; Theologos M Michaelidis
Journal:  Front Endocrinol (Lausanne)       Date:  2021-07-21       Impact factor: 5.555

  2 in total

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