Literature DB >> 33034675

[Mechanisms of cardiotoxicity of oncological therapies].

L H Lehmann1,2,3, S Fröhling4,5.   

Abstract

BACKGROUND: Oncological therapies show a number of undesired adverse effects on the cardiovascular system. In particular, the side effects of recently established oncological therapies are incompletely understood and clinical data are lacking in the interpretation of novel cardiac complications.
OBJECTIVE: This article provides a short overview of the mechanisms of cardiac side effects of certain oncological therapies.
MATERIAL AND METHODS: The review is mainly based on data from preclinical studies.
RESULTS: Numerous toxic side effects have already been described and investigated in preclinical models. For certain groups of drugs (e.g. anthracyclines, tyrosine kinase inhibitors and immune checkpoint inhibitors) the underlying molecular mechanisms are still not fully understood.
CONCLUSION: An improved understanding of the molecular mechanism involved in cardiotoxicity might help improve the quality of clinical decisions. Additionally, it will provide new insights into the pathophysiology of cardiac diseases. The aim is to use the results of translational research and to clinically implement them in suitable cardio-oncology units.

Entities:  

Keywords:  Anthracyclines; Cardio-oncology; Doxorubicin; Heart failure; Immune checkpoint inhibitor

Mesh:

Substances:

Year:  2020        PMID: 33034675     DOI: 10.1007/s00108-020-00881-2

Source DB:  PubMed          Journal:  Internist (Berl)        ISSN: 0020-9554            Impact factor:   0.743


  30 in total

1.  A quantitative analysis of kinase inhibitor selectivity.

Authors:  Mazen W Karaman; Sanna Herrgard; Daniel K Treiber; Paul Gallant; Corey E Atteridge; Brian T Campbell; Katrina W Chan; Pietro Ciceri; Mindy I Davis; Philip T Edeen; Raffaella Faraoni; Mark Floyd; Jeremy P Hunt; Daniel J Lockhart; Zdravko V Milanov; Michael J Morrison; Gabriel Pallares; Hitesh K Patel; Stephanie Pritchard; Lisa M Wodicka; Patrick P Zarrinkar
Journal:  Nat Biotechnol       Date:  2008-01       Impact factor: 54.908

2.  p53 prevents doxorubicin cardiotoxicity independently of its prototypical tumor suppressor activities.

Authors:  Jie Li; Ping-Yuan Wang; Nathaniel A Long; Jie Zhuang; Danielle A Springer; Jizhong Zou; Yongshun Lin; Christopher K E Bleck; Ji-Hoon Park; Ju-Gyeong Kang; Paul M Hwang
Journal:  Proc Natl Acad Sci U S A       Date:  2019-09-05       Impact factor: 11.205

3.  Prospective Study of Cardiac Events During Proteasome Inhibitor Therapy for Relapsed Multiple Myeloma.

Authors:  Robert F Cornell; Bonnie Ky; Brendan M Weiss; Cherie N Dahm; Deepak K Gupta; Liping Du; Joseph R Carver; Adam D Cohen; Brian G Engelhardt; Alfred L Garfall; Stacey A Goodman; Shelton Lacy Harrell; Adetola A Kassim; Trafina Jadhav; Madan Jagasia; Javid Moslehi; Rupal O'Quinn; Michael R Savona; David Slosky; Amanda Smith; Edward A Stadtmauer; Dan T Vogl; Adam Waxman; Daniel Lenihan
Journal:  J Clin Oncol       Date:  2019-06-12       Impact factor: 44.544

4.  P53 inhibition exacerbates late-stage anthracycline cardiotoxicity.

Authors:  Wuqiang Zhu; Wenjun Zhang; Weinian Shou; Loren J Field
Journal:  Cardiovasc Res       Date:  2014-05-08       Impact factor: 10.787

Review 5.  Cardiovascular adverse events in multiple myeloma patients.

Authors:  Markus B Heckmann; Shirin Doroudgar; Hugo A Katus; Lorenz H Lehmann
Journal:  J Thorac Dis       Date:  2018-12       Impact factor: 2.895

6.  Identification of the molecular basis of doxorubicin-induced cardiotoxicity.

Authors:  Sui Zhang; Xiaobing Liu; Tasneem Bawa-Khalfe; Long-Sheng Lu; Yi Lisa Lyu; Leroy F Liu; Edward T H Yeh
Journal:  Nat Med       Date:  2012-10-28       Impact factor: 53.440

7.  Dexrazoxane may prevent doxorubicin-induced DNA damage via depleting both topoisomerase II isoforms.

Authors:  Shiwei Deng; Tiandong Yan; Cathleen Jendrny; Andrea Nemecek; Mladen Vincetic; Ute Gödtel-Armbrust; Leszek Wojnowski
Journal:  BMC Cancer       Date:  2014-11-18       Impact factor: 4.430

8.  Deficiency in Cardiolipin Reduces Doxorubicin-Induced Oxidative Stress and Mitochondrial Damage in Human B-Lymphocytes.

Authors:  Baikuntha Aryal; V Ashutosh Rao
Journal:  PLoS One       Date:  2016-07-19       Impact factor: 3.240

9.  CaMKII activation participates in doxorubicin cardiotoxicity and is attenuated by moderate GRP78 overexpression.

Authors:  Henrike Tscheschner; Eric Meinhardt; Philipp Schlegel; Andreas Jungmann; Lorenz H Lehmann; Oliver J Müller; Patrick Most; Hugo A Katus; Philip W Raake
Journal:  PLoS One       Date:  2019-04-29       Impact factor: 3.240

10.  A small-molecule allosteric inhibitor of BAX protects against doxorubicin-induced cardiomyopathy.

Authors:  Dulguun Amgalan; Thomas P Garner; Ryan Pekson; Xiaotong F Jia; Mounica Yanamandala; Victor Paulino; Felix G Liang; J Jose Corbalan; Jaehoon Lee; Yun Chen; George S Karagiannis; Luis Rivera Sanchez; Huizhi Liang; Swathi-Rao Narayanagari; Kelly Mitchell; Andrea Lopez; Victoria Margulets; Marco Scarlata; Gaetano Santulli; Aarti Asnani; Randall T Peterson; Rachel B Hazan; John S Condeelis; Maja H Oktay; Ulrich Steidl; Lorrie A Kirshenbaum; Evripidis Gavathiotis; Richard N Kitsis
Journal:  Nat Cancer       Date:  2020-03-02
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