Literature DB >> 32966988

Modulation of Immune Cell Subsets by Hepatitis C Virus and Antiviral Therapy in Early Virological Response HCV Genotype 4-Infected Patients with Compensated Liver Disease.

Sahar Essa1, Raja'a Al-Attiyah2, Iqbal Siddique3,4, Widad Al-Nakib2.   

Abstract

BACKGROUND: Resolution of chronic hepatitis C virus (HCV) infection requires a complicated interaction between immune cell subsets. The effect of antiviral therapy on immune cell subsets remains to be defined. This study aimed to investigate the absolute count of certain immune cell subsets during therapy with pegylated interferon-α and ribavirin (PegIFN/RBV).
MATERIALS AND METHODS: Sixty HCV genotype 4-infected patients with compensated liver disease were treated with PegIFN/RBV therapy for 52 weeks. Efficacy was measured by studying the early virological response (EVR) at post-therapy week 12. Absolute counts of mature T cells, T helper cells, T cytotoxic cells, activated T cells, natural killer cells, natural killer/T (NKT) cells, B cells, and T regulatory cells (Treg), and the ratio of T helper to T cytotoxic cells were longitudinally analyzed by flow cytometry throughout the treatment and follow-up course.
RESULTS: Of the 60 genotype 4-infected subjects, 39 (65%) had EVR and 21 (35%) were non-EVR patients. In the first part of this study, there were significantly lower mean absolute count values of mature T, T cytotoxic, B, and NKT cells. Also, we detected statistically significantly lower mean values for the percentages of T cytotoxic, NKT, Treg, and activated T cells of HCV-infected patients at baseline values when compared with healthy subjects. After the initiation of PegIFN/RBV therapy, frequencies of T helper cells, activated T cells, Treg cells, B cells, and T helper:T cytotoxic ratio were found to be significantly lower in EVR patients than in non-EVR patients (p < 0.05). In contrast, frequencies of T cytotoxic and NKT cells were significantly increased in EVR patients when compared to non-EVR patients (p < 0.05).
CONCLUSION: These results suggest a pattern of higher levels of T cytotoxic and NKT cells, and lower levels of T helper, activated T, Treg, and B cell populations in patients who respond favorably to PegIFN/RBV therapy.
© 2020 The Author(s). Published by S. Karger AG, Basel.

Entities:  

Keywords:  Hepatitis C virus; Immune cell subsets; Immunophenotyping

Year:  2020        PMID: 32966988      PMCID: PMC8114070          DOI: 10.1159/000511783

Source DB:  PubMed          Journal:  Med Princ Pract        ISSN: 1011-7571            Impact factor:   1.927


  30 in total

1.  A 2-log drop in viral load at 1 month is the best predictor of sustained response in HCV patients with normal ALT: a kinetic prospective study.

Authors:  P Deltenre; V Canva; M El Nady; C François; S Castelain; S Dharancy; A Louvet; L Bocket; M Lazrek; A Hollebecque; F Wartel; J Henrion; G Duverlie; P Mathurin
Journal:  J Viral Hepat       Date:  2009-05-08       Impact factor: 3.728

2.  Intrahepatic CD8+ T-cell failure during chronic hepatitis C virus infection.

Authors:  Hans Christian Spangenberg; Sergei Viazov; Nadine Kersting; Christoph Neumann-Haefelin; Denise McKinney; Michael Roggendorf; Fritz von Weizsäcker; Hubert E Blum; Robert Thimme
Journal:  Hepatology       Date:  2005-10       Impact factor: 17.425

3.  Restoration of HCV-specific CD8+ T cell function by interferon-free therapy.

Authors:  Bianca Martin; Nadine Hennecke; Volker Lohmann; Antonin Kayser; Christoph Neumann-Haefelin; George Kukolj; Wulf-Otto Böcher; Robert Thimme
Journal:  J Hepatol       Date:  2014-06-04       Impact factor: 25.083

4.  Imbalance of regulatory T cells and T helper type 17 cells in patients with chronic hepatitis C.

Authors:  Chunqiu Hao; Yun Zhou; Yu He; Chao Fan; Li Sun; Xin Wei; Linxu Wang; Meijuan Peng; Pingzhong Wang; Jianqi Lian; Zhansheng Jia
Journal:  Immunology       Date:  2014-12       Impact factor: 7.397

5.  Compromised function of natural killer cells in acute and chronic viral hepatitis.

Authors:  Sebastian Lunemann; David F G Malone; Julia Hengst; Kerstin Port; Jan Grabowski; Katja Deterding; Antoaneta Markova; Birgit Bremer; Verena Schlaphoff; Markus Cornberg; Michael P Manns; Johan K Sandberg; Hans-Gustaf Ljunggren; Niklas K Björkström; Heiner Wedemeyer
Journal:  J Infect Dis       Date:  2013-10-23       Impact factor: 5.226

6.  Decreased NK cell frequency in chronic hepatitis C does not affect ex vivo cytolytic killing.

Authors:  Chihiro Morishima; Denise M Paschal; Chia C Wang; Christina S Yoshihara; Brent L Wood; Anthony E T Yeo; Scott S Emerson; Margaret C Shuhart; David R Gretch
Journal:  Hepatology       Date:  2006-03       Impact factor: 17.425

7.  Phenotypic and functional changes of cytotoxic CD56pos natural T cells determine outcome of acute hepatitis C virus infection.

Authors:  Lucy Golden-Mason; Nicole Castelblanco; Cliona O'Farrelly; Hugo R Rosen
Journal:  J Virol       Date:  2007-06-06       Impact factor: 5.103

8.  Sustained response to interferon-alpha plus ribavirin therapy for chronic hepatitis C is closely associated with increased dynamism of intrahepatic natural killer and natural killer T cells.

Authors:  Satoshi Yamagiwa; Yasunobu Matsuda; Takafumi Ichida; Yutaka Honda; Masaaki Takamura; Satoshi Sugahara; Toru Ishikawa; Shogo Ohkoshi; Yoshinobu Sato; Yutaka Aoyagi
Journal:  Hepatol Res       Date:  2008-03-04       Impact factor: 4.288

Review 9.  Immunological aspects of antiviral therapy of chronic hepatitis B virus and hepatitis C virus infections.

Authors:  Barbara Rehermann; Antonio Bertoletti
Journal:  Hepatology       Date:  2015-02       Impact factor: 17.425

Review 10.  Hepatitis C Virus Infection: Host⁻Virus Interaction and Mechanisms of Viral Persistence.

Authors:  DeGaulle I Chigbu; Ronak Loonawat; Mohit Sehgal; Dip Patel; Pooja Jain
Journal:  Cells       Date:  2019-04-25       Impact factor: 6.600

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.