Literature DB >> 32954514

Developmental and epilepsy spectrum of KCNB1 encephalopathy with long-term outcome.

Claire Bar1,2, Mathieu Kuchenbuch1,2, Giulia Barcia2,3, Amy Schneider4, Mélanie Jennesson5, Gwenaël Le Guyader6,7, Gaetan Lesca8,9, Cyril Mignot10,11, Martino Montomoli12, Elena Parrini12, Hervé Isnard13, Anne Rolland14, Boris Keren11, Alexandra Afenjar15, Nathalie Dorison16,17, Lynette G Sadleir18, Delphine Breuillard1,2, Raphael Levy19, Marlène Rio3,20, Sophie Dupont10,21, Susanna Negrin22, Alberto Danieli22, Emmanuel Scalais23, Anne De Saint Martin24, Salima El Chehadeh25, Jamel Chelly25, Alice Poisson26, Anne-Sophie Lebre27, Anca Nica28,29, Sylvie Odent30,31, Tayeb Sekhara32, Vesna Brankovic33, Alice Goldenberg34, Pascal Vrielynck35, Damien Lederer36, Hélène Maurey37, Gaetano Terrone38, Claude Besmond39, Laurence Hubert39, Patrick Berquin40, Thierry Billette de Villemeur17, Bertrand Isidor41, Jeremy L Freeman42,43, Heather C Mefford44, Candace T Myers44, Katherine B Howell42,43, Andrés Rodríguez-Sacristán Cascajo45,46, Pierre Meyer47,48, David Genevieve49, Agnès Guët50, Diane Doummar17, Julien Durigneux42, Marieke F van Dooren51, Marie Claire Y de Wit52, Marion Gerard53, Isabelle Marey11, Arnold Munnich2,3, Renzo Guerrini12, Ingrid E Scheffer4,42,43,54, Edor Kabashi2, Rima Nabbout1,2.   

Abstract

OBJECTIVE: We aimed to delineate the phenotypic spectrum and long-term outcome of individuals with KCNB1 encephalopathy.
METHODS: We collected genetic, clinical, electroencephalographic, and imaging data of individuals with KCNB1 pathogenic variants recruited through an international collaboration, with the support of the family association "KCNB1 France." Patients were classified as having developmental and epileptic encephalopathy (DEE) or developmental encephalopathy (DE). In addition, we reviewed published cases and provided the long-term outcome in patients older than 12 years from our series and from literature.
RESULTS: Our series included 36 patients (21 males, median age = 10 years, range = 1.6 months-34 years). Twenty patients (56%) had DEE with infantile onset seizures (seizure onset = 10 months, range = 10 days-3.5 years), whereas 16 (33%) had DE with late onset epilepsy in 10 (seizure onset = 5 years, range = 18 months-25 years) and without epilepsy in six. Cognitive impairment was more severe in individuals with DEE compared to those with DE. Analysis of 73 individuals with KCNB1 pathogenic variants (36 from our series and 37 published individuals in nine reports) showed developmental delay in all with severe to profound intellectual disability in 67% (n = 41/61) and autistic features in 56% (n = 32/57). Long-term outcome in 22 individuals older than 12 years (14 in our series and eight published individuals) showed poor cognitive, psychiatric, and behavioral outcome. Epilepsy course was variable. Missense variants were associated with more frequent and more severe epilepsy compared to truncating variants. SIGNIFICANCE: Our study describes the phenotypic spectrum of KCNB1 encephalopathy, which varies from severe DEE to DE with or without epilepsy. Although cognitive impairment is worse in patients with DEE, long-term outcome is poor for most and missense variants are associated with more severe epilepsy outcome. Further understanding of disease mechanisms should facilitate the development of targeted therapies, much needed to improve the neurodevelopmental prognosis.
© 2020 International League Against Epilepsy.

Entities:  

Keywords:  autism spectrum disorders; developmental and epileptic encephalopathy; developmental encephalopathy; drug-resistant epilepsy; potassium channels; sudden unexpected death in epilepsy

Mesh:

Substances:

Year:  2020        PMID: 32954514     DOI: 10.1111/epi.16679

Source DB:  PubMed          Journal:  Epilepsia        ISSN: 0013-9580            Impact factor:   5.864


  4 in total

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Journal:  Biophys J       Date:  2022-03-18       Impact factor: 3.699

2.  A novel de novo KCNB1 variant altering channel characteristics in a patient with periventricular heterotopia, abnormal corpus callosum, and mild seizure outcome.

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3.  Mild phenotype in a patient with developmental and epileptic encephalopathy carrying a novel de novo KCNB1 variant.

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Journal:  Neurol Sci       Date:  2021-06-26       Impact factor: 3.307

4.  Correlation Analyses of Clinical Manifestations and Variant Effects in KCNB1-Related Neurodevelopmental Disorder.

Authors:  Juan Xiong; Zhonghua Liu; Shimeng Chen; Miriam Kessi; Baiyu Chen; Haolin Duan; Xiaolu Deng; Lifen Yang; Jing Peng; Fei Yin
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  4 in total

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