Literature DB >> 32914373

The Real-Life Data of BRAF Mutation on the Treatment of Colorectal Cancer: a TOG Study.

Ismail Beypinar1, Hacer Demir2, Abdullah Sakin3, Burcu Yapar Taskoylu4, Teoman Sakalar5, Yakup Ergun6, Mustafa Korkmaz7, Ozturk Ates8, Tulay Eren9, Serdar Turhal10, Mehmet Artac7.   

Abstract

PURPOSE: Colorectal cancer is the third leading diagnosis accounting for nearly 10% of all new cancers worldwide. The distinct features among BRAF mutant colorectal cancers make these tumor groups hard to treat for oncologists. The median overall survival (OS) of these types of cancers is reported to be 9 to 14 months.
METHODS: The study was declared on the Turkish Oncology Study Group Conference and approved. The patients' data was received from the centers who confirmed to participate. The BRAF-mutated patients were included in the study. The demographic features (age, gender, etc.), type of mutation, tumor localizations, histology, microsatellite instability (MSI) status, metastasis patterns chemotherapeutic agents and progression, and death times were recorded.
RESULTS: Thirty-nine patients were enrolled in the study. Sixteen patients had concurrent KRAS mutations, while 7 had NRAS mutations. Most of the patients received doublet chemotherapies in combination with anti-VEGF agents in the first and second line of the treatment. There was a significant difference in OS according to the stage which showed a decreased survival in stage IV patients at the time of diagnosis. Concurrent KRAS mutation resulted in increased OS. The median OS was 47 and 24 months favoring the KRAS mutant group. The patients whose primary tumor operated had better survival when compared with other patients. The median OS of the operated group was 47 months, while the non-operated group was 24 months. Liver metastasis was related to worse prognosis at the time of diagnosis in univariate analysis.
CONCLUSION: In our study we found a high concurrent RAS mutation ratio in a BRAF mutant patient group which was different from prior studies. The concurrent mutations resulted in a favorable outcome in terms of OS which is also different from the current knowledge. More prospective studies are needed especially BRAF-mutated patient population and especially with concurrent RAS mutations.
© 2020. Springer Science+Business Media, LLC, part of Springer Nature.

Entities:  

Keywords:  BRAF; Colon cancer; Concurrent mutation; KRAS; Real-life experience

Mesh:

Substances:

Year:  2021        PMID: 32914373     DOI: 10.1007/s12029-020-00514-9

Source DB:  PubMed          Journal:  J Gastrointest Cancer


  34 in total

1.  Relationship of Ki-ras mutations in colon cancers to tumor location, stage, and survival: a population-based study.

Authors:  W S Samowitz; K Curtin; D Schaffer; M Robertson; M Leppert; M L Slattery
Journal:  Cancer Epidemiol Biomarkers Prev       Date:  2000-11       Impact factor: 4.254

2.  Kirsten ras mutations in patients with colorectal cancer: the multicenter "RASCAL" study.

Authors:  H J Andreyev; A R Norman; D Cunningham; J R Oates; P A Clarke
Journal:  J Natl Cancer Inst       Date:  1998-05-06       Impact factor: 13.506

3.  Oncogenic KRAS and BRAF differentially regulate hypoxia-inducible factor-1alpha and -2alpha in colon cancer.

Authors:  Hirotoshi Kikuchi; Maria S Pino; Min Zeng; Senji Shirasawa; Daniel C Chung
Journal:  Cancer Res       Date:  2009-10-20       Impact factor: 12.701

4.  The type of K-ras mutation determines prognosis in colorectal cancer.

Authors:  J P Cerottini; S Caplin; E Saraga; J C Givel; J Benhattar
Journal:  Am J Surg       Date:  1998-03       Impact factor: 2.565

5.  Oligonucleotide microarray analysis of distinct gene expression patterns in colorectal cancer tissues harboring BRAF and K-ras mutations.

Authors:  Il-Jin Kim; Hio Chung Kang; Sang-Geun Jang; Kun Kim; Sun-A Ahn; Hyun-Ju Yoon; Sang Nam Yoon; Jae-Gahb Park
Journal:  Carcinogenesis       Date:  2005-10-11       Impact factor: 4.944

6.  ESMO Consensus Guidelines for management of patients with colon and rectal cancer. a personalized approach to clinical decision making.

Authors:  H J Schmoll; E Van Cutsem; A Stein; V Valentini; B Glimelius; K Haustermans; B Nordlinger; C J van de Velde; J Balmana; J Regula; I D Nagtegaal; R G Beets-Tan; D Arnold; F Ciardiello; P Hoff; D Kerr; C H Köhne; R Labianca; T Price; W Scheithauer; A Sobrero; J Tabernero; D Aderka; S Barroso; G Bodoky; J Y Douillard; H El Ghazaly; J Gallardo; A Garin; R Glynne-Jones; K Jordan; A Meshcheryakov; D Papamichail; P Pfeiffer; I Souglakos; S Turhal; A Cervantes
Journal:  Ann Oncol       Date:  2012-10       Impact factor: 32.976

7.  Global cancer statistics, 2012.

Authors:  Lindsey A Torre; Freddie Bray; Rebecca L Siegel; Jacques Ferlay; Joannie Lortet-Tieulent; Ahmedin Jemal
Journal:  CA Cancer J Clin       Date:  2015-02-04       Impact factor: 508.702

8.  BRAF(V600E) efficient transformation and induction of microsatellite instability versus KRAS(G12V) induction of senescence markers in human colon cancer cells.

Authors:  Eftychia Oikonomou; Eleni Makrodouli; Maria Evagelidou; Tobias Joyce; Lesley Probert; Alexander Pintzas
Journal:  Neoplasia       Date:  2009-11       Impact factor: 5.715

9.  Different genetic features associated with colon and rectal carcinogenesis.

Authors:  Milo Frattini; Debora Balestra; Simona Suardi; Maria Oggionni; Paola Alberici; Paolo Radice; Aurora Costa; Maria Grazia Daidone; Ermanno Leo; Silvana Pilotti; Lucio Bertario; Marco A Pierotti
Journal:  Clin Cancer Res       Date:  2004-06-15       Impact factor: 12.531

10.  Rare Though Not Mutually Exclusive: A Report of Three Cases of Concomitant KRAS and BRAF Mutation and a Review of the Literature.

Authors:  Ibrahim Halil Sahin; Syed M A Kazmi; Jeffrey T Yorio; Nishin A Bhadkamkar; Bryan K Kee; Christopher R Garrett
Journal:  J Cancer       Date:  2013-03-28       Impact factor: 4.207

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.